Joaquin M. Muriel

998 total citations
23 papers, 709 citations indexed

About

Joaquin M. Muriel is a scholar working on Molecular Biology, Cell Biology and Aging. According to data from OpenAlex, Joaquin M. Muriel has authored 23 papers receiving a total of 709 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 9 papers in Cell Biology and 5 papers in Aging. Recurrent topics in Joaquin M. Muriel's work include Muscle Physiology and Disorders (10 papers), Cellular Mechanics and Interactions (6 papers) and Ion channel regulation and function (6 papers). Joaquin M. Muriel is often cited by papers focused on Muscle Physiology and Disorders (10 papers), Cellular Mechanics and Interactions (6 papers) and Ion channel regulation and function (6 papers). Joaquin M. Muriel collaborates with scholars based in United States, Germany and United Kingdom. Joaquin M. Muriel's co-authors include Richard M. Lovering, Robert J. Bloch, Leigh Ann Curl, Richard Y. Hinton, Bruce E. Vogel, Iain L. Johnstone, Laura McMahon, Harald Hutter, Jaclyn P. Kerr and Andrea O’Neill and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and SHILAP Revista de lepidopterología.

In The Last Decade

Joaquin M. Muriel

22 papers receiving 700 citations

Peers

Joaquin M. Muriel

SURGE

AGING

OSM

Tetsuo Kobayashi Japan

Ching‐Yan Chloé Yeung Denmark

Luisa Boldrin United Kingdom

Pierre Rocheteau France

Joana Esteves de Lima France

Kathleen Kelly United States

Cecilia Riquelme Chile

Daniel W. Youngstrom United States

Jason O. Brant United States

Byeong Cha United States

Tetsuo Kobayashi Japan

Joaquin M. Muriel

324 ×1.0

189 ×1.1

SURGE

100 ×0.7

13 ×0.1

AGING

140 ×1.1

OSM

Citations per year, relative to Joaquin M. Muriel Joaquin M. Muriel (= 1×) peers Tetsuo Kobayashi

Countries citing papers authored by Joaquin M. Muriel

Since Specialization

Citations

This map shows the geographic impact of Joaquin M. Muriel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joaquin M. Muriel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joaquin M. Muriel more than expected).

Fields of papers citing papers by Joaquin M. Muriel

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joaquin M. Muriel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joaquin M. Muriel. The network helps show where Joaquin M. Muriel may publish in the future.

Co-authorship network of co-authors of Joaquin M. Muriel

This figure shows the co-authorship network connecting the top 25 collaborators of Joaquin M. Muriel. A scholar is included among the top collaborators of Joaquin M. Muriel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joaquin M. Muriel. Joaquin M. Muriel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Muriel, Joaquin M., et al.. (2024). Nanodysferlins support membrane repair and binding to TRIM72/MG53 but do not localize to t-tubules or stabilize Ca2+ signaling. Molecular Therapy — Methods & Clinical Development. 32(2). 101257–101257.

Lukyanenko, Valeriy, et al.. (2022). Elevated Ca2+ at the triad junction underlies dysregulation of Ca2+ signaling in dysferlin-null skeletal muscle. Frontiers in Physiology. 13. 1032447–1032447. 5 indexed citations

Muriel, Joaquin M., Valeriy Lukyanenko, Tom Kwiatkowski, et al.. (2022). The C2 domains of dysferlin: roles in membrane localization, Ca²⁺ signalling and sarcolemmal repair. The Journal of Physiology. 600(8). 1953–1968. 16 indexed citations

O’Neill, Andrea, Weiliang Huang, Joaquin M. Muriel, et al.. (2021). μ-Crystallin in Mouse Skeletal Muscle Promotes a Shift from Glycolytic toward Oxidative Metabolism. SHILAP Revista de lepidopterología. 4. 47–59. 2 indexed citations

Lukyanenko, Valeriy, Joaquin M. Muriel, & Robert J. Bloch. (2020). Effect of Bapta and Dysferlin's C2A Domain on Recovery of Ca2D Transients After Osmotic Shock in Dysferlin-Null Myofibers. Biophysical Journal. 118(3). 35a–35a. 1 indexed citations

