John G. Compton

9.2k citations

54 papers · 3.9k · 1 hit paper · h-index 30

Impact in

Cell Biology top 0.5%
- Skin and Cellular Biology Research
Dermatology top 0.5%
- Dermatology and Skin Diseases

Papers in

Molecular Biology 36
- Plant Reproductive Biology 7
- Wnt/β-catenin signaling in development and cancer 4
- Hedgehog Signaling Pathway Studies 4
- Connexins and lens biology 3
Cell Biology 31
- Skin and Cellular Biology Research 27
- melanin and skin pigmentation 5

Co-authors: Sherri J. Bale (29 shared papers)John J. DiGiovanna (16 shared papers)Peter M. Steinert (10 shared papers)Geraldine R. Rogers (5 shared papers)Bernhard Korge (6 shared papers)Bert W. O’Malley (7 shared papers)William T. Schrader (7 shared papers)Laura Russell (3 shared papers)
Journals: Journal of Investigative Dermatology (11 papers)Nature Genetics (8 papers)Genomics (4 papers)Genetics in Medicine (2 papers)Human Genetics (2 papers)
Partner nations: United States Italy Canada

In The Last Decade

John G. Compton

53 papers receiving 3.8k citations

John G. Compton's Hit Papers

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Loss-of-function mutations in the gene encoding filaggrin cause ichthyosis vulgaris 2006 · 681 citations

Peers

Countries citing papers authored by John G. Compton

Since Specialization

Citations

This map shows the geographic impact of John G. Compton's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John G. Compton with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John G. Compton more than expected).

Fields of papers citing papers by John G. Compton

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John G. Compton. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John G. Compton. The network helps show where John G. Compton may publish in the future.

