John W. Chamberlain

703 total citations
31 papers, 516 citations indexed

About

John W. Chamberlain is a scholar working on Immunology, Molecular Biology and Surgery. According to data from OpenAlex, John W. Chamberlain has authored 31 papers receiving a total of 516 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 8 papers in Molecular Biology and 6 papers in Surgery. Recurrent topics in John W. Chamberlain's work include T-cell and B-cell Immunology (12 papers), Immunotherapy and Immune Responses (10 papers) and Immune Cell Function and Interaction (9 papers). John W. Chamberlain is often cited by papers focused on T-cell and B-cell Immunology (12 papers), Immunotherapy and Immune Responses (10 papers) and Immune Cell Function and Interaction (9 papers). John W. Chamberlain collaborates with scholars based in Canada, United States and United Kingdom. John W. Chamberlain's co-authors include Jane R. Parnes, Rho Hyun Seong, Gordon F. Vawter, Sherman M. Weissman, Alan D. Perlmutter, Roger Hewson, Rumina Hasan, Kevin R. Bewley, Bushra Jamil and Subinay Ganguly and has published in prestigious journals such as Nature, New England Journal of Medicine and Nucleic Acids Research.

In The Last Decade

John W. Chamberlain

31 papers receiving 481 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John W. Chamberlain Canada 12 209 127 97 96 88 31 516
Kenji Tadokoro Japan 11 409 2.0× 53 0.4× 37 0.4× 24 0.3× 45 0.5× 17 561
J L Sullivan United States 6 90 0.4× 86 0.7× 24 0.2× 63 0.7× 87 1.0× 6 338
Marc Andrien Belgium 13 257 1.2× 103 0.8× 21 0.2× 188 2.0× 114 1.3× 41 596
Valentina Caputo Italy 10 107 0.5× 74 0.6× 26 0.3× 37 0.4× 52 0.6× 27 397
Karen James United States 13 126 0.6× 96 0.8× 22 0.2× 61 0.6× 19 0.2× 30 466
Georg Kienzle Germany 5 224 1.1× 34 0.3× 153 1.6× 26 0.3× 25 0.3× 6 394
Guido Hegasy Germany 9 230 1.1× 58 0.5× 61 0.6× 67 0.7× 97 1.1× 12 564
Keisuke Hagiwara Japan 12 65 0.3× 88 0.7× 57 0.6× 91 0.9× 38 0.4× 30 419
Vojislav Jovanović Singapore 10 227 1.1× 110 0.9× 155 1.6× 47 0.5× 133 1.5× 14 591
Katie Matthews United States 13 254 1.2× 116 0.9× 34 0.4× 94 1.0× 87 1.0× 20 617

Countries citing papers authored by John W. Chamberlain

Since Specialization
Citations

This map shows the geographic impact of John W. Chamberlain's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John W. Chamberlain with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John W. Chamberlain more than expected).

Fields of papers citing papers by John W. Chamberlain

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John W. Chamberlain. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John W. Chamberlain. The network helps show where John W. Chamberlain may publish in the future.

