John T. Butler

1.2k total citations · 1 hit paper
16 papers, 994 citations indexed

About

John T. Butler is a scholar working on Molecular Biology, Immunology and Cancer Research. According to data from OpenAlex, John T. Butler has authored 16 papers receiving a total of 994 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 7 papers in Immunology and 5 papers in Cancer Research. Recurrent topics in John T. Butler's work include Extracellular vesicles in disease (6 papers), Acute Myeloid Leukemia Research (4 papers) and Immune cells in cancer (4 papers). John T. Butler is often cited by papers focused on Extracellular vesicles in disease (6 papers), Acute Myeloid Leukemia Research (4 papers) and Immune cells in cancer (4 papers). John T. Butler collaborates with scholars based in United States, Japan and Israel. John T. Butler's co-authors include Peter Kurre, Sherif Abdelhamed, Jeanne B. Lawrence, Lisa L. Hall, Kelly P. Smith, Terzah M. Horton, Ding-Wen Chen, Ben Doron, Kyriakos A. Kirou and Daniel L. Marks and has published in prestigious journals such as Nucleic Acids Research, Nature Communications and Blood.

In The Last Decade

John T. Butler

16 papers receiving 973 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Free Radicals, Lipid Peroxidation and Cancer

1982 452 citations John T. Butler British Journal of Cancer profile →

Peers

John T. Butler

MB

CR

IMMUN

OC

BIOCH

Zhuoxiao Cao United States

S. Kulkarni United States

Jesús Sánchez‐Yagüe Spain

Ron Pinkus Israel

Marcial Llanillo Spain

Carlos Fernández Spain

Saravanakumar Dhakshinamoorthy United States

Cristina Toaldo Italy

De Lin United States

Chang-Mo Kang South Korea

Zhuoxiao Cao United States

John T. Butler

880 ×1.6

MB

105 ×0.7

CR

265 ×1.8

IMMUN

73 ×0.6

OC

92 ×0.8

BIOCH

Citations per year, relative to John T. Butler John T. Butler (= 1×) peers Zhuoxiao Cao

Countries citing papers authored by John T. Butler

Since Specialization

Citations

This map shows the geographic impact of John T. Butler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John T. Butler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John T. Butler more than expected).

Fields of papers citing papers by John T. Butler

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John T. Butler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John T. Butler. The network helps show where John T. Butler may publish in the future.

Co-authorship network of co-authors of John T. Butler

This figure shows the co-authorship network connecting the top 25 collaborators of John T. Butler. A scholar is included among the top collaborators of John T. Butler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John T. Butler. John T. Butler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

1.

Butler, John T., et al.. (2025). Breaking the Bone Marrow Barrier: Peripheral Blood as a Gateway to Measurable Residual Disease Detection in Acute Myelogenous Leukemia. American Journal of Hematology. 100(4). 638–651. 3 indexed citations

2.

Olson, Brennan, Xinxia Zhu, Mason A. Norgard, et al.. (2021). Lipocalin 2 mediates appetite suppression during pancreatic cancer cachexia. Nature Communications. 12(1). 2057–2057. 74 indexed citations

3.

Abdelhamed, Sherif, John T. Butler, Ding-Wen Chen, et al.. (2021). Rational biomarker development for the early and minimally invasive monitoring of AML. Blood Advances. 5(21). 4515–4520. 9 indexed citations

4.

Butler, John T., et al.. (2020). Extracellular Vesicles and Chemotherapy Resistance in the AML Microenvironment. Frontiers in Oncology. 10. 90–90. 43 indexed citations

5.

Butler, John T. & Peter Kurre. (2019). Transmissible ER stress shapes the leukemic microenvironment. Oncotarget. 10(41). 4080–4082. 3 indexed citations

6.

Abdelhamed, Sherif, John T. Butler, Ben Doron, et al.. (2019). Extracellular vesicles impose quiescence on residual hematopoietic stem cells in the leukemic niche. EMBO Reports. 20(7). e47546–e47546. 43 indexed citations

7.

Butler, John T., Sherif Abdelhamed, Lina Gao, et al.. (2019). Leukemic Stress Targets the mTOR Pathway to Suppress Residual HSC in the BM Microenvironment. Blood. 134(Supplement_1). 3730–3730. 1 indexed citations

8.

Butler, John T., Sherif Abdelhamed, & Peter Kurre. (2018). Extracellular vesicles in the hematopoietic microenvironment. Haematologica. 103(3). 382–394. 74 indexed citations

9.

Doron, Ben, Sherif Abdelhamed, John T. Butler, et al.. (2018). Transmissible ER stress reconfigures the AML bone marrow compartment. Leukemia. 33(4). 918–930. 48 indexed citations

10.

Harada, Akihito, Chandrashekara Mallappa, Seiji Okada, et al.. (2015). Spatial re-organization of myogenic regulatory sequences temporally controls gene expression. Nucleic Acids Research. 43(4). 2008–2021. 31 indexed citations

11.

Benezra, Miriam, Elizabeth Phillips, Derya Tilki, et al.. (2012). Serial monitoring of human systemic and xenograft models of leukemia using a novel vascular disrupting agent. Leukemia. 26(8). 1771–1778. 16 indexed citations

12.

Butler, John T., Lisa L. Hall, Kelly P. Smith, & Jeanne B. Lawrence. (2009). Changing nuclear landscape and unique PML structures during early epigenetic transitions of human embryonic stem cells. Journal of Cellular Biochemistry. 107(4). 609–621. 51 indexed citations

13.

Hall, Lisa L., Meg Byron, John T. Butler, et al.. (2008). X‐inactivation reveals epigenetic anomalies in most hESC but identifies sublines that initiate as expected. Journal of Cellular Physiology. 216(2). 445–452. 91 indexed citations

14.

Kirou, Kyriakos A., et al.. (2000). Induction of Fas Ligand-Mediated Apoptosis by Interferon-α. Clinical Immunology. 95(3). 218–226. 51 indexed citations

15.

Butler, John T.. (1982). Free Radicals, Lipid Peroxidation and Cancer. British Journal of Cancer. 46(5). 832–832. 452 indexed citations breakdown →

16.

Rocklin, Ross E., et al.. (1981). Partial characterization of a lymphoid cell line (Reh) product with leukocyte inhibitory factor (LIF) activity.. The Journal of Immunology. 127(2). 534–539. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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