Co-authors

The 25 scholars most cited alongside John G. Compton, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John G. Compton Line = papers co-authored together John G. Compton links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 54 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	Loss-of-function mutations in the gene encoding filaggrin cause ichthyosis vulgaris Nature Genetics ·Frances J.D. Smith, Alan D. Irvine, Ana Terron-Kwiatkowski, Aileen Sandilands, Linda Campbell, Yiwei Zhao, Haihui Liao, Alan Evans, David Goudie, Sue Lewis‐Jones, Gehan Arseculeratne, Colin S. Munro, Ann Sergeant, Gráinne M. O’Regan, Sherri J. Bale, John G. Compton, John J. DiGiovanna, Richard B. Presland, Philip Fleckman, W.H. Irwin McLean Hit paper breakdown →	2006	681
2	Mutations in the gene for transglutaminase 1 in autosomal recessive lamellar ichthyosis Nature Genetics ·Laura Russell, John J. DiGiovanna, Geraldine R. Rogers, Peter M. Steinert, Nemat Hashem, John G. Compton, Sherri J. Bale	1995	298
3	Mutations in the human connexin gene GJB3 cause erythrokeratodermia variabilis Nature Genetics ·Gabriele Richard, Lisa E. Smith, Regina A. Bailey, Peter Itin, Daniel Hohl, Ervin H. Epstein, John J. DiGiovanna, John G. Compton, Sherri J. Bale	1998	248
4	A leucine→proline mutation in the H1 subdomain of keratin 1 causes epidermolytic hyperkeratosis Cell ·Constantin C. Chipev, Bernhard Korge, Nedialka G. Markova, Sherri J. Bale, John J. DiGiovanna, John G. Compton, Peter M. Steinert	1992	237
5	Functional defects of Cx26 resulting from a heterozygous missense mutation in a family with dominant deaf-mutism and palmoplantar keratoderma Human Genetics ·Gabriela Richard, Thomas W. White, Lisa E. Smith, Regina A. Bailey, John G. Compton, David L. Paul, Sherri J. Bale	1998	217
6	Sjögren–Larsson syndrome is caused by mutations in the fatty aldehyde dehydrogenase gene Nature Genetics ·Vincenzo De Laurenzi, Geraldine R. Rogers, David J. Hamrock, Lyuben N. Marekov, Peter M. Steinert, John G. Compton, Nelli Markova, William B. Rizzo	1996	187
7	Mutation of a new gene causes a unique form of Hermansky–Pudlak syndrome in a genetic isolate of central Puerto Rico Nature Genetics ·Yair Anikster, Marjan Huizing, James G. White, Yuriy O. Shevchenko, Diana L. Fitzpatrick, Jeffrey W. Touchman, John G. Compton, Sherri J. Bale, Richard T. Swank, William A. Gahl, Jorge R. Toro	2001	159
8	Physical Mapping of a Functional Cluster of Epidermal Differentiation Genes on Chromosome 1q21 Genomics ·Armin Volz, Bernhard Korge, John G. Compton, Andreas Ziegler, Peter M. Steinert, Dietmar Mischke	1993	134
9	Allosteric regulation of aspartate transcarbamoylase. Analysis of the structural and functional behavior in terms of a two-state model Biochemistry ·G.J. Howlett, Michael N. Blackburn, John G. Compton, H. K. Schachman	1977	125
10	A Mutation in the V1 End Domain of Keratin 1 in Non-Epidermolytic Palmar-Plantar Keratoderma Journal of Investigative Dermatology ·Virginia Kimonis, Jun-Mo Yang, Sharon Z Doyle, Sherri J. Bale, John G. Compton, John J. DiGiovanna	1994	111
11	DNA sequence preference of the progesterone receptor. Proceedings of the National Academy of Sciences ·John G. Compton, William T. Schrader, Bert W. O’Malley	1983	108
12	A highly conserved lysine residue on the head domain of type II keratins is essential for the attachment of keratin intermediate filaments to the cornified cell envelope through isopeptide crosslinking by transglutaminases Proceedings of the National Academy of Sciences ·Eleonora Candi, Edit Tarcsa, John J. DiGiovanna, John G. Compton, Peter M. Elias, Lyuben N. Marekov, Peter M. Steinert	1998	101
13	Keratin 13 point mutation underlies the hereditary mucosal epithelia disorder white sponge nevus Nature Genetics ·Gabriela Richard, Vincenzo De Laurenzi, Biagio Didona, Sherri J. Bale, John G. Compton	1995	87
14	Linkage of epidermolytic hyperkeratosis to the type II keratin gene cluster on chromosome 12q Nature Genetics ·John G. Compton, John J. DiGiovanna, Sandra Santucci, Kathleen S. Kearns, Christopher I. Amos, Donita L. Abangan, Bernhard Korge, O. Wesley McBride, Peter M. Steinert, Sherri J. Bale	1992	85
15	Linkage of autosomal recessive lamellar ichthyosis to chromosome 14q. PubMed ·Laura Russell, John J. DiGiovanna, Nemat Hashem, John G. Compton, Sherri J. Bale	1994	80
16	Genetic Heterogeneity in Erythrokeratodermia Variabilis: Novel Mutations in the Connexin Gene GJB4 (Cx30.3) and Genotype-Phenotype Correlations Journal of Investigative Dermatology ·Gabriele Richard, Nkecha Brown, Fatima Rouan, Carmen Campanelli, Jouni Uitto, Jan-Gerrit van der Schroeff, Emilia Bijlsma, Lawrence F. Eichenfield, Virginia P. Sybert, Kenneth E. Greer, P Hogan, John G. Compton, Sherri J. Bale, John J. DiGiovanna	2003	77
17	Concurrence between the Molecular Overlap Regions in Keratin Intermediate Filaments and the Locations of Keratin Mutations in Genodermatoses Biochemical and Biophysical Research Communications ·Peter M. Steinert, Jun Yang, Sherri J. Bale, John G. Compton	1993	75
18	Mutations in the H1 and 1A Domains in the Keratin 1 Gene in Epidermolytic Hyperkeratosis Journal of Investigative Dermatology ·Jun-Mo Yang, Constantin C. Chipev, John J. DiGiovanna, Sherri J. Bale, Lyuben N. Marekov, Peter M. Steinert, John G. Compton	1994	65
19	Preferential sites in keratin 10 that are mutated in epidermolytic hyperkeratosis. PubMed ·Constantin C. Chipev, Jun Yang, John J. DiGiovanna, Peter M. Steinert, Lyuben N. Marekov, John G. Compton, Sherri J. Bale	1994	54
20	Genomic Organization and Expression of the Human Fatty Aldehyde Dehydrogenase Gene (FALDH) Genomics ·Geraldine R. Rogers, Nedialka G. Markova, Vincenzo De Laurenzi, William B. Rizzo, John G. Compton	1997	53

About John G. Compton

John G. Compton is a scholar working on Molecular Biology, Cell Biology, Genetics, Urology and Dermatology, having authored 54 papers that have together received 3.9k indexed citations. Recurring topics across this work include Skin and Cellular Biology Research (27 papers), Hair Growth and Disorders (9 papers), Plant Reproductive Biology (7 papers), melanin and skin pigmentation (5 papers), Wnt/β-catenin signaling in development and cancer (4 papers), Hedgehog Signaling Pathway Studies (4 papers), Animal Genetics and Reproduction (3 papers) and Connexins and lens biology (3 papers). The work is most often cited by research in Cell Biology (2.0k citations), Dermatology (834 citations), Immunology and Allergy (437 citations), Urology (434 citations) and Sensory Systems (246 citations). John G. Compton has collaborated with scholars based in United States, Italy and Canada. Frequent co-authors include Sherri J. Bale, John J. DiGiovanna, Peter M. Steinert, Geraldine R. Rogers, Bernhard Korge, Bert W. O’Malley, William T. Schrader, Laura Russell, Nemat Hashem and Lyuben N. Marekov. Their work appears in journals such as Journal of Investigative Dermatology, Nature Genetics, Genomics, Genetics in Medicine and Human Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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