Co-authorship network of co-authors of John W. Chamberlain

This figure shows the co-authorship network connecting the top 25 collaborators of John W. Chamberlain. A scholar is included among the top collaborators of John W. Chamberlain based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John W. Chamberlain. John W. Chamberlain is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Benoît, Loralyn A., John P. Shannon, John W. Chamberlain, & Richard G. Miller. (2005). Influence of Xenogeneic β2-Microglobulin on Functional Recognition of H-2Kb by the NK Cell Inhibitory Receptor Ly49C. The Journal of Immunology. 175(6). 3542–3553. 9 indexed citations
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Yang, Yongping, Colin McKerlie, Steven H. Borenstein, et al.. (2002). Transgenic Expression in Mouse Lung Reveals Distinct Biological Roles for the Adenovirus Type 5 E1A 243- and 289-Amino-Acid Proteins. Journal of Virology. 76(17). 8910–8919. 9 indexed citations
6.
Borenstein, Steven H., et al.. (2001). EXTRATHYMIC DELETION OF CD8+ ALLOREACTIVE T CELLS IN A TRANSGENIC T CELL RECEPTOR MODEL OF NEONATAL TOLERANCE1. Transplantation. 72(11). 1807–1816. 6 indexed citations
7.
Borenstein, Steven H., et al.. (2000). CD8+ T Cells Are Necessary for Recognition of Allelic, But Not Locus-Mismatched or Xeno-, HLA Class I Transplantation Antigens. The Journal of Immunology. 165(5). 2341–2353. 15 indexed citations
8.
Kung, Sam K. P., et al.. (1998). NK Cells from Human MHC Class I (HLA-B) Transgenic Mice Do Not Mediate Hybrid Resistance Killing Against Parental Nontransgenic cells. The Journal of Immunology. 160(2). 674–680. 8 indexed citations
9.
Zhang, Xiaoli, Rho Hyun Seong, Mani Larijani, et al.. (1998). Distinct Stage-Specific cis-Active Transcriptional Mechanisms Control Expression of T Cell Coreceptor CD8α at Double- and Single-Positive Stages of Thymic Development. The Journal of Immunology. 161(5). 2254–2266. 20 indexed citations
10.
Zhang, Xiaoli, Henry H. Heng, Yang Ye, et al.. (1996). Chromosomal mapping of the second humanCD8B gene locus. Immunogenetics. 43(4). 220–226. 3 indexed citations
11.
Zhang, Xiaoli, Henry H. Heng, Lap‐Chee Tsui, et al.. (1994). Isolation of P1 bacteriophage clones containing large contiguous segments of the human and mouse loci for the T-cell coreceptor molecule CD8. Genetic Analysis Biomolecular Engineering. 11(5-6). 129–139. 4 indexed citations
12.
Seong, Rho Hyun, John W. Chamberlain, & Jane R. Parnes. (1992). Signal for T-cell differentiation to a CD4 cell lineage is delivered by CD4 transmembrane region and/or cytoplasmic tail. Nature. 356(6371). 718–720. 79 indexed citations
13.
Dey, Anup, et al.. (1992). Occupancy of Upstream Regulatory Sites In Vivo Coincides with Major Histocompatibility Complex Class I Gene Expression in Mouse Tissues. Molecular and Cellular Biology. 12(8). 3590–3599. 18 indexed citations
14.
McArthur, James G., Lenore K. Beitel, John W. Chamberlain, & Clifford P. Stanners. (1991). Elements which stimulate gene amplification in mammalian cells: role of recombinogenic sequences/structures and transcriptional activation. Nucleic Acids Research. 19(9). 2477–2484. 14 indexed citations
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Tanaka, Minoru, Ajay Bhargava, Francis S. Collins, et al.. (1990). Expression of Human Globin Genes in Transgenic Mice Carrying the β‐Globin Gene Cluster with a Mutation Causing Gγβ+ Hereditary Persistence of Fetal Hemoglobina. Annals of the New York Academy of Sciences. 612(1). 167–178. 6 indexed citations
17.
Chamberlain, John W., Graham Henderson, Teresa Lam, et al.. (1986). The structure of HSAG-1, a middle repetitive genetic element which elicits a leukemia-related cellular surface antigen. Nucleic Acids Research. 14(8). 3409–3424. 11 indexed citations
18.
Chamberlain, John W.. (1967). Congenital epulis (granular cell myoblastoma). Journal of Pediatric Surgery. 2(2). 158–163. 4 indexed citations
19.
Chamberlain, John W., et al.. (1963). Traumatic Hemobilia. New England Journal of Medicine. 268(11). 565–568. 11 indexed citations
20.
Chamberlain, John W., et al.. (1960). TORSION OF THE APPENDIX TESTIS WITH OBSERVATIONS AS TO ITS ETIOLOGY. PEDIATRICS. 26(4). 611–615. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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