John W. Burch

1.5k total citations · 1 hit paper
17 papers, 1.2k citations indexed

About

John W. Burch is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Pharmacology. According to data from OpenAlex, John W. Burch has authored 17 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cardiology and Cardiovascular Medicine, 5 papers in Molecular Biology and 5 papers in Pharmacology. Recurrent topics in John W. Burch's work include Antiplatelet Therapy and Cardiovascular Diseases (6 papers), Inflammatory mediators and NSAID effects (5 papers) and Eicosanoids and Hypertension Pharmacology (3 papers). John W. Burch is often cited by papers focused on Antiplatelet Therapy and Cardiovascular Diseases (6 papers), Inflammatory mediators and NSAID effects (5 papers) and Eicosanoids and Hypertension Pharmacology (3 papers). John W. Burch collaborates with scholars based in United States, Australia and United Kingdom. John W. Burch's co-authors include Philip W. Majerus, N Stanford, O. Wesley McBride, David B. P. Goodman, Craig A. Dise, Nancy Lewis Baenziger, James A. Delmez, Carol Weerts, Herschel R. Harter and Charles B. Anderson and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

John W. Burch

17 papers receiving 978 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Inhibition of platelet prostaglandin synthetase by oral aspirin.

1978 427 citations John W. Burch, N Stanford et al. Journal of Clinical Investigation profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
John W. Burch United States	11	519	407	202	176	176	17	1.2k
G. Defreyn Belgium	20	684 1.3×	223 0.5×	289 1.4×	288 1.6×	244 1.4×	34	1.7k
E. Seifried Germany	18	367 0.7×	125 0.3×	251 1.2×	153 0.9×	242 1.4×	44	1.9k
Lenore B. Safier United States	10	241 0.5×	196 0.5×	307 1.5×	165 0.9×	67 0.4×	12	1.2k
A. K. Pedersen Denmark	17	543 1.0×	363 0.9×	152 0.8×	118 0.7×	58 0.3×	38	1.1k
J.A. Jakubowski United States	17	410 0.8×	103 0.3×	178 0.9×	230 1.3×	183 1.0×	32	1.3k
Kenneth A. Schwartz United States	22	505 1.0×	185 0.5×	230 1.1×	248 1.4×	204 1.2×	58	1.5k
Gennaro Vecchione Italy	22	430 0.8×	104 0.3×	246 1.2×	194 1.1×	166 0.9×	50	1.6k
M Hanano Japan	23	101 0.2×	142 0.3×	300 1.5×	208 1.2×	113 0.6×	68	1.6k
Michele Nutini Italy	8	218 0.4×	122 0.3×	226 1.1×	152 0.9×	32 0.2×	9	1.0k
Zhongren Ding China	18	462 0.9×	106 0.3×	386 1.9×	245 1.4×	182 1.0×	40	1.6k

Countries citing papers authored by John W. Burch

Since Specialization

Citations

This map shows the geographic impact of John W. Burch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John W. Burch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John W. Burch more than expected).

Fields of papers citing papers by John W. Burch

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John W. Burch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John W. Burch. The network helps show where John W. Burch may publish in the future.

Co-authorship network of co-authors of John W. Burch

This figure shows the co-authorship network connecting the top 25 collaborators of John W. Burch. A scholar is included among the top collaborators of John W. Burch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John W. Burch. John W. Burch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Mansergh, Ian, et al.. (2022). Something went missing: cessation of Traditional Owner land management and rapid mammalian population collapses in the semi-arid region of the Murray–Darling Basin, southeastern Australia. Proceedings of the Royal Society of Victoria. 134(1). 45–84. 4 indexed citations

Reik, Rita A., et al.. (2006). Unique donor‐suitability issues. Vox Sanguinis. 90(4). 255–264. 3 indexed citations

Burch, John W., David Mair, Geralyn M. Meny, et al.. (2005). The risk of posterior subcapsular cataracts in granulocyte donors. Transfusion. 45(11). 1701–1708. 10 indexed citations

Burch, John W., et al.. (1994). Modification of glutathione content in platelet concentrates by the use of acivicin. Transfusion. 34(5). 421–426. 4 indexed citations

Burch, John W., et al.. (1990). Glutathione disulfide production during arachidonic acid oxygenation in human platelets. Prostaglandins. 39(2). 123–134. 20 indexed citations

Burch, John W., et al.. (1987). Alterations in glutathione during storage of human platelet concentrates. Transfusion. 27(4). 342–346. 21 indexed citations

Martin, Jack L., John R. Wilson, John W. Burch, et al.. (1983). Effect of atrial pacing on intracoronary thromboxane production in coronary artery disease. Journal of the American College of Cardiology. 1(5). 1194–1200. 3 indexed citations

Burch, John W. & Roberta J. Lamb. (1983). Sodium arachidonate induced platelet aggregation is independent of secreted adenosine diphosphate. Thrombosis Research. 31(5). 747–758. 5 indexed citations

Dise, Craig A., John W. Burch, & David B. P. Goodman. (1982). Direct interaction of mepacrine with erythrocyte and platelet membrane phospholipid.. Journal of Biological Chemistry. 257(9). 4701–4704. 112 indexed citations

10.

Bennett, Joel, Gaston Vilaire, & John W. Burch. (1981). A role for prostaglandins and thromboxanes in the exposure of platelet fibrinogen receptors.. Journal of Clinical Investigation. 68(4). 981–987. 65 indexed citations

11.

Burch, John W. & Bonnie L. Blazer‐Yost. (1981). Acetylation of albumin by low doses of aspirin. Thrombosis Research. 23(4-5). 447–452. 14 indexed citations

12.

Harter, Herschel R., John W. Burch, Philip W. Majerus, et al.. (1979). Prevention of Thrombosis in Patients on Hemodialysis by Low-Dose Aspirin. New England Journal of Medicine. 301(11). 577–579. 183 indexed citations

13.

Burch, John W. & Philip W. Majerus. (1979). The role of prostaglandins in platelet function.. PubMed. 16(3). 196–207. 59 indexed citations

14.

Burch, John W., Nancy Lewis Baenziger, N Stanford, & Philip W. Majerus. (1978). Sensitivity of fatty acid cyclooxygenase from human aorta to acetylation by aspirin.. Proceedings of the National Academy of Sciences. 75(10). 5181–5184. 119 indexed citations

15.

McBride, O. Wesley, John W. Burch, & F.H. Ruddle. (1978). Cotransfer of thymidine kinase and galactokinase genes by chromosome-mediated gene transfer.. Proceedings of the National Academy of Sciences. 75(2). 914–918. 47 indexed citations

16.

Burch, John W., N Stanford, & Philip W. Majerus. (1978). Inhibition of platelet prostaglandin synthetase by oral aspirin.. Journal of Clinical Investigation. 61(2). 314–319. 427 indexed citations breakdown →

17.

Burch, John W. & O. Wesley McBride. (1975). Human gene expression in rodent cells after uptake of isolated metaphase chromosomes.. Proceedings of the National Academy of Sciences. 72(5). 1797–1801. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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