Jon P. Connelly

2.7k total citations · 1 hit paper
29 papers, 1.4k citations indexed

About

Jon P. Connelly is a scholar working on Molecular Biology, Immunology and Genetics. According to data from OpenAlex, Jon P. Connelly has authored 29 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Molecular Biology, 7 papers in Immunology and 5 papers in Genetics. Recurrent topics in Jon P. Connelly's work include CRISPR and Genetic Engineering (12 papers), Epigenetics and DNA Methylation (5 papers) and RNA Interference and Gene Delivery (5 papers). Jon P. Connelly is often cited by papers focused on CRISPR and Genetic Engineering (12 papers), Epigenetics and DNA Methylation (5 papers) and RNA Interference and Gene Delivery (5 papers). Jon P. Connelly collaborates with scholars based in United States, United Kingdom and United Arab Emirates. Jon P. Connelly's co-authors include Shondra M. Pruett‐Miller, Matthew H. Porteus, Colin P. Florian, Monica F. Sentmanat, Samuel T. Peters, Jenny C. Barker, Matthew L. Hirsch, Brian L. Ellis, Robert J. Steininger and Raghvendra Mall and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Jon P. Connelly

27 papers receiving 1.4k citations

Hit Papers

ZBP1-dependent inflammatory cell death, PANoptosis, and c... 2022 2026 2023 2024 2022 50 100 150 200

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jon P. Connelly United States 15 1.1k 369 264 144 102 29 1.4k
Dana L. Jackson United States 16 1.5k 1.3× 200 0.5× 244 0.9× 173 1.2× 236 2.3× 27 1.8k
Gaël A. Millot France 20 1.0k 0.9× 443 1.2× 232 0.9× 188 1.3× 99 1.0× 36 1.5k
H Ariga Japan 18 905 0.8× 286 0.8× 190 0.7× 198 1.4× 86 0.8× 39 1.3k
Abel Acosta‐Sanchez Belgium 7 882 0.8× 560 1.5× 152 0.6× 192 1.3× 33 0.3× 8 1.2k
Ingrid Ehrmann United Kingdom 23 1.2k 1.1× 552 1.5× 341 1.3× 60 0.4× 146 1.4× 32 1.8k
Hélène Strick‐Marchand France 21 976 0.9× 229 0.6× 460 1.7× 236 1.6× 83 0.8× 34 2.1k
T. Wilson United States 16 944 0.9× 194 0.5× 667 2.5× 282 2.0× 76 0.7× 30 1.7k
Jiazhi Hu China 21 1.9k 1.8× 349 0.9× 354 1.3× 255 1.8× 107 1.0× 40 2.2k
Andrea Calabria Italy 17 1.1k 1.0× 736 2.0× 233 0.9× 400 2.8× 92 0.9× 35 1.6k
Jacques Bollekens United States 14 983 0.9× 275 0.7× 265 1.0× 141 1.0× 130 1.3× 18 1.5k

Countries citing papers authored by Jon P. Connelly

Since Specialization
Citations

This map shows the geographic impact of Jon P. Connelly's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jon P. Connelly with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jon P. Connelly more than expected).

Fields of papers citing papers by Jon P. Connelly

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jon P. Connelly. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jon P. Connelly. The network helps show where Jon P. Connelly may publish in the future.

