Jon E. Wergedal

7.3k total citations · 1 hit paper
109 papers, 5.9k citations indexed

About

Jon E. Wergedal is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Orthopedics and Sports Medicine. According to data from OpenAlex, Jon E. Wergedal has authored 109 papers receiving a total of 5.9k indexed citations (citations by other indexed papers that have themselves been cited), including 64 papers in Molecular Biology, 35 papers in Endocrinology, Diabetes and Metabolism and 29 papers in Orthopedics and Sports Medicine. Recurrent topics in Jon E. Wergedal's work include Bone Metabolism and Diseases (31 papers), Bone health and osteoporosis research (26 papers) and Growth Hormone and Insulin-like Growth Factors (21 papers). Jon E. Wergedal is often cited by papers focused on Bone Metabolism and Diseases (31 papers), Bone health and osteoporosis research (26 papers) and Growth Hormone and Insulin-like Growth Factors (21 papers). Jon E. Wergedal collaborates with scholars based in United States, United Kingdom and Canada. Jon E. Wergedal's co-authors include David J. Baylink, Subburaman Mohan, David J. Baylink, Jonathan Farley, K.‐H. William Lau, Donna D. Strong, Thomas A. Linkhart, Matilda H.‐C. Sheng, Christian Kasperk and Chandrasekhar Kesavan and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Jon E. Wergedal

109 papers receiving 5.7k citations

Hit Papers

Fluoride Directly Stimulates Proliferation and Alkaline P... 1983 2026 1997 2011 1983 100 200 300 400
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Fluoride Directly Stimulates Proliferation and Alkaline Phosphatase Activity of Bone-Forming Cells
    1983 499 citations Jonathan Farley, Jon E. Wergedal et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jon E. Wergedal United States 47 2.9k 1.3k 1.2k 1.2k 905 109 5.9k
K.‐H. William Lau United States 43 2.9k 1.0× 777 0.6× 573 0.5× 973 0.8× 937 1.0× 165 5.8k
Thomas A. Linkhart United States 40 3.2k 1.1× 1.6k 1.2× 787 0.6× 708 0.6× 992 1.1× 80 5.1k
Barbara E. Kream United States 41 3.1k 1.1× 1.0k 0.8× 1.3k 1.1× 813 0.7× 1.4k 1.5× 102 6.0k
Hiroshi Kaji Japan 48 3.0k 1.0× 688 0.5× 633 0.5× 1.6k 1.4× 1.7k 1.9× 228 6.8k
Luc Malaval France 41 2.3k 0.8× 759 0.6× 564 0.5× 1.3k 1.1× 1.3k 1.4× 98 6.1k
Jonathan Farley United States 39 1.9k 0.7× 1.2k 0.9× 425 0.3× 1.3k 1.1× 761 0.8× 76 4.4k
Harry C. Blair United States 49 5.3k 1.8× 1.3k 1.0× 1.0k 0.8× 1.6k 1.4× 2.8k 3.1× 181 9.4k
Maria Almeida United States 49 5.2k 1.8× 696 0.5× 1.3k 1.0× 2.1k 1.8× 1.9k 2.1× 86 8.8k
Matthias Priemel Germany 30 2.1k 0.7× 582 0.4× 697 0.6× 1.4k 1.2× 1.1k 1.2× 76 5.6k
Stavroula Kousteni United States 36 3.5k 1.2× 782 0.6× 1.5k 1.2× 1.5k 1.3× 1.9k 2.1× 62 6.6k

Countries citing papers authored by Jon E. Wergedal

Since Specialization
Citations

This map shows the geographic impact of Jon E. Wergedal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jon E. Wergedal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jon E. Wergedal more than expected).

Fields of papers citing papers by Jon E. Wergedal

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jon E. Wergedal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jon E. Wergedal. The network helps show where Jon E. Wergedal may publish in the future.

