Joan M. Carboni

5.5k total citations
75 papers, 4.0k citations indexed

About

Joan M. Carboni is a scholar working on Molecular Biology, Endocrinology, Diabetes and Metabolism and Oncology. According to data from OpenAlex, Joan M. Carboni has authored 75 papers receiving a total of 4.0k indexed citations (citations by other indexed papers that have themselves been cited), including 40 papers in Molecular Biology, 38 papers in Endocrinology, Diabetes and Metabolism and 32 papers in Oncology. Recurrent topics in Joan M. Carboni's work include Growth Hormone and Insulin-like Growth Factors (38 papers), Metabolism, Diabetes, and Cancer (16 papers) and Cancer, Hypoxia, and Metabolism (13 papers). Joan M. Carboni is often cited by papers focused on Growth Hormone and Insulin-like Growth Factors (38 papers), Metabolism, Diabetes, and Cancer (16 papers) and Cancer, Hypoxia, and Metabolism (13 papers). Joan M. Carboni collaborates with scholars based in United States, Germany and Canada. Joan M. Carboni's co-authors include Marco M. Gottardis, Mark S. Mooseker, Ricardo M. Attar, Anna Maria Casazza, Michaël Pollak, Thomas Lindel, Paul R. Jensen, William Fenical, Ann Greer and John S. Condeelis and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of the American Chemical Society and Journal of Biological Chemistry.

In The Last Decade

Joan M. Carboni

75 papers receiving 3.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Joan M. Carboni United States	34	2.4k	1.4k	1.0k	804	570	75	4.0k
Paul Kussie United States	30	2.1k 0.9×	1.1k 0.8×	284 0.3×	441 0.5×	649 1.1×	47	3.5k
Ricardo M. Attar United States	30	1.7k 0.7×	877 0.6×	864 0.9×	652 0.8×	176 0.3×	72	3.2k
Angelika M. Burger United States	37	2.7k 1.1×	981 0.7×	189 0.2×	684 0.9×	223 0.4×	81	4.1k
Athan Kuliopulos United States	48	2.9k 1.2×	777 0.6×	276 0.3×	1.3k 1.6×	398 0.7×	94	6.6k
Richard Kendall United States	24	1.8k 0.7×	837 0.6×	186 0.2×	476 0.6×	236 0.4×	43	3.0k
Francesca Pentimalli Italy	38	2.3k 0.9×	1.1k 0.8×	522 0.5×	922 1.1×	213 0.4×	120	3.8k
Junichi Kurebayashi Japan	37	1.8k 0.7×	1.9k 1.4×	497 0.5×	999 1.2×	208 0.4×	157	3.8k
Peng Hou China	41	3.3k 1.3×	1.3k 0.9×	1.6k 1.6×	1.1k 1.4×	192 0.3×	177	5.7k
Harshani R. Lawrence United States	35	2.1k 0.9×	1.3k 0.9×	272 0.3×	283 0.4×	253 0.4×	75	3.5k
Judith S. Sebolt–Leopold United States	33	4.9k 2.0×	2.7k 2.0×	156 0.2×	790 1.0×	576 1.0×	67	6.8k

Countries citing papers authored by Joan M. Carboni

Since Specialization

Citations

This map shows the geographic impact of Joan M. Carboni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joan M. Carboni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joan M. Carboni more than expected).

Fields of papers citing papers by Joan M. Carboni

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joan M. Carboni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joan M. Carboni. The network helps show where Joan M. Carboni may publish in the future.

Co-authorship network of co-authors of Joan M. Carboni

This figure shows the co-authorship network connecting the top 25 collaborators of Joan M. Carboni. A scholar is included among the top collaborators of Joan M. Carboni based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joan M. Carboni. Joan M. Carboni is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Huang, Fei, Chang Han, Ann Greer, et al.. (2014). IRS2 Copy Number Gain, KRAS and BRAF Mutation Status as Predictive Biomarkers for Response to the IGF-1R/IR Inhibitor BMS-754807 in Colorectal Cancer Cell Lines. Molecular Cancer Therapeutics. 14(2). 620–630. 25 indexed citations

