Jonathan C. Kentish

4.6k total citations · 1 hit paper
51 papers, 3.8k citations indexed

About

Jonathan C. Kentish is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Biomedical Engineering. According to data from OpenAlex, Jonathan C. Kentish has authored 51 papers receiving a total of 3.8k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Cardiology and Cardiovascular Medicine, 27 papers in Molecular Biology and 9 papers in Biomedical Engineering. Recurrent topics in Jonathan C. Kentish's work include Cardiomyopathy and Myosin Studies (26 papers), Cardiac electrophysiology and arrhythmias (18 papers) and Ion channel regulation and function (15 papers). Jonathan C. Kentish is often cited by papers focused on Cardiomyopathy and Myosin Studies (26 papers), Cardiac electrophysiology and arrhythmias (18 papers) and Ion channel regulation and function (15 papers). Jonathan C. Kentish collaborates with scholars based in United Kingdom, United States and Australia. Jonathan C. Kentish's co-authors include David J. Allen, Joanne Layland, David G. Allen, Sue M. Palmer, Sonya C. Bardswell, Antoni Wrzosek, Metin Avkiran, H E ter Keurs, L Ricciardi and Jeroen J.J. Bucx and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Circulation Research.

In The Last Decade

Jonathan C. Kentish

50 papers receiving 3.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The cellular basis of the length-tension relation in cardiac muscle

1985 466 citations Jonathan C. Kentish et al. Journal of Molecular and Cellular Cardiology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jonathan C. Kentish United Kingdom	29	3.0k	1.9k	490	343	287	51	3.8k
Norman R. Alpert United States	41	4.5k 1.5×	2.6k 1.4×	567 1.2×	393 1.1×	300 1.0×	112	5.6k
Saul Winegrad United States	34	2.6k 0.9×	2.0k 1.1×	395 0.8×	506 1.5×	165 0.6×	74	3.5k
Pieter P. de Tombe United States	52	6.1k 2.0×	3.4k 1.8×	615 1.3×	374 1.1×	236 0.8×	158	7.3k
L. A. Mulieri United States	24	2.1k 0.7×	981 0.5×	470 1.0×	239 0.7×	146 0.5×	57	2.5k
Bradley M. Palmer United States	31	2.2k 0.7×	1.3k 0.7×	253 0.5×	115 0.3×	95 0.3×	92	3.0k
Gonzalo Pizarro United States	30	1.9k 0.6×	1.9k 1.0×	320 0.7×	1.2k 3.5×	249 0.9×	100	3.3k
Martina Krüger Germany	29	2.0k 0.7×	1.6k 0.8×	296 0.6×	109 0.3×	70 0.2×	49	3.0k
Beata M. Wolska United States	33	2.4k 0.8×	2.0k 1.1×	92 0.2×	280 0.8×	185 0.6×	87	3.5k
Anthony J. Baker United States	27	1.0k 0.3×	945 0.5×	298 0.6×	299 0.9×	223 0.8×	77	2.2k
Todd J. Herron United States	33	1.7k 0.6×	2.1k 1.1×	565 1.2×	672 2.0×	67 0.2×	72	3.5k

Countries citing papers authored by Jonathan C. Kentish

Since Specialization

Citations

This map shows the geographic impact of Jonathan C. Kentish's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan C. Kentish with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan C. Kentish more than expected).

Fields of papers citing papers by Jonathan C. Kentish

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan C. Kentish. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan C. Kentish. The network helps show where Jonathan C. Kentish may publish in the future.

Co-authorship network of co-authors of Jonathan C. Kentish

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan C. Kentish. A scholar is included among the top collaborators of Jonathan C. Kentish based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan C. Kentish. Jonathan C. Kentish is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Bardswell, Sonya C., Matthew Arno, Lu Han, et al.. (2020). Right Ventricle Has Normal Myofilament Function But Shows Perturbations in the Expression of Extracellular Matrix Genes in Patients With Tetralogy of Fallot Undergoing Pulmonary Valve Replacement. Journal of the American Heart Association. 9(16). e015342–e015342. 11 indexed citations

Land, Sander, et al.. (2017). A model of cardiac contraction based on novel measurements of tension development in human cardiomyocytes. Journal of Molecular and Cellular Cardiology. 106. 68–83. 102 indexed citations

Fraysse, B., Sonya C. Bardswell, Nicolas Vignier, et al.. (2012). Increased myofilament Ca2+ sensitivity and diastolic dysfunction as early consequences of Mybpc3 mutation in heterozygous knock-in mice. Journal of Molecular and Cellular Cardiology. 52(6). 1299–1307. 115 indexed citations

