John Malysz

4.0k total citations · 1 hit paper
63 papers, 3.0k citations indexed

About

John Malysz is a scholar working on Molecular Biology, Sensory Systems and Urology. According to data from OpenAlex, John Malysz has authored 63 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 44 papers in Molecular Biology, 20 papers in Sensory Systems and 16 papers in Urology. Recurrent topics in John Malysz's work include Ion channel regulation and function (27 papers), Ion Channels and Receptors (20 papers) and Nicotinic Acetylcholine Receptors Study (17 papers). John Malysz is often cited by papers focused on Ion channel regulation and function (27 papers), Ion Channels and Receptors (20 papers) and Nicotinic Acetylcholine Receptors Study (17 papers). John Malysz collaborates with scholars based in United States, Canada and United Kingdom. John Malysz's co-authors include Michael Klüppel, Alan Bernstein, H. B. Mikkelsen, Lars Thuneberg, Jan D. Huizinga, Murali Gopalakrishnan, Jens Halvard Grønlien, Clark A. Briggs, Hilde Ween and Kirsten Thorin‐Hagene and has published in prestigious journals such as Nature, Gastroenterology and PLoS ONE.

In The Last Decade

John Malysz

63 papers receiving 3.0k citations

Hit Papers

W/kit gene required for interstitial cells of Cajal and f... 1995 2026 2005 2015 1995 250 500 750 1000
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. W/kit gene required for interstitial cells of Cajal and for intestinal pacemaker activity
    1995 1.2k citations Michael Klüppel, John Malysz et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John Malysz United States 23 1.5k 1.4k 753 542 398 63 3.0k
H. B. Mikkelsen Denmark 25 1.7k 1.1× 841 0.6× 922 1.2× 368 0.7× 521 1.3× 48 2.8k
Kazuhide Horiguchi Japan 25 841 0.6× 859 0.6× 499 0.7× 237 0.4× 326 0.8× 44 2.2k
Maria Simonetta Faussone‐Pellegrini Italy 27 1.4k 0.9× 643 0.5× 1.1k 1.4× 155 0.3× 734 1.8× 68 2.8k
Elizabeth A. H. Beckett Australia 23 823 0.6× 603 0.4× 409 0.5× 278 0.5× 341 0.9× 38 1.7k
Katsuhide Nishi Japan 18 1.0k 0.7× 636 0.5× 661 0.9× 200 0.4× 384 1.0× 77 2.1k
G.M. Makhlouf United States 30 406 0.3× 981 0.7× 564 0.7× 123 0.2× 494 1.2× 54 2.2k
Carlos Barajas‐López Canada 24 379 0.3× 609 0.4× 162 0.2× 254 0.5× 394 1.0× 67 1.8k
C.A. Maggi Italy 34 251 0.2× 1.4k 1.0× 363 0.5× 371 0.7× 990 2.5× 124 3.3k
M. Jill Saffrey United Kingdom 29 622 0.4× 792 0.6× 611 0.8× 40 0.1× 974 2.4× 69 2.8k
Harald Schwörer Germany 21 336 0.2× 338 0.2× 392 0.5× 59 0.1× 273 0.7× 58 1.4k

Countries citing papers authored by John Malysz

Since Specialization
Citations

This map shows the geographic impact of John Malysz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John Malysz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John Malysz more than expected).

Fields of papers citing papers by John Malysz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John Malysz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John Malysz. The network helps show where John Malysz may publish in the future.

