John E. Morales

928 total citations
19 papers, 692 citations indexed

About

John E. Morales is a scholar working on Molecular Biology, Immunology and Allergy and Neurology. According to data from OpenAlex, John E. Morales has authored 19 papers receiving a total of 692 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 5 papers in Immunology and Allergy and 4 papers in Neurology. Recurrent topics in John E. Morales's work include Barrier Structure and Function Studies (4 papers), Cell Adhesion Molecules Research (4 papers) and Axon Guidance and Neuronal Signaling (3 papers). John E. Morales is often cited by papers focused on Barrier Structure and Function Studies (4 papers), Cell Adhesion Molecules Research (4 papers) and Axon Guidance and Neuronal Signaling (3 papers). John E. Morales collaborates with scholars based in United States and South Korea. John E. Morales's co-authors include Rick A. Wetsel, Dachun Wang, Joseph H. McCarty, Qing Yan, Quan Yuan, Daniel G. Calame, Frederick F. Lang, Ganesh Rao, Joseph L. Alcorn and Stacey L. Mueller‐Ortiz and has published in prestigious journals such as Journal of Neuroscience, The Journal of Immunology and PLoS ONE.

In The Last Decade

John E. Morales

18 papers receiving 683 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
John E. Morales United States 13 334 192 153 133 110 19 692
Sherry L. Taylor United States 10 473 1.4× 205 1.1× 165 1.1× 92 0.7× 159 1.4× 15 813
Alexandra Demory Germany 10 323 1.0× 185 1.0× 189 1.2× 52 0.4× 101 0.9× 15 830
David J. Taggart United States 12 238 0.7× 230 1.2× 148 1.0× 158 1.2× 56 0.5× 22 602
Nicolas Gengenbacher Germany 8 306 0.9× 194 1.0× 99 0.6× 67 0.5× 29 0.3× 10 629
Noboru Ando Japan 14 333 1.0× 240 1.3× 68 0.4× 178 1.3× 99 0.9× 25 752
Hideo Takeshima Japan 13 219 0.7× 166 0.9× 137 0.9× 48 0.4× 113 1.0× 18 512
Dennis Pfaff Switzerland 18 494 1.5× 141 0.7× 109 0.7× 53 0.4× 53 0.5× 26 828
Michela Corsini Italy 17 454 1.4× 89 0.5× 161 1.1× 61 0.5× 37 0.3× 41 753
Andreas Hippe Germany 7 216 0.6× 127 0.7× 519 3.4× 54 0.4× 90 0.8× 8 965
Michael Faibish United States 8 532 1.6× 220 1.1× 187 1.2× 45 0.3× 93 0.8× 10 701

Countries citing papers authored by John E. Morales

Since Specialization
Citations

This map shows the geographic impact of John E. Morales's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John E. Morales with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John E. Morales more than expected).

Fields of papers citing papers by John E. Morales

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John E. Morales. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John E. Morales. The network helps show where John E. Morales may publish in the future.

