Johannes Stöckl

7.4k total citations · 1 hit paper
93 papers, 5.9k citations indexed

About

Johannes Stöckl is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Johannes Stöckl has authored 93 papers receiving a total of 5.9k indexed citations (citations by other indexed papers that have themselves been cited), including 60 papers in Immunology, 29 papers in Molecular Biology and 12 papers in Oncology. Recurrent topics in Johannes Stöckl's work include Immunotherapy and Immune Responses (36 papers), Immune Cell Function and Interaction (31 papers) and T-cell and B-cell Immunology (27 papers). Johannes Stöckl is often cited by papers focused on Immunotherapy and Immune Responses (36 papers), Immune Cell Function and Interaction (31 papers) and T-cell and B-cell Immunology (27 papers). Johannes Stöckl collaborates with scholars based in Austria, Croatia and Germany. Johannes Stöckl's co-authors include Otto Majdic, Walter Knapp, Gerhard J. Zlabinger, Winfried F. Pickl, Peter Steinberger, Stefanie Kirchberger, Valery N. Bochkov, Olga Oskolkova, Elisabeth Riedl and Clemens Scheinecker and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Clinical Investigation and The Journal of Experimental Medicine.

In The Last Decade

Johannes Stöckl

90 papers receiving 5.8k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Tumor-Associated Macrophages Express Lymphatic Endothelial Growth Factors and Are Related to Peritumoral Lymphangiogenesis

2002 640 citations Johannes Stöckl et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Johannes Stöckl Austria	39	2.9k	2.0k	1.5k	537	521	93	5.9k
Alice Mui Canada	40	3.0k 1.0×	2.6k 1.3×	2.6k 1.7×	353 0.7×	424 0.8×	92	6.5k
Thibaut De Smedt Belgium	28	5.1k 1.7×	2.6k 1.3×	1.4k 1.0×	481 0.9×	608 1.2×	36	8.0k
Shaun R. McColl Australia	56	5.1k 1.7×	2.3k 1.2×	2.3k 1.6×	710 1.3×	692 1.3×	170	8.8k
Stephen T. Smiley United States	35	4.3k 1.5×	1.6k 0.8×	1.6k 1.1×	276 0.5×	732 1.4×	64	7.4k
Bernd Echtenacher Germany	30	3.0k 1.0×	1.2k 0.6×	761 0.5×	522 1.0×	670 1.3×	56	5.1k
Roberto Benelli Italy	45	1.7k 0.6×	2.6k 1.3×	2.0k 1.4×	444 0.8×	443 0.9×	123	6.7k
Alison Finnegan United States	46	2.8k 0.9×	1.4k 0.7×	993 0.7×	408 0.8×	554 1.1×	91	5.6k
Hans A. Lehr Germany	34	2.8k 1.0×	1.9k 1.0×	1.2k 0.8×	256 0.5×	799 1.5×	65	6.3k
Kamran Ghoreschi Germany	44	4.4k 1.5×	1.7k 0.9×	2.0k 1.3×	305 0.6×	599 1.1×	143	8.2k
Frank Petersen Germany	41	1.5k 0.5×	2.1k 1.1×	1.3k 0.9×	582 1.1×	322 0.6×	143	5.6k

Countries citing papers authored by Johannes Stöckl

Since Specialization

Citations

This map shows the geographic impact of Johannes Stöckl's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Johannes Stöckl with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Johannes Stöckl more than expected).

Fields of papers citing papers by Johannes Stöckl

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Johannes Stöckl. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Johannes Stöckl. The network helps show where Johannes Stöckl may publish in the future.

Co-authorship network of co-authors of Johannes Stöckl

This figure shows the co-authorship network connecting the top 25 collaborators of Johannes Stöckl. A scholar is included among the top collaborators of Johannes Stöckl based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Johannes Stöckl. Johannes Stöckl is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wideman, Sarah K., et al.. (2025). Nebulized 2-deoxylated glucose analogues inhibit respiratory viral infection in advanced in vitro airway models. Scientific Reports. 15(1). 9515–9515.

Prencipe, Giuseppe, Alexandra Correia, Sami Hietala, et al.. (2025). Development of Amniotic Epithelial Stem Cells Secretome-Loaded In Situ Inverse Electron Demand Diels–Alder-Cross-Linked Hydrogel as a Potential Immunomodulatory Therapeutical Tool. ACS Applied Materials & Interfaces. 17(2). 2977–2990. 1 indexed citations

Jonak, Constanze, et al.. (2024). Regulation of the Immune Cell Repertoire in Psoriasis Patients Upon Blockade of IL-17A or TNFα. Dermatology and Therapy. 14(3). 613–626. 4 indexed citations

Russo, Valentina, Giuseppe Prencipe, Annunziata Mauro, et al.. (2024). Assessing the functional potential of conditioned media derived from amniotic epithelial stem cells engineered on 3D biomimetic scaffolds: An in vitro model for tendon regeneration. Materials Today Bio. 25. 101001–101001. 1 indexed citations

Stöckl, Johannes, et al.. (2023). Lightweight Potential and Crash Performance of Friction Stir Welded Tailored Blanks. SAE International Journal of Advances and Current Practices in Mobility. 6(2). 1100–1106.

