Jonathan W. Lovelace

1.3k total citations
23 papers, 842 citations indexed

About

Jonathan W. Lovelace is a scholar working on Cognitive Neuroscience, Molecular Biology and Genetics. According to data from OpenAlex, Jonathan W. Lovelace has authored 23 papers receiving a total of 842 indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Cognitive Neuroscience, 12 papers in Molecular Biology and 12 papers in Genetics. Recurrent topics in Jonathan W. Lovelace's work include Genetics and Neurodevelopmental Disorders (12 papers), Autism Spectrum Disorder Research (10 papers) and Congenital heart defects research (8 papers). Jonathan W. Lovelace is often cited by papers focused on Genetics and Neurodevelopmental Disorders (12 papers), Autism Spectrum Disorder Research (10 papers) and Congenital heart defects research (8 papers). Jonathan W. Lovelace collaborates with scholars based in United States, Germany and Canada. Jonathan W. Lovelace's co-authors include Devin K. Binder, Khaleel A. Razak, Iryna M. Ethell, Edward Korzus, Philip A. Vieira, Teresa H. Wen, Maham Rais, Carrie R. Jonak, Sarah M. Reinhard and Mike S. Hsu and has published in prestigious journals such as Nature, Neuron and The FASEB Journal.

In The Last Decade

Jonathan W. Lovelace

23 papers receiving 838 citations

Peers

Jonathan W. Lovelace

GENET

CMN

PARAS

Haley E. Speed United States

Jin Nakatani Japan

Hsiao‐Mei Liao United States

Kimie Niimi Japan

Katherine R. Croce United States

Joseph L. Cheatwood United States

Sehoon Keum South Korea

Arturo Sierra Mexico

Bong-June Yoon South Korea

Hidetoshi Ishibashi Japan

Haley E. Speed United States

Jonathan W. Lovelace

466 ×1.0

484 ×1.2

GENET

357 ×1.1

265 ×1.2

CMN

18 ×0.2

PARAS

Citations per year, relative to Jonathan W. Lovelace Jonathan W. Lovelace (= 1×) peers Haley E. Speed

Countries citing papers authored by Jonathan W. Lovelace

Since Specialization

Citations

This map shows the geographic impact of Jonathan W. Lovelace's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan W. Lovelace with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan W. Lovelace more than expected).

Fields of papers citing papers by Jonathan W. Lovelace

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan W. Lovelace. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan W. Lovelace. The network helps show where Jonathan W. Lovelace may publish in the future.

Co-authorship network of co-authors of Jonathan W. Lovelace

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan W. Lovelace. A scholar is included among the top collaborators of Jonathan W. Lovelace based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan W. Lovelace. Jonathan W. Lovelace is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Lovelace, Jonathan W., et al.. (2024). Defining cardioception: Heart-brain crosstalk. Neuron. 112(22). 3671–3674. 7 indexed citations

Lovelace, Jonathan W., Zhengyuan Pang, Yunxiao Zhang, et al.. (2023). Vagal sensory neurons mediate the Bezold–Jarisch reflex and induce syncope. Nature. 623(7986). 387–396. 49 indexed citations

Lovelace, Jonathan W., et al.. (2022). Nicotine reduces age-related changes in cortical neural oscillations without affecting auditory brainstem responses. Neurobiology of Aging. 120. 10–26. 2 indexed citations

Lovelace, Jonathan W., Craig A. Erickson, Christina Groß, et al.. (2022). A sound-driven cortical phase-locking change in the Fmr1 KO mouse requires Fmr1 deletion in a subpopulation of brainstem neurons. Neurobiology of Disease. 170. 105767–105767. 8 indexed citations

Rais, Maham, Jonathan W. Lovelace, Austin Nguy, et al.. (2021). Functional consequences of postnatal interventions in a mouse model of Fragile X syndrome. Neurobiology of Disease. 162. 105577–105577. 14 indexed citations