Muriel, Joaquin M., et al.. (2017). Interactions between small ankyrin 1 and sarcolipin coordinately regulate activity of the sarco(endo)plasmic reticulum Ca2+-ATPase (SERCA1). Journal of Biological Chemistry. 292(26). 10961–10972. 16 indexed citations

Lukyanenko, Valeriy, Joaquin M. Muriel, & Robert J. Bloch. (2017). Coupling of excitation to Ca²⁺release is modulated by dysferlin. The Journal of Physiology. 595(15). 5191–5207. 23 indexed citations

Muriel, Joaquin M., et al.. (2015). Identification of Small Ankyrin 1 as a Novel Sarco(endo)plasmic Reticulum Ca2+-ATPase 1 (SERCA1) Regulatory Protein in Skeletal Muscle. Journal of Biological Chemistry. 290(46). 27854–27867. 18 indexed citations

Muriel, Joaquin M., Andrea O’Neill, Richard M. Lovering, et al.. (2015). Myopathic changes in murine skeletal muscle lacking synemin. American Journal of Physiology-Cell Physiology. 308(6). C448–C462. 29 indexed citations

10.

Kerr, Jaclyn P., Andrew P. Ziman, Amber L. Mueller, et al.. (2013). Dysferlin stabilizes stress-induced Ca ²⁺ signaling in the transverse tubule membrane. Proceedings of the National Academy of Sciences. 110(51). 20831–20836. 105 indexed citations

11.

Muriel, Joaquin M., et al.. (2012). Distinct regions within fibulin-1D modulate interactions with hemicentin. Experimental Cell Research. 318(20). 2543–2547. 9 indexed citations

12.

Lovering, Richard M., Andrea O’Neill, Joaquin M. Muriel, et al.. (2011). Physiology, structure, and susceptibility to injury of skeletal muscle in mice lacking keratin 19-based and desmin-based intermediate filaments. American Journal of Physiology-Cell Physiology. 300(4). C803–C813. 44 indexed citations

13.

Lovering, Richard M., et al.. (2010). Muscle Physiology, Membrane Structure, and Susceptibility to Injury in Mice Lacking Intermediate Filaments. Medicine & Science in Sports & Exercise. 42(5). 8–8. 1 indexed citations

14.

Hinton, Richard Y., et al.. (2009). Use of Autologous Platelet-rich Plasma to Treat Muscle Strain Injuries. The American Journal of Sports Medicine. 37(6). 1135–1142. 200 indexed citations

15.

Muriel, Joaquin M., Harald Hutter, Edward M. Hedgecock, et al.. (2006). Hemicentin Assembly in the Extracellular Matrix Is Mediated by Distinct Structural Modules. Journal of Biological Chemistry. 281(33). 23606–23610. 25 indexed citations

16.

Muriel, Joaquin M., Xuehong Xu, James M. Kramer, & Bruce E. Vogel. (2006). Selective assembly of fibulin‐1 splice variants reveals distinct extracellular matrix networks and novel functions for perlecan/UNC‐52 splice variants. Developmental Dynamics. 235(10). 2632–2640. 15 indexed citations

17.

Vogel, Bruce E., et al.. (2006). Hemicentins: What have we learned from worms?. Cell Research. 16(11). 872–878. 30 indexed citations

18.

Muriel, Joaquin M., et al.. (2005). Fibulin-1C and Fibulin-1D splice variants have distinct functions and assemble in a hemicentin-dependent manner. Development. 132(19). 4223–4234. 47 indexed citations

19.

McMahon, Laura, et al.. (2003). Two Sets of Interacting Collagens Form Functionally Distinct Substructures within aCaenorhabditis elegansExtracellular Matrix. Molecular Biology of the Cell. 14(4). 1366–1378. 80 indexed citations

20.

Muriel, Joaquin M., et al.. (2003). M142.2 (cut-6), a novel Caenorhabditis elegans matrix gene important for dauer body shape. Developmental Biology. 260(2). 339–351. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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