Co-authorship network of co-authors of John Malysz

This figure shows the co-authorship network connecting the top 25 collaborators of John Malysz. A scholar is included among the top collaborators of John Malysz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John Malysz. John Malysz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Leo, M. Dennis, et al.. (2021). Age‐dependent decrease in TRPM4 channel expression but not trafficking alters urinary bladder smooth muscle contractility. Physiological Reports. 9(4). e14754–e14754. 4 indexed citations
2.
Malysz, John & Georgi V. Petkov. (2020). Detrusor Smooth Muscle KV7 Channels: Emerging New Regulators of Urinary Bladder Function. Frontiers in Physiology. 11. 1004–1004. 8 indexed citations
3.
Yarotskyy, Viktor, John Malysz, & Georgi V. Petkov. (2019). Extracellular pH and intracellular phosphatidylinositol 4,5-bisphosphate control Cl− currents in guinea pig detrusor smooth muscle cells. American Journal of Physiology-Cell Physiology. 317(6). C1268–C1277. 5 indexed citations
4.
Hristov, Kiril L., et al.. (2016). Novel regulatory mechanism in human urinary bladder: central role of transient receptor potential melastatin 4 channels in detrusor smooth muscle function. American Journal of Physiology-Cell Physiology. 310(7). C600–C611. 20 indexed citations
5.
Malysz, John, et al.. (2015). The Novel KV7.2/KV7.3 Channel Opener ICA-069673 Reveals Subtype-Specific Functional Roles in Guinea Pig Detrusor Smooth Muscle Excitability and Contractility. Journal of Pharmacology and Experimental Therapeutics. 354(3). 290–301. 15 indexed citations
6.
Parajuli, Shankar P., Kiril L. Hristov, Qiuping Cheng, et al.. (2014). Functional link between muscarinic receptors and large-conductance Ca2+-activated K+ channels in freshly isolated human detrusor smooth muscle cells. Pflügers Archiv - European Journal of Physiology. 467(4). 665–675. 11 indexed citations
7.
Parajuli, Shankar P., Kiril L. Hristov, Qiuping Cheng, et al.. (2013). TRPM4 channel: a new player in urinary bladder smooth muscle function in rats. American Journal of Physiology-Renal Physiology. 304(7). F918–F929. 31 indexed citations
8.
Gopalakrishnan, Sujatha M., Jens Halvard Grønlien, John Malysz, et al.. (2011). Functional Characterization and High-Throughput Screening of Positive Allosteric Modulators of α7 Nicotinic Acetylcholine Receptors in IMR-32 Neuroblastoma Cells. Assay and Drug Development Technologies. 9(6). 635–645. 9 indexed citations
9.
Lee, Chih‐Hung, Chang Zhu, John Malysz, et al.. (2011). α4β2 neuronal nicotinic receptor positive allosteric modulation: An approach for improving the therapeutic index of α4β2 nAChR agonists in pain. Biochemical Pharmacology. 82(8). 959–966. 49 indexed citations
10.
Malysz, John, Jens Halvard Grønlien, Daniel B. Timmermann, et al.. (2009). Evaluation of α7 Nicotinic Acetylcholine Receptor Agonists and Positive Allosteric Modulators Using the Parallel Oocyte Electrophysiology Test Station. Assay and Drug Development Technologies. 7(4). 374–390. 24 indexed citations
11.
Malysz, John, Jens Halvard Grønlien, David J. Anderson, et al.. (2009). In Vitro Pharmacological Characterization of a Novel Allosteric Modulator of α7 Neuronal Acetylcholine Receptor, 4-(5-(4-Chlorophenyl)-2-methyl-3-propionyl-1H-pyrrol-1-yl)benzenesulfonamide (A-867744), Exhibiting Unique Pharmacological Profile. Journal of Pharmacology and Experimental Therapeutics. 330(1). 257–267. 54 indexed citations
12.
Li, Jinhe, Richard Harris, Jianguo Ji, et al.. (2009). Role of α7 nicotinic acetylcholine receptors in regulating tumor necrosis factor-α (TNF-α) as Revealed by subtype selective agonists. Biochemical Pharmacology. 78(7). 924–925. 2 indexed citations
13.
Briggs, Clark A., Jens Halvard Grønlien, Peter Curzon, et al.. (2009). Role of channel activation in cognitive enhancement mediated by α7 nicotinic acetylcholine receptors. British Journal of Pharmacology. 158(6). 1486–1494. 58 indexed citations
14.
Malysz, John, A.V. Daza, Karen Kage, et al.. (2008). Characterization of human cannabinoid CB2 receptor coupled to chimeric Gαqi5 and Gαqo5 proteins. European Journal of Pharmacology. 603(1-3). 12–21. 12 indexed citations
15.
Malysz, John, Gianrico Farrugia, Yijun Ou, et al.. (2002). The Kv2.2 α Subunit Contributes to Delayed Rectifier K+ Currents in Myocytes From Rabbit Corpus Cavernosum. Journal of Andrology. 23(6). 899–910. 8 indexed citations
16.
Rich, Adam, Menachem Hanani, Leonid G. Ermilov, et al.. (2002). Physiological study of interstitial cells of Cajal identified by vital staining. Neurogastroenterology & Motility. 14(2). 189–196. 16 indexed citations
17.
Malysz, John, et al.. (1998). Interstitial cells of Cajal direct normal propulsive contractile activity in the mouse small intestine. Gastroenterology. 114(4). 724–736. 205 indexed citations
18.
Klüppel, Michael, Jan D. Huizinga, John Malysz, & Alan Bernstein. (1998). Developmental origin andkit-dependent development of the interstitial cells of cajal in the mammalian small intestine. Developmental Dynamics. 211(1). 60–71. 197 indexed citations
19.
Klüppel, Michael, Jan D. Huizinga, John Malysz, & Alan Bernstein. (1998). Developmental origin and kit‐dependent development of the interstitial cells of cajal in the mammalian small intestine. Developmental Dynamics. 211(1). 60–71. 3 indexed citations
20.
Malysz, John, et al.. (1995). Generation of slow wave type action potentials in the mouse small intestine involves a non-L-type calcium channel. Canadian Journal of Physiology and Pharmacology. 73(10). 1502–1511. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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