Jonak, Carrie R., Donovan A. Argueta, Jonathan W. Lovelace, et al.. (2021). Increased 2-arachidonoyl-sn-glycerol levels normalize cortical responses to sound and improve behaviors in Fmr1 KO mice. Journal of Neurodevelopmental Disorders. 13(1). 47–47. 14 indexed citations

Jonak, Carrie R., Jonathan W. Lovelace, Iryna M. Ethell, Khaleel A. Razak, & Devin K. Binder. (2020). Multielectrode array analysis of EEG biomarkers in a mouse model of Fragile X Syndrome. Neurobiology of Disease. 138. 104794–104794. 46 indexed citations

Lovelace, Jonathan W., Iryna M. Ethell, Devin K. Binder, & Khaleel A. Razak. (2020). Minocycline Treatment Reverses Sound Evoked EEG Abnormalities in a Mouse Model of Fragile X Syndrome. Frontiers in Neuroscience. 14. 771–771. 25 indexed citations

Lovelace, Jonathan W., et al.. (2020). Age- and movement-related modulation of cortical oscillations in a mouse model of presbycusis. Hearing Research. 402. 108095–108095. 11 indexed citations

10.

Rais, Maham, et al.. (2020). Acute pharmacological inhibition of matrix metalloproteinase‐9 activity during development restores perineuronal net formation and normalizes auditory processing in Fmr1 KO mice. Journal of Neurochemistry. 155(5). 538–558. 44 indexed citations

11.

Kulinich, Anna, Sarah M. Reinhard, Maham Rais, et al.. (2019). Beneficial effects of sound exposure on auditory cortex development in a mouse model of Fragile X Syndrome. Neurobiology of Disease. 134. 104622–104622. 19 indexed citations

12.

Jonak, Carrie R., Jonathan W. Lovelace, Iryna M. Ethell, Khaleel A. Razak, & Devin K. Binder. (2018). Reusable Multielectrode Array Technique for Electroencephalography in Awake Freely Moving Mice. Frontiers in Integrative Neuroscience. 12. 53–53. 20 indexed citations

13.

Lovelace, Jonathan W., Iryna M. Ethell, Devin K. Binder, & Khaleel A. Razak. (2018). Translation-relevant EEG phenotypes in a mouse model of Fragile X Syndrome. Neurobiology of Disease. 115. 39–48. 102 indexed citations

14.

Wen, Teresa H., Jonathan W. Lovelace, Iryna M. Ethell, Devin K. Binder, & Khaleel A. Razak. (2018). Developmental Changes in EEG Phenotypes in a Mouse Model of Fragile X Syndrome. Neuroscience. 398. 126–143. 52 indexed citations

15.

Lovelace, Jonathan W., Teresa H. Wen, Sarah M. Reinhard, et al.. (2016). Matrix metalloproteinase-9 deletion rescues auditory evoked potential habituation deficit in a mouse model of Fragile X Syndrome. Neurobiology of Disease. 89. 126–135. 82 indexed citations

16.

David, Clément N., Elma S. Frias, Jenny I. Szu, et al.. (2016). GLT-1-Dependent Disruption of CNS Glutamate Homeostasis and Neuronal Function by the Protozoan Parasite Toxoplasma gondii. PLoS Pathogens. 12(6). e1005643–e1005643. 112 indexed citations

17.

Lovelace, Jonathan W., et al.. (2015). An animal model of female adolescent cannabinoid exposure elicits a long-lasting deficit in presynaptic long-term plasticity. Neuropharmacology. 99. 242–255. 33 indexed citations

18.

Vieira, Philip A., et al.. (2015). Prefrontal NMDA receptors expressed in excitatory neurons control fear discrimination and fear extinction. Neurobiology of Learning and Memory. 119. 52–62. 42 indexed citations

19.

Vieira, Philip A., et al.. (2014). Prefrontal consolidation supports the attainment of fear memory accuracy. Learning & Memory. 21(8). 394–405. 25 indexed citations

20.

Lovelace, Jonathan W., et al.. (2014). Impaired Fear Memory Specificity Associated with Deficient Endocannabinoid-Dependent Long-Term Plasticity. Neuropsychopharmacology. 39(7). 1685–1693. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact