Jonathan B. Coulter

1.5k total citations
25 papers, 1.1k citations indexed

About

Jonathan B. Coulter is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cancer Research. According to data from OpenAlex, Jonathan B. Coulter has authored 25 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 8 papers in Pulmonary and Respiratory Medicine and 6 papers in Cancer Research. Recurrent topics in Jonathan B. Coulter's work include Epigenetics and DNA Methylation (8 papers), Prostate Cancer Treatment and Research (6 papers) and DNA Repair Mechanisms (4 papers). Jonathan B. Coulter is often cited by papers focused on Epigenetics and DNA Methylation (8 papers), Prostate Cancer Treatment and Research (6 papers) and DNA Repair Mechanisms (4 papers). Jonathan B. Coulter collaborates with scholars based in United States, Japan and Netherlands. Jonathan B. Coulter's co-authors include Edward Gabrielson, Ellen Tully, Hajime Orita, Edward Gabrielson, Joseph Bressler, Cliona O’Driscoll, Francis P. Kuhajda, Kathleen Wilsbach, Srikanta Kumar Rath and Elena Feinstein and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Jonathan B. Coulter

23 papers receiving 1.1k citations

Peers

Jonathan B. Coulter
Jiaze An China
Tin Lok Wong Hong Kong
Albert A. Geldof Netherlands
Nina Gellert Germany
Laura Poillet-Perez United States
Jonathan B. Coulter
Citations per year, relative to Jonathan B. Coulter Jonathan B. Coulter (= 1×) peers Riccardo Di Fiore

Countries citing papers authored by Jonathan B. Coulter

Since Specialization
Citations

This map shows the geographic impact of Jonathan B. Coulter's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jonathan B. Coulter with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jonathan B. Coulter more than expected).

Fields of papers citing papers by Jonathan B. Coulter

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jonathan B. Coulter. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jonathan B. Coulter. The network helps show where Jonathan B. Coulter may publish in the future.