Bardswell, Sonya C., Friederike Cuello, Jonathan C. Kentish, & Metin Avkiran. (2011). cMyBP-C as a promiscuous substrate: phosphorylation by non-PKA kinases and its potential significance. Journal of Muscle Research and Cell Motility. 33(1). 53–60. 33 indexed citations

Hoskins, Anita C., et al.. (2010). Enhanced length-dependent Ca2+ activation in fish cardiomyocytes permits a large operating range of sarcomere lengths. Journal of Molecular and Cellular Cardiology. 48(5). 917–924. 24 indexed citations

Fallouh, Hazem, Sonya C. Bardswell, Linda M. McLatchie, et al.. (2010). Esmolol cardioplegia: the cellular mechanism of diastolic arrest. Cardiovascular Research. 87(3). 552–560. 23 indexed citations

Cuello, Friederike, Sonya C. Bardswell, Robert S. Haworth, et al.. (2010). Novel Role for p90 Ribosomal S6 Kinase in the Regulation of Cardiac Myofilament Phosphorylation. Journal of Biological Chemistry. 286(7). 5300–5310. 43 indexed citations

Hoskins, Anita C., Adam Jacques, Sonya C. Bardswell, et al.. (2010). Normal passive viscoelasticity but abnormal myofibrillar force generation in human hypertrophic cardiomyopathy. Journal of Molecular and Cellular Cardiology. 49(5). 737–745. 58 indexed citations

Marshall, Melanie, Narayana Anilkumar, Joanne Layland, et al.. (2009). Protein phosphatase 2A contributes to the cardiac dysfunction induced by endotoxemia. Cardiovascular Research. 82(1). 67–76. 24 indexed citations

10.

Bardswell, Sonya C., Friederike Cuello, Sakthivel Sadayappan, et al.. (2009). Distinct Sarcomeric Substrates Are Responsible for Protein Kinase D-mediated Regulation of Cardiac Myofilament Ca2+ Sensitivity and Cross-bridge Cycling. Journal of Biological Chemistry. 285(8). 5674–5682. 92 indexed citations

11.

Jacques, Adam, Anita C. Hoskins, Jonathan C. Kentish, & Steven B. Marston. (2008). From genotype to phenotype: a longitudinal study of a patient with hypertrophic cardiomyopathy due to a mutation in the MYBPC3 gene. Journal of Muscle Research and Cell Motility. 29(6-8). 239–246. 30 indexed citations

12.

Brennan, Jonathan P., Sonya C. Bardswell, Joseph R. Burgoyne, et al.. (2006). Direct activation of Type I protein kinase a (PKA) by oxidants independently of cAMP is mediated by RI subunit interprotein disulphide bond formation. Circulation. 114(18). 1–2. 24 indexed citations

13.

Brennan, Jonathan P., Sonya C. Bardswell, Joseph R. Burgoyne, et al.. (2006). Oxidant-induced Activation of Type I Protein Kinase A Is Mediated by RI Subunit Interprotein Disulfide Bond Formation. Journal of Biological Chemistry. 281(31). 21827–21836. 203 indexed citations

14.

Bardswell, Sonya C. & Jonathan C. Kentish. (2005). Effect of beta-adrenoceptor stimulation on the slow force response to a length change in cardiac muscle. Journal of Molecular and Cellular Cardiology. 39(1).

15.

Herron, Todd J., et al.. (2005). Age dependence of isometric crossbridge cycling and alpha-MyHC isoform expression in human ventricular myocytes. Biophysical Journal. 88(1). 4 indexed citations

16.

Layland, Joanne & Jonathan C. Kentish. (2002). Myofilament‐based relaxant effect of isoprenaline revealed during work‐loop contractions in rat cardiac trabeculae. The Journal of Physiology. 544(1). 171–182. 17 indexed citations

17.

Kentish, Jonathan C. & Antoni Wrzosek. (1998). Changes in force and cytosolic Ca²⁺ concentration after length changes in isolated rat ventricular trabeculae. The Journal of Physiology. 506(2). 431–444. 139 indexed citations

18.

Kentish, Jonathan C.. (1997). Ca2+- and caffeine-induced Ca2+ release from the sarcoplasmic reticulum in rat skinned trabeculae: effects of pH and Pi. Cardiovascular Research. 33(2). 314–323. 24 indexed citations

19.

Kentish, Jonathan C., et al.. (1992). Isoprenaline reverses the slow force responses to a length change in isolated rabbit papillary muscle. Pflügers Archiv - European Journal of Physiology. 421(5). 519–521. 17 indexed citations

20.

Kentish, Jonathan C.. (1991). Combined inhibitory actions of acidosis and phosphate on maximum force production in rat skinned cardiac muscle. Pflügers Archiv - European Journal of Physiology. 419(3-4). 310–318. 50 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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