John W. Bodnar

816 total citations
23 papers, 664 citations indexed

About

John W. Bodnar is a scholar working on Molecular Biology, Genetics and Infectious Diseases. According to data from OpenAlex, John W. Bodnar has authored 23 papers receiving a total of 664 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 12 papers in Genetics and 7 papers in Infectious Diseases. Recurrent topics in John W. Bodnar's work include Virus-based gene therapy research (8 papers), Bacteriophages and microbial interactions (7 papers) and Genomics and Chromatin Dynamics (6 papers). John W. Bodnar is often cited by papers focused on Virus-based gene therapy research (8 papers), Bacteriophages and microbial interactions (7 papers) and Genomics and Chromatin Dynamics (6 papers). John W. Bodnar collaborates with scholars based in United States, United Kingdom and Russia. John W. Bodnar's co-authors include David C. Ward, George D. Pearson, Elizabeth Fox, Phillip T. Moen, J L Kolman, Shirley Jankelevich, George Miller, David H. Coombs, Clare Bergson and Daryl E. Warder and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and The Journal of Chemical Physics.

In The Last Decade

John W. Bodnar

23 papers receiving 623 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John W. Bodnar United States 15 469 286 126 81 66 23 664
Philip A. Jennings Australia 10 583 1.2× 251 0.9× 71 0.6× 28 0.3× 127 1.9× 15 714
Zvee Gilead United States 11 351 0.7× 357 1.2× 85 0.7× 116 1.4× 56 0.8× 23 522
Mervyn G. Smith New Zealand 10 499 1.1× 355 1.2× 65 0.5× 81 1.0× 62 0.9× 14 661
K W Berry United States 7 427 0.9× 198 0.7× 66 0.5× 67 0.8× 16 0.2× 8 527
E.M. Atkinson United Kingdom 9 250 0.5× 248 0.9× 53 0.4× 49 0.6× 59 0.9× 17 438
Richard P. Price United States 7 471 1.0× 164 0.6× 40 0.3× 33 0.4× 28 0.4× 8 592
Irving L. Miller United States 11 433 0.9× 527 1.8× 139 1.1× 109 1.3× 121 1.8× 15 664
Susan C. Milburn United States 10 1.3k 2.8× 191 0.7× 169 1.3× 39 0.5× 52 0.8× 12 1.6k
H. Subak-Sharpe United Kingdom 12 504 1.1× 124 0.4× 54 0.4× 87 1.1× 142 2.2× 15 722
Lester Carter United Kingdom 6 541 1.2× 107 0.4× 202 1.6× 43 0.5× 46 0.7× 11 773

Countries citing papers authored by John W. Bodnar

Since Specialization
Citations

This map shows the geographic impact of John W. Bodnar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John W. Bodnar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John W. Bodnar more than expected).

Fields of papers citing papers by John W. Bodnar

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John W. Bodnar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John W. Bodnar. The network helps show where John W. Bodnar may publish in the future.

Co-authorship network of co-authors of John W. Bodnar

This figure shows the co-authorship network connecting the top 25 collaborators of John W. Bodnar. A scholar is included among the top collaborators of John W. Bodnar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John W. Bodnar. John W. Bodnar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bodnar, John W. & Margaret K. Bradley. (2001). Programming the Drosophila Embryo 2: From Genotype to Phenotype. Cell Biochemistry and Biophysics. 34(2). 153–190. 10 indexed citations
2.
Bodnar, John W., et al.. (1997). Deciphering the Language of the Genome. Journal of Theoretical Biology. 189(2). 183–193. 8 indexed citations
3.
Bodnar, John W.. (1997). Programming theDrosophilaEmbryo. Journal of Theoretical Biology. 188(4). 391–445. 38 indexed citations
4.
Bodnar, John W., et al.. (1996). The Emergence of a Command Network. Naval War College review. 49(4). 8. 1 indexed citations
5.
Bodnar, John W. & Margaret K. Bradley. (1996). A Chromatin Switch. Journal of Theoretical Biology. 183(1). 1–7. 9 indexed citations
6.
Jankelevich, Shirley, J L Kolman, John W. Bodnar, & George Miller. (1992). A nuclear matrix attachment region organizes the Epstein-Barr viral plasmid in Raji cells into a single DNA domain.. The EMBO Journal. 11(3). 1165–1176. 94 indexed citations
7.
Moen, Phillip T., Elizabeth Fox, & John W. Bodnar. (1990). Adenovirus and minute virus of mice DNAs are localized at the nuclear periphery. Nucleic Acids Research. 18(3). 513–520. 19 indexed citations
8.
Fox, Elizabeth, Phillip T. Moen, & John W. Bodnar. (1990). Replication of minute virus of mice dna in adenovirus-infected or adenovirus-transformed cells. Virology. 176(2). 403–412. 21 indexed citations
9.
Bodnar, John W., et al.. (1989). The domain model for eukaryotic DNA organization 2: A molecular basis for constraints on development and evolution. Journal of Theoretical Biology. 137(3). 281–320. 9 indexed citations
10.
Bodnar, John W.. (1989). Sequence organization in regulatory regions of DNA of minute virus of mice. Virus Genes. 2(2). 167–182. 15 indexed citations
11.
Moen, Phillip T., et al.. (1989). Interactions of minute virus of mice and adenovirus with host nucleoli. Journal of Virology. 63(9). 3651–3660. 55 indexed citations
12.
Bodnar, John W., et al.. (1989). The terminal regions of adenovirus and minute virus of mice DNAs are preferentially associated with the nuclear matrix in infected cells. Journal of Virology. 63(10). 4344–4353. 39 indexed citations
13.
Bodnar, John W.. (1988). A domain model for eukaryotic DNA organization: A molecular basis for cell differentiation and chromosome evolution. Journal of Theoretical Biology. 132(4). 479–507. 84 indexed citations
14.
Bodnar, John W. & David C. Ward. (1987). Highly recurring sequence elements identified in eukaryotic DNAs by computer analysis are often homologous to regulatory sequences or protein binding sites. Nucleic Acids Research. 15(4). 1835–1851. 14 indexed citations
15.
Chow, M, et al.. (1986). Identification and characterization of a protein covalently bound to DNA of minute virus of mice. Journal of Virology. 57(3). 1094–1104. 28 indexed citations
16.
Bodnar, John W., et al.. (1983). Effect of nucleotide analogs on the cleavage of DNA by the restriction enzymes AluI, DdeI, HinfI, RsaI, and TaqI.. Journal of Biological Chemistry. 258(24). 15206–15213. 58 indexed citations
17.
Bodnar, John W., et al.. (1983). Proteins tightly bound to HeLa cell DNA at nuclear matrix attachment sites.. Molecular and Cellular Biology. 3(9). 1567–1579. 36 indexed citations
18.
Bodnar, John W., Carol Jones, David H. Coombs, George D. Pearson, & David C. Ward. (1983). Proteins Tightly Bound to HeLa Cell DNA at Nuclear Matrix Attachment Sites. Molecular and Cellular Biology. 3(9). 1567–1579. 59 indexed citations
19.
Coombs, David H., et al.. (1979). Detection of Covalent DNA-Protein Complexes: The Adenovirus DNA-Terminal Protein Complex and HeLa DNA-Protein Complexes. Cold Spring Harbor Symposia on Quantitative Biology. 43(0). 741–753. 19 indexed citations
20.
Brănișteanu, Dumitru, et al.. (1968). [Clinical, functional and biochemical manifestations of the early stages of carbon disulfide poisoning].. PubMed. 29(3). 109–20. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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