Becker, Marc A., Xiaonan Hou, Sean C. Harrington, et al.. (2012). IGFBP Ratio Confers Resistance to IGF Targeting and Correlates with Increased Invasion and Poor Outcome in Breast Tumors. Clinical Cancer Research. 18(6). 1808–1817. 28 indexed citations

Jenkins, Catherine E., Olena O Shevchuk, Vincenzo Giambra, et al.. (2012). IGF signaling contributes to malignant transformation of hematopoietic progenitors by the MLL-AF9 oncoprotein. Experimental Hematology. 40(9). 715–723.e6. 17 indexed citations

Hou, Xiaonan, Fei Huang, Joan M. Carboni, et al.. (2011). Drug Efflux by Breast Cancer Resistance Protein Is a Mechanism of Resistance to the Benzimidazole Insulin-Like Growth Factor Receptor/Insulin Receptor Inhibitor, BMS-536924. Molecular Cancer Therapeutics. 10(1). 117–125. 16 indexed citations

Hou, Xiaonan, Fei Huang, Luciana F. Macedo, et al.. (2011). Dual IGF-1R/InsR Inhibitor BMS-754807 Synergizes with Hormonal Agents in Treatment of Estrogen-Dependent Breast Cancer. Cancer Research. 71(24). 7597–7607. 63 indexed citations

Litzenburger, Beate C., Chad J. Creighton, Anna Tsimelzon, et al.. (2010). High IGF-IR Activity in Triple-Negative Breast Cancer Cell Lines and Tumorgrafts Correlates with Sensitivity to Anti–IGF-IR Therapy. Clinical Cancer Research. 17(8). 2314–2327. 109 indexed citations

Huang, Fei, Warren Hurlburt, Ann Greer, et al.. (2010). Differential Mechanisms of Acquired Resistance to Insulin-like Growth Factor-I Receptor Antibody Therapy or to a Small-Molecule Inhibitor, BMS-754807, in a Human Rhabdomyosarcoma Model. Cancer Research. 70(18). 7221–7231. 97 indexed citations

Novosyadlyy, Ruslan, Archana Vijayakumar, Anne Rowzee, et al.. (2010). Insulin-Mediated Acceleration of Breast Cancer Development and Progression in a Nonobese Model of Type 2 Diabetes. Cancer Research. 70(2). 741–751. 219 indexed citations

Potratz, Jenny, Darren N. Saunders, Daniel Wai, et al.. (2010). Synthetic Lethality Screens Reveal RPS6 and MST1R as Modifiers of Insulin-like Growth Factor-1 Receptor Inhibitor Activity in Childhood Sarcomas. Cancer Research. 70(21). 8770–8781. 55 indexed citations

10.

Tognon, Cristina E., Aruna M. Somasiri, Valentina Evdokimova, et al.. (2010). ETV6-NTRK3–Mediated Breast Epithelial Cell Transformation Is Blocked by Targeting the IGF1R Signaling Pathway. Cancer Research. 71(3). 1060–1070. 52 indexed citations

11.

Carboni, Joan M., Mark D. Wittman, Zheng Yang, et al.. (2009). BMS-754807, a small molecule inhibitor of insulin-like growth factor-1R/IR. Molecular Cancer Therapeutics. 8(12). 3341–3349. 218 indexed citations

12.

Hendrickson, Andrea E. Wahner, Paul Haluska, Paula A. Schneider, et al.. (2009). Expression of Insulin Receptor Isoform A and Insulin-like Growth Factor-1 Receptor in Human Acute Myelogenous Leukemia: Effect of the Dual-Receptor Inhibitor BMS-536924 In vitro. Cancer Research. 69(19). 7635–7643. 43 indexed citations

13.

Haluska, Paul, Joan M. Carboni, Cynthia J. TenEyck, et al.. (2008). HER receptor signaling confers resistance to the insulin-like growth factor-I receptor inhibitor, BMS-536924. Molecular Cancer Therapeutics. 7(9). 2589–2598. 85 indexed citations

14.

Law, Jennifer, Golareh Habibi, Kaiji Hu, et al.. (2008). Phosphorylated Insulin