Co-authorship network of co-authors of Jon P. Connelly

This figure shows the co-authorship network connecting the top 25 collaborators of Jon P. Connelly. A scholar is included among the top collaborators of Jon P. Connelly based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jon P. Connelly. Jon P. Connelly is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Xu, Yuanlin, et al.. (2025). Megabase-scale loss of heterozygosity provoked by CRISPR-Cas9 DNA double-strand breaks. Molecular Cell. 85(22). 4119–4137.e10. 1 indexed citations
3.
Singh, Shivendra V., Qiong Wu, Hongjian Jin, et al.. (2025). The context-dependent epigenetic and organogenesis programs determine 3D vs. 2D cellular fitness of MYC-driven murine liver cancer cells. eLife. 14. 1 indexed citations
4.
Malireddi, R. K. Subbarao, et al.. (2024). The protein phosphatase PP6 promotes RIPK1-dependent PANoptosis. BMC Biology. 22(1). 122–122. 20 indexed citations
5.
Connelly, Jon P., et al.. (2024). Generation of iPSC lines and isogenic gene-corrected lines from two individuals with RPS19-mutated Diamond-Blackfan anemia syndrome. Stem Cell Research. 79. 103479–103479. 2 indexed citations
6.
Malireddi, R. K. Subbarao, et al.. (2023). Inflammatory cell death, PANoptosis, screen identifies host factors in coronavirus innate immune response as therapeutic targets. Communications Biology. 6(1). 1071–1071. 7 indexed citations
7.
Connelly, Jon P., et al.. (2023). Short tandem repeat profiling via next-generation sequencing for cell line authentication. Disease Models & Mechanisms. 16(10). 2 indexed citations
8.
Narina, Shilpa, Jon P. Connelly, & Shondra M. Pruett‐Miller. (2023). High-Throughput Analysis of CRISPR-Cas9 Editing Outcomes in Cell and Animal Models Using CRIS.py. Methods in molecular biology. 2631. 155–182. 11 indexed citations
9.
Karki, Rajendra, SangJoon Lee, Raghvendra Mall, et al.. (2022). ZBP1-dependent inflammatory cell death, PANoptosis, and cytokine storm disrupt IFN therapeutic efficacy during coronavirus infection. Science Immunology. 7(74). eabo6294–eabo6294. 209 indexed citations breakdown →
10.
Gonzalez-Pena, Veronica, Sivaraman Natarajan, Yuntao Xia, et al.. (2021). Accurate genomic variant detection in single cells with primary template-directed amplification. Proceedings of the National Academy of Sciences. 118(24). 87 indexed citations
11.
Diedrich, Jonathan D., Qian Dong, Daniel C. Ferguson, et al.. (2021). Profiling chromatin accessibility in pediatric acute lymphoblastic leukemia identifies subtype-specific chromatin landscapes and gene regulatory networks. Leukemia. 35(11). 3078–3091. 10 indexed citations
12.
Hao, Xiaolei, Beisi Xu, Jeremy Chase Crawford, et al.. (2021). Foxp3 enhancers synergize to maximize regulatory T cell suppressive capacity. The Journal of Experimental Medicine. 218(8). 10 indexed citations
13.
Budhraja, Amit, J.A. Lynch, Kathryn G. Roberts, et al.. (2020). The Heme-Regulated Inhibitor Pathway Modulates Susceptibility of Poor Prognosis B-Lineage Acute Leukemia to BH3-Mimetics. Molecular Cancer Research. 19(4). 636–650. 12 indexed citations
14.
Tacer, Klementina Fon, Melissa J. Oatley, Tessa Lord, et al.. (2019). MAGE cancer-testis antigens protect the mammalian germline under environmental stress. Science Advances. 5(5). eaav4832–eaav4832. 60 indexed citations
15.
Connelly, Jon P. & Shondra M. Pruett‐Miller. (2019). CRIS.py: A Versatile and High-throughput Analysis Program for CRISPR-based Genome Editing. Scientific Reports. 9(1). 4194–4194. 97 indexed citations
16.
Sentmanat, Monica F., Samuel T. Peters, Colin P. Florian, Jon P. Connelly, & Shondra M. Pruett‐Miller. (2018). A Survey of Validation Strategies for CRISPR-Cas9 Editing. Scientific Reports. 8(1). 888–888. 236 indexed citations
17.
Wen, Yahong, Grace Liao, Thomas Pritchard, et al.. (2017). A stable but reversible integrated surrogate reporter for assaying CRISPR/Cas9-stimulated homology-directed repair. Journal of Biological Chemistry. 292(15). 6148–6162. 12 indexed citations
18.
Connelly, Jon P., Erika M. Kwon, Yongxing Gao, et al.. (2014). Targeted correction of RUNX1 mutation in FPD patient-specific induced pluripotent stem cells rescues megakaryopoietic defects. Blood. 124(12). 1926–1930. 54 indexed citations
19.
20.
Porteus, Matthew H., et al.. (2006). A Look to Future Directions in Gene Therapy Research for Monogenic Diseases. PLoS Genetics. 2(9). e133–e133. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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