Jonathan E. Boyson

4.0k total citations · 1 hit paper
55 papers, 3.2k citations indexed

About

Jonathan E. Boyson is a scholar working on Immunology, Molecular Biology and Infectious Diseases. According to data from OpenAlex, Jonathan E. Boyson has authored 55 papers receiving a total of 3.2k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Immunology, 7 papers in Molecular Biology and 6 papers in Infectious Diseases. Recurrent topics in Jonathan E. Boyson's work include Immune Cell Function and Interaction (30 papers), T-cell and B-cell Immunology (27 papers) and Reproductive System and Pregnancy (10 papers). Jonathan E. Boyson is often cited by papers focused on Immune Cell Function and Interaction (30 papers), T-cell and B-cell Immunology (27 papers) and Reproductive System and Pregnancy (10 papers). Jonathan E. Boyson collaborates with scholars based in United States, United Kingdom and China. Jonathan E. Boyson's co-authors include Jack L. Strominger, Louise A. Koopman, Jordan S. Orange, Frederick Schatz, Basya Rybalov, Rachel Masch, Charles J. Lockwood, Asher D. Schachter, Hernan D. Kopcow and Peter J. Park and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and Nature Immunology.

In The Last Decade

Jonathan E. Boyson

54 papers receiving 3.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Human Decidual Natural Killer Cells Are a Unique NK Cell Subset with Immunomodulatory Potential

2003 696 citations Jonathan E. Boyson, Jack L. Strominger et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jonathan E. Boyson United States	27	2.6k	412	408	383	375	55	3.2k
Tal I. Arnon Israel	25	4.3k 1.7×	483 1.2×	421 1.0×	605 1.6×	717 1.9×	33	5.1k
R Biagiotti Italy	24	2.7k 1.0×	235 0.6×	432 1.1×	359 0.9×	158 0.4×	65	4.1k
Béatrice Riteau France	31	2.8k 1.1×	529 1.3×	553 1.4×	508 1.3×	219 0.6×	42	3.6k
David J. Sharkey Australia	23	1.4k 0.5×	730 1.8×	625 1.5×	629 1.6×	494 1.3×	59	2.7k
Earl L. Parr United States	35	2.0k 0.8×	500 1.2×	431 1.1×	383 1.0×	310 0.8×	89	3.1k
Margaret B. Parr United States	31	1.6k 0.6×	410 1.0×	251 0.6×	256 0.7×	266 0.7×	62	2.2k
Salvatore Sampognaro Italy	21	2.0k 0.8×	136 0.3×	257 0.6×	200 0.5×	98 0.3×	26	3.1k
M G Giudizi Italy	22	3.1k 1.2×	148 0.4×	374 0.9×	402 1.0×	103 0.3×	29	4.5k
J. Martal France	30	1.9k 0.7×	507 1.2×	899 2.2×	484 1.3×	594 1.6×	77	3.4k
Marina Zaitseva United States	29	1.1k 0.4×	220 0.5×	79 0.2×	420 1.1×	255 0.7×	56	2.3k

Countries citing papers authored by Jonathan E. Boyson

Since Specialization

Citations

This map shows the geographic impact of Jonathan E. Boyson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan E. Boyson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan E. Boyson more than expected).

Fields of papers citing papers by Jonathan E. Boyson

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan E. Boyson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan E. Boyson. The network helps show where Jonathan E. Boyson may publish in the future.

Co-authorship network of co-authors of Jonathan E. Boyson

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan E. Boyson. A scholar is included among the top collaborators of Jonathan E. Boyson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan E. Boyson. Jonathan E. Boyson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Dienz, Oliver, et al.. (2024). SLAM/SAP signaling regulates discrete γδ T cell developmental checkpoints and shapes the innate-like γδ TCR repertoire. eLife. 13.

Villalba, Nuria, Yen‐Lin Chen, Swapnil K. Sonkusare, et al.. (2023). The Polyanionic Drug Suramin Neutralizes Histones and Prevents Endotheliopathy. The Journal of Immunology. 211(4). 648–657. 10 indexed citations

Dienz, Oliver, et al.. (2020). Critical Role for SLAM/SAP Signaling in the Thymic Developmental Programming of IL-17– and IFN-γ–Producing γδ T Cells. The Journal of Immunology. 204(6). 1521–1534. 17 indexed citations

Nivarthi, Usha K., Kristen K. Pierce, Stephen S. Whitehead, et al.. (2020). Stimulation of B Cell Immunity in Flavivirus-Naïve Individuals by the Tetravalent Live Attenuated Dengue Vaccine TV003. SSRN Electronic Journal. 1 indexed citations

