Jonathan L. Tobin

1.8k total citations
8 papers, 900 citations indexed

About

Jonathan L. Tobin is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Jonathan L. Tobin has authored 8 papers receiving a total of 900 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 6 papers in Genetics and 1 paper in Cellular and Molecular Neuroscience. Recurrent topics in Jonathan L. Tobin's work include Genetic and Kidney Cyst Diseases (6 papers), Genetic Syndromes and Imprinting (3 papers) and Hedgehog Signaling Pathway Studies (3 papers). Jonathan L. Tobin is often cited by papers focused on Genetic and Kidney Cyst Diseases (6 papers), Genetic Syndromes and Imprinting (3 papers) and Hedgehog Signaling Pathway Studies (3 papers). Jonathan L. Tobin collaborates with scholars based in United Kingdom, United States and Australia. Jonathan L. Tobin's co-authors include Philip L. Beales, Robyn Quinlan, Masazumi Tada, Beyhan Tüysüz, Melita Irving, Chad G. Pearson, Suzanne Rix, Nursel Elçioğlu, Masatake Kai and Peter Scambler and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Genetics and BMC Evolutionary Biology.

In The Last Decade

Jonathan L. Tobin

8 papers receiving 888 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jonathan L. Tobin United Kingdom 8 690 690 152 91 72 8 900
Phil Beales United Kingdom 6 748 1.1× 916 1.3× 212 1.4× 137 1.5× 57 0.8× 8 1.1k
Mandy J. Croyle United States 11 538 0.8× 608 0.9× 105 0.7× 61 0.7× 90 1.3× 21 758
Emilie Legué United States 10 524 0.8× 392 0.6× 147 1.0× 69 0.8× 46 0.6× 13 654
Bozena Polok Switzerland 7 697 1.0× 491 0.7× 148 1.0× 42 0.5× 53 0.7× 10 837
Lorraine Eley United Kingdom 15 559 0.8× 415 0.6× 109 0.7× 91 1.0× 91 1.3× 28 720
Daniel P. S. Osborn United Kingdom 16 812 1.2× 450 0.7× 202 1.3× 51 0.6× 56 0.8× 23 975
Alison Ross United Kingdom 7 1.1k 1.7× 1.1k 1.6× 274 1.8× 96 1.1× 211 2.9× 10 1.6k
Shiaulou Yuan United States 16 778 1.1× 475 0.7× 205 1.3× 50 0.5× 57 0.8× 20 988
Christel Thauvin‐Robinet France 12 417 0.6× 524 0.8× 76 0.5× 93 1.0× 36 0.5× 15 660
Judith B. Kenyon United States 9 490 0.7× 401 0.6× 114 0.8× 64 0.7× 33 0.5× 10 772

Countries citing papers authored by Jonathan L. Tobin

Since Specialization
Citations

This map shows the geographic impact of Jonathan L. Tobin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan L. Tobin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan L. Tobin more than expected).

Fields of papers citing papers by Jonathan L. Tobin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan L. Tobin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan L. Tobin. The network helps show where Jonathan L. Tobin may publish in the future.

Co-authorship network of co-authors of Jonathan L. Tobin

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan L. Tobin. A scholar is included among the top collaborators of Jonathan L. Tobin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan L. Tobin. Jonathan L. Tobin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

8 of 8 papers shown
1.
Mahmood, Fahad, Monika Mozere, Anselm A. Zdebik, et al.. (2013). Generation and validation of a zebrafish model of EAST (Epilepsy, ataxia, sensorineural deafness and tubulopathy) syndrome. Disease Models & Mechanisms. 6(3). 652–60. 43 indexed citations
2.
McDougall, Carmel, et al.. (2011). Annelid Distal-less/Dlx duplications reveal varied post-duplication fates. BMC Evolutionary Biology. 11(1). 241–241. 15 indexed citations
3.
Tobin, Jonathan L. & Philip L. Beales. (2009). The nonmotile ciliopathies. Genetics in Medicine. 11(6). 386–402. 149 indexed citations
4.
Tobin, Jonathan L. & Philip L. Beales. (2008). Restoration of renal function in zebrafish models of ciliopathies. Pediatric Nephrology. 23(11). 2095–2099. 63 indexed citations
5.
Quinlan, Robyn, Jonathan L. Tobin, & Philip L. Beales. (2008). Chapter 5 Modeling Ciliopathies. Current topics in developmental biology. 84. 249–310. 113 indexed citations
6.
Tobin, Jonathan L., Erica R. Eichers, Helen May‐Simera, et al.. (2008). Inhibition of neural crest migration underlies craniofacial dysmorphology and Hirschsprung's disease in Bardet–Biedl syndrome. Proceedings of the National Academy of Sciences. 105(18). 6714–6719. 154 indexed citations
7.
Beales, Philip L., Jonathan L. Tobin, Chiara Bacchelli, et al.. (2007). IFT80, which encodes a conserved intraflagellar transport protein, is mutated in Jeune asphyxiating thoracic dystrophy. Nature Genetics. 39(6). 727–729. 227 indexed citations
8.
Tobin, Jonathan L. & Philip L. Beales. (2007). Bardet–Biedl syndrome: beyond the cilium. Pediatric Nephrology. 22(7). 926–936. 136 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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