Russo, Valentina, Mohammad El Khatib, Giuseppe Prencipe, et al.. (2022). Tendon Immune Regeneration: Insights on the Synergetic Role of Stem and Immune Cells during Tendon Regeneration. Cells. 11(3). 434–434. 43 indexed citations

Santarsiero, Anna, Paolo Convertini, Antonio Vassallo, et al.. (2021). Phenolic Compounds of Red Wine Aglianico del Vulture Modulate the Functional Activity of Macrophages via Inhibition of NF-κB and the Citrate Pathway. Oxidative Medicine and Cellular Longevity. 2021(1). 5533793–5533793. 14 indexed citations

Bochkov, Valery N., Olga Oskolkova, Konstantin G. Birukov, et al.. (2009). Generation and Biological Activities of Oxidized Phospholipids. Antioxidants and Redox Signaling. 12(8). 1009–1059. 465 indexed citations

Kirchberger, Stefanie, Otto Majdic, Gerhard J. Zlabinger, et al.. (2009). The ssRNA Genome of Human Rhinovirus Induces a Type I IFN Response but Fails to Induce Maturation in Human Monocyte-Derived Dendritic Cells. The Journal of Immunology. 183(7). 4440–4448. 13 indexed citations

10.

Blüml, Stephan, Stefanie Kirchberger, Valery N. Bochkov, et al.. (2008). Oxidized phospholipids induce anergy in human peripheral blood T cells. European Journal of Immunology. 38(3). 778–787. 31 indexed citations

11.

Blüml, Stephan, Ákos M. Lőrincz, Stefanie Kirchberger, et al.. (2008). The Oxidation State of Phospholipids Controls the Oxidative Burst in Neutrophil Granulocytes. The Journal of Immunology. 181(6). 4347–4353. 32 indexed citations

12.

Grabmeier‐Pfistershammer, Katharina, Christoph Klauser, Winfried F. Pickl, et al.. (2006). No evidence for dualism in function and receptors: PD‐L2/B7‐DC is an inhibitory regulator of human T cell activation. European Journal of Immunology. 36(5). 1104–1113. 45 indexed citations

13.

Kirchberger, Stefanie, Otto Majdic, Peter Steinberger, et al.. (2005). Human Rhinoviruses Inhibit the Accessory Function of Dendritic Cells by Inducing Sialoadhesin and B7-H1 Expression. The Journal of Immunology. 175(2). 1145–1152. 78 indexed citations

14.

Steinberger, Peter, Otto Majdic, Sophia Derdak, et al.. (2004). Molecular Characterization of Human 4Ig-B7-H3, a Member of the B7 Family with Four Ig-Like Domains. The Journal of Immunology. 172(4). 2352–2359. 224 indexed citations

15.

Grabmeier‐Pfistershammer, Katharina, Otto Majdic, Johannes Stöckl, et al.. (2004). CD63 as an Activation-Linked T Cell Costimulatory Element. The Journal of Immunology. 173(10). 6000–6008. 58 indexed citations

16.

Selenko-Gebauer, Nicole, Otto Majdic, Andreas Szekeres, et al.. (2003). B7-H1 (Programmed Death-1 Ligand) on Dendritic Cells Is Involved in the Induction and Maintenance of T Cell Anergy. The Journal of Immunology. 170(7). 3637–3644. 225 indexed citations

17.

Szekeres, Andreas, Otto Majdic, Elisabeth Prager, et al.. (2001). Characterization of CDw92 as a Member of the Choline Transporter-Like Protein Family Regulated Specifically on Dendritic Cells. The Journal of Immunology. 167(10). 5795–5804. 33 indexed citations

18.

Stöckl, Johannes, Otto Majdic, Gottfried Fischer, Dieter Maurer, & Walter Knapp. (2001). Monomorphic Molecules Function as Additional Recognition Structures on Haptenated Target Cells for HLA-A1-Restricted, Hapten-Specific CTL. The Journal of Immunology. 167(5). 2724–2733. 17 indexed citations

19.

Berer, A., Johannes Stöckl, Otto Majdic, et al.. (2000). 1,25-Dihydroxyvitamin D3 inhibits dendritic cell differentiation and maturation in vitro. Experimental Hematology. 28(5). 575–583. 175 indexed citations

20.

Riedl, Elisabeth, Johannes Stöckl, Otto Majdic, et al.. (2000). Functional Involvement of E-Cadherin in TGF-β1-Induced Cell Cluster Formation of In Vitro Developing Human Langerhans-Type Dendritic Cells. The Journal of Immunology. 165(3). 1381–1386. 53 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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