Diehl, Sean A., Kristen K. Pierce, Stephen S. Whitehead, et al.. (2020). Rapid Induction and Maintenance of Virus-Specific CD8+ TEMRA and CD4+ TEM Cells Following Protective Vaccination Against Dengue Virus Challenge in Humans. Frontiers in Immunology. 11. 479–479. 35 indexed citations

Randall, Matthew J., Philip Eisenhauer, Jason Botten, et al.. (2018). Bacterial Lipoproteins Constitute the TLR2-Stimulating Activity of Serum Amyloid A. The Journal of Immunology. 201(8). 2377–2384. 25 indexed citations

Ather, Jennifer L., Oliver Dienz, Jonathan E. Boyson, et al.. (2018). Serum Amyloid A3 is required for normal lung development and survival following influenza infection. Scientific Reports. 8(1). 16571–16571. 22 indexed citations

Krementsov, Dimitry N., Laure K. Case, Oliver Dienz, et al.. (2017). Genetic variation in chromosome Y regulates susceptibility to influenza A virus infection. Proceedings of the National Academy of Sciences. 114(13). 3491–3496. 47 indexed citations

Ather, Jennifer L., Matthew J. Randall, Mridul Kanti Mandal, et al.. (2016). Uricase Inhibits Nitrogen Dioxide–Promoted Allergic Sensitization to Inhaled Ovalbumin Independent of Uric Acid Catabolism. The Journal of Immunology. 197(5). 1720–1732. 1 indexed citations

10.

Ather, Jennifer L., Lennart K. A. Lundblad, Benjamin T. Suratt, et al.. (2013). Interleukin-1 Receptor and Caspase-1 Are Required for the Th17 Response in Nitrogen Dioxide–Promoted Allergic Airway Disease. American Journal of Respiratory Cell and Molecular Biology. 48(5). 655–664. 44 indexed citations

11.

Klaus, Joseph P., Philip Eisenhauer, Anne B. Mason, et al.. (2013). The Intracellular Cargo Receptor ERGIC-53 Is Required for the Production of Infectious Arenavirus, Coronavirus, and Filovirus Particles. Cell Host & Microbe. 14(5). 522–534. 54 indexed citations

12.

Huber, Sally A., Brian Roberts, Mohamad Moussawi, & Jonathan E. Boyson. (2012). Slam Haplotype 2 Promotes NKT But Suppresses Vγ4+ T-Cell Activation in Coxsackievirus B3 Infection Leading to Increased Liver Damage But Reduced Myocarditis. American Journal Of Pathology. 182(2). 401–409. 6 indexed citations

13.

Ather, Jennifer L., Karina Ckless, Roland Martinꝉ, et al.. (2011). Serum Amyloid A Activates the NLRP3 Inflammasome and Promotes Th17 Allergic Asthma in Mice. The Journal of Immunology. 187(1). 64–73. 193 indexed citations

14.

Olson, Chris M., Tonya C. Bates, Hooman Izadi, et al.. (2009). Local Production of IFN-γ by Invariant NKT Cells Modulates Acute Lyme Carditis. The Journal of Immunology. 182(6). 3728–3734. 84 indexed citations

15.

Exley, Mark A., Runhua Hou, Angela Shaulov, et al.. (2008). Selective activation, expansion, and monitoring of human iNKT cells with a monoclonal antibody specific for the TCR α‐chain CDR3 loop. European Journal of Immunology. 38(6). 1756–1766. 85 indexed citations

16.

Smiley, Stephen T., Paula A. Lanthier, Kevin N. Couper, et al.. (2005). Exacerbated Susceptibility to Infection-Stimulated Immunopathology in CD1d-Deficient Mice. The Journal of Immunology. 174(12). 7904–7911. 30 indexed citations

17.

Thomas, Seddon Y., Runhua Hou, Jonathan E. Boyson, et al.. (2003). CD1d-Restricted NKT Cells Express a Chemokine Receptor Profile Indicative of Th1-Type Inflammatory Homing Cells. The Journal of Immunology. 171(5). 2571–2580. 186 indexed citations

18.

Malik, Pratap, et al.. (2002). Uniquely Conformed Peptide-Containing β2-Microglobulin- Free Heavy Chains of HLA-B2705 on the Cell Surface. The Journal of Immunology. 169(8). 4379–4387. 33 indexed citations

19.

Evans, David T., Marian S. Piekarczyk, Todd M. Allen, et al.. (1997). Immunodominance of a single CTL epitope in a primate species with limited MHC class I polymorphism. The Journal of Immunology. 159(3). 1374–1382. 20 indexed citations

20.

Boyson, Jonathan E., et al.. (1996). Identification of the rhesus monkey HLA-G ortholog. Mamu-G is a pseudogene. The Journal of Immunology. 157(12). 5428–5437. 59 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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