Co-authorship network of co-authors of Jonathan B. Coulter

This figure shows the co-authorship network connecting the top 25 collaborators of Jonathan B. Coulter. A scholar is included among the top collaborators of Jonathan B. Coulter based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jonathan B. Coulter. Jonathan B. Coulter is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Coulter, Jonathan B., et al.. (2025). Effects of hypoxia and iron on ascorbic acid-mediated cytotoxicity in prostate cancer cell lines. Toxicology and Applied Pharmacology. 497. 117259–117259.
3.
Sforza, Daniel, John Wong, Kathleen L. Gabrielson, et al.. (2023). FLASH Effects Induced by Orthovoltage X-Rays. International Journal of Radiation Oncology*Biology*Physics. 117(4). 1018–1027. 21 indexed citations
4.
López-Bertoni, Hernando, Amanda Johnson, Yuan Rui, et al.. (2022). Sox2 induces glioblastoma cell stemness and tumor propagation by repressing TET2 and deregulating 5hmC and 5mC DNA modifications. Signal Transduction and Targeted Therapy. 7(1). 37–37. 67 indexed citations
5.
Shrestha, Eva, Jonathan B. Coulter, Sarah E. Ernst, et al.. (2021). Oncogenic gene fusions in nonneoplastic precursors as evidence that bacterial infection can initiate prostate cancer. Proceedings of the National Academy of Sciences. 118(32). 32 indexed citations
6.
Coulter, Jonathan B., Daniel Y. Song, Theodore L. DeWeese, & Srinivasan Yegnasubramanian. (2021). Mechanisms, Challenges, and Opportunities in Combined Radiation and Hormonal Therapies. Seminars in Radiation Oncology. 32(1). 76–81. 2 indexed citations
7.
Oei, Arlene L., Preethi Korangath, Kathleen R. Mulka, et al.. (2019). Enhancing the abscopal effect of radiation and immune checkpoint inhibitor therapies with magnetic nanoparticle hyperthermia in a model of metastatic breast cancer. International Journal of Hyperthermia. 36(sup1). 47–63. 46 indexed citations
8.
López-Bertoni, Hernando, et al.. (2019). TET1 regulates DNA repair in human glial cells. Toxicology and Applied Pharmacology. 380. 114646–114646. 6 indexed citations
9.
Coulter, Jonathan B., et al.. (2017). TET1 deficiency attenuates the DNA damage response and promotes resistance to DNA damaging agents. Epigenetics. 12(10). 854–864. 21 indexed citations
10.
Haffner, Michael C., Jonathan B. Coulter, Raju R. Raval, et al.. (2016). Androgen Deprivation Followed by Acute Androgen Stimulation Selectively Sensitizes AR-Positive Prostate Cancer Cells to Ionizing Radiation. Clinical Cancer Research. 22(13). 3310–3319. 33 indexed citations
11.
Coulter, Jonathan B., et al.. (2015). Novel Role of Ten-Eleven-Translocation Methylcytosine Dioxygenase 1 (TET1) in DNA Damage Response. The FASEB Journal. 29. 1 indexed citations
12.
Hatano, Koji, Binod Kumar, Yonggang Zhang, et al.. (2015). A functional screen identifies miRNAs that inhibit DNA repair and sensitize prostate cancer cells to ionizing radiation. Nucleic Acids Research. 43(8). 4075–4086. 68 indexed citations
13.
Rudra, Arnab, Yonggang Zhang, Jonathan B. Coulter, et al.. (2015). Bromopyridone Nucleotide Analogues, Anoxic Selective Radiosensitizing Agents That Are Incorporated in DNA by Polymerases. The Journal of Organic Chemistry. 80(21). 10675–10685. 6 indexed citations
14.
Mian, Omar Y., Mohamed H. Khattab, Mohammad Hedayati, et al.. (2015). GSTP1 Loss results in accumulation of oxidative DNA base damage and promotes prostate cancer cell survival following exposure to protracted oxidative stress. The Prostate. 76(2). 199–206. 41 indexed citations
15.
Coulter, Jonathan B., Cliona O’Driscoll, & Joseph Bressler. (2013). Hydroquinone Increases 5-Hydroxymethylcytosine Formation through Ten Eleven Translocation 1 (TET1) 5-Methylcytosine Dioxygenase. Journal of Biological Chemistry. 288(40). 28792–28800. 77 indexed citations
16.
O’Driscoll, Cliona, Jonathan B. Coulter, & Joseph Bressler. (2012). Induction of a trophoblast-like phenotype by hydralazine in the p19 embryonic carcinoma cell line. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1833(3). 460–467. 7 indexed citations
17.
Orita, Hajime, Jonathan B. Coulter, Ellen Tully, et al.. (2010). High levels of fatty acid synthase expression in esophageal cancers represent a potential target for therapy. Cancer Biology & Therapy. 10(6). 549–554. 25 indexed citations
18.
Orita, Hajime, Jonathan B. Coulter, Ellen Tully, Francis P. Kuhajda, & Edward Gabrielson. (2008). Inhibiting Fatty Acid Synthase for Chemoprevention of Chemically Induced Lung Tumors. Clinical Cancer Research. 14(8). 2458–2464. 76 indexed citations
19.
Singh, Anju, Swetlana Boldin‐Adamsky, Rajesh K. Thimmulappa, et al.. (2008). RNAi -Mediated Silencing of Nuclear Factor Erythroid-2–Related Factor 2 Gene Expression in Non–Small Cell Lung Cancer Inhibits Tumor Growth and Increases Efficacy of Chemotherapy. Cancer Research. 68(19). 7975–7984. 313 indexed citations
20.
Li, Dapeng, Hajime Orita, Jonathan B. Coulter, et al.. (2007). Coix seed extract, A commonly used treatment for cancer in china, inhibits NFκB and protein kinase C signaling. Cancer Biology & Therapy. 6(12). 2005–2011. 70 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026