Co-authorship network of co-authors of Jon E. Wergedal

This figure shows the co-authorship network connecting the top 25 collaborators of Jon E. Wergedal. A scholar is included among the top collaborators of Jon E. Wergedal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jon E. Wergedal. Jon E. Wergedal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wergedal, Jon E., et al.. (2013). Urokinase plasminogen activator gene deficiency inhibits fracture cartilage remodeling. Journal of Bone and Mineral Metabolism. 32(2). 124–135. 17 indexed citations
2.
Yu, Hongrun, Jon E. Wergedal, Yongliang Zhao, & Subburaman Mohan. (2012). Targeted Disruption of TGFBI in Mice Reveals Its Role in Regulating Bone Mass and Bone Size through Periosteal Bone Formation. Calcified Tissue International. 91(1). 81–87. 27 indexed citations
3.
4.
Sheng, Matilda H.‐C., Jon E. Wergedal, Subburaman Mohan, et al.. (2012). Targeted Overexpression of Osteoactivin in Cells of Osteoclastic Lineage Promotes Osteoclastic Resorption and Bone Loss in Mice. PLoS ONE. 7(4). e35280–e35280. 24 indexed citations
5.
Hall, Susan L., Shin‐Tai Chen, Jon E. Wergedal, et al.. (2011). Stem cell antigen‐1 positive cell‐based systemic human growth hormone gene transfer strategy increases endosteal bone resorption and bone loss in mice. The Journal of Gene Medicine. 13(2). 77–88. 4 indexed citations
6.
7.
Rundle, Charles H., Xiaoguang Wang, Matilda H.‐C. Sheng, et al.. (2008). Bax deficiency in mice increases cartilage production during fracture repair through a mechanism involving increased chondrocyte proliferation without changes in apoptosis. Bone. 43(5). 880–888. 19 indexed citations
8.
Baylink, David J., et al.. (2007). Identification of mouse Duffy Antigen Receptor for Chemokines (Darc) as a BMD QTL gene. Genome Research. 17(5). 577–585. 44 indexed citations
9.
Kesavan, Chandrasekhar, et al.. (2005). Mechanical loading-induced gene expression and BMD changes are different in two inbred mouse strains. Journal of Applied Physiology. 99(5). 1951–1957. 50 indexed citations
10.
Mohan, Subburaman, Anil Kapoor, Zhang Zhang, et al.. (2005). Spontaneous Fractures in the Mouse Mutant sfx Are Caused by Deletion of the Gulonolactone Oxidase Gene, Causing Vitamin C Deficiency. Journal of Bone and Mineral Research. 20(9). 1597–1610. 46 indexed citations
11.
Srivastava, Apurva K., Sanjay Kapur, S. Murali Mohan, et al.. (2005). Identification of Novel Genetic Loci for Bone Size and Mechanosensitivity in an ENU Mutant Exhibiting Decreased Bone Size. Journal of Bone and Mineral Research. 20(6). 1041–1050. 14 indexed citations
12.
Linkhart, Thomas A., Shin‐Tai Chen, Hairong Peng, et al.. (2004). Local ex vivo gene therapy with bone marrow stromal cells expressing human BMP4 promotes endosteal bone formation in mice. The Journal of Gene Medicine. 6(1). 4–15. 52 indexed citations
13.
Salih, Derviş A., Subburaman Mohan, Yuji Kasukawa, et al.. (2004). Insulin-Like Growth Factor-Binding Protein-5 Induces a Gender-Related Decrease in Bone Mineral Density in Transgenic Mice. Endocrinology. 146(2). 931–940. 60 indexed citations
14.
Wergedal, Jon E., Wendy Balemans, Cesar Libanati, et al.. (2003). Patients with Van Buchem Disease, an Osteosclerotic Genetic Disease, Have Elevated Bone Formation Markers, Higher Bone Density, and Greater Derived Polar Moment of Inertia than Normal. The Journal of Clinical Endocrinology & Metabolism. 88(12). 5778–5783. 67 indexed citations
15.
Rundle, Charles H., Naohisa Miyakoshi, E. Minaya Ramirez, et al.. (2002). Expression of the Fibroblast Growth Factor Receptor Genes in Fracture Repair. Clinical Orthopaedics and Related Research. 403(403). 253–263. 27 indexed citations
16.
17.
Mohan, Subburaman, et al.. (1992). Studies on regulation of insulin-like growth factor binding protein (IGFBP)-3 and IGFBP-4 production in human bone cells. European Journal of Endocrinology. 127(6). 555–564. 48 indexed citations
18.
19.
Lau, K.‐H. William, et al.. (1990). Monolayer cultures of normal human bone cells contain multiple subpopulations of alkaline phosphatase positive cells. Calcified Tissue International. 47(5). 276–283. 34 indexed citations
20.
Baylink, David J., et al.. (1974). Systemic factors in alveolar bone loss. Journal of Prosthetic Dentistry. 31(5). 486–505. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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