Co-authorship network of co-authors of John E. Morales

This figure shows the co-authorship network connecting the top 25 collaborators of John E. Morales. A scholar is included among the top collaborators of John E. Morales based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with John E. Morales. John E. Morales is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
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Morales, John E., Xiaofeng Zheng, Zhihua Chen, et al.. (2023). Glial Cell Adhesion Molecule (GlialCAM) Determines Proliferative versus Invasive Cell States in Glioblastoma. Journal of Neuroscience. 43(47). 8043–8057. 3 indexed citations
3.
Chen, Zhihua, et al.. (2023). The alpha7 integrin subunit in astrocytes promotes endothelial blood–brain barrier integrity. Development. 150(6). 12 indexed citations
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Chen, Zhihua, John E. Morales, Naze G. Avci, et al.. (2020). The vascular endothelial cell-expressed prion protein Prnd/Doppel promotes angiogenesis and blood-brain barrier development. Development. 147(18). 13 indexed citations
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Chen, Zhihua, John E. Morales, Paola A. Guerrero, Huandong Sun, & Joseph H. McCarty. (2018). PTPN12/PTP-PEST Regulates Phosphorylation-Dependent Ubiquitination and Stability of Focal Adhesion Substrates in Invasive Glioblastoma Cells. Cancer Research. 78(14). 3809–3822. 25 indexed citations
9.
Guerrero, Paola A., Jeremy H. Tchaicha, Z. Chen, et al.. (2017). Glioblastoma stem cells exploit the αvβ8 integrin-TGFβ1 signaling axis to drive tumor initiation and progression. Oncogene. 36(47). 6568–6580. 48 indexed citations
10.
Guerrero, Paola A., et al.. (2017). Neuropilin-1 modulates TGFβ signaling to drive glioblastoma growth and recurrence after anti-angiogenic therapy. PLoS ONE. 12(9). e0185065–e0185065. 36 indexed citations
11.
Cheerathodi, Mujeeburahiman, Naze G. Avci, Paola A. Guerrero, et al.. (2016). The Cytoskeletal Adapter Protein Spinophilin Regulates Invadopodia Dynamics and Tumor Cell Invasion in Glioblastoma. Molecular Cancer Research. 14(12). 1277–1287. 17 indexed citations
12.
Wang, Dachun, Quan Yuan, Qing Yan, John E. Morales, & Rick A. Wetsel. (2015). Targeted Disruption of the β2-Microglobulin Gene Minimizes the Immunogenicity of Human Embryonic Stem Cells. Stem Cells Translational Medicine. 4(10). 1234–1245. 146 indexed citations
13.
Clements, Thomas P., John E. Morales, Hye Shin Lee, et al.. (2015). Neuropilin-1 balances β8 integrin-activated TGFβ signaling to control sprouting angiogenesis in the brain. Development. 142(24). 4363–73. 59 indexed citations
14.
Mueller‐Ortiz, Stacey L., John E. Morales, & Rick A. Wetsel. (2014). The Receptor for the Complement C3a Anaphylatoxin (C3aR) Provides Host Protection against Listeria monocytogenes–Induced Apoptosis. The Journal of Immunology. 193(3). 1278–1289. 32 indexed citations
15.
Calame, Daniel G., Stacey L. Mueller‐Ortiz, John E. Morales, & Rick A. Wetsel. (2014). The C5a Anaphylatoxin Receptor (C5aR1) Protects against Listeria monocytogenes Infection by Inhibiting Type 1 IFN Expression. The Journal of Immunology. 193(10). 5099–5107. 20 indexed citations
16.
Morales, John E., et al.. (2013). Clinical studies support a role for trem-like transcript-1 during the progression of sepsis.. PubMed. 102(3). 59–61. 7 indexed citations
17.
Mueller‐Ortiz, Stacey L., John E. Morales, Amanda M. Clark, & Rick A. Wetsel. (2012). The complement anaphylatoxin C3a is critical in generating both innate and adaptive immune responses to Listeria monocytogenes due to its indispensible role in immune cell survival. Immunobiology. 217(11). 1209–1209. 1 indexed citations
18.
Wang, Dachun, John E. Morales, Daniel G. Calame, Joseph L. Alcorn, & Rick A. Wetsel. (2010). Transplantation of Human Embryonic Stem Cell–Derived Alveolar Epithelial Type II Cells Abrogates Acute Lung Injury in Mice. Molecular Therapy. 18(3). 625–634. 108 indexed citations
19.
Mueller‐Ortiz, Stacey L., Dachun Wang, John E. Morales, et al.. (2009). Targeted Disruption of the Gene Encoding the Murine Small Subunit of Carboxypeptidase N (CPN1) Causes Susceptibility to C5a Anaphylatoxin-Mediated Shock. The Journal of Immunology. 182(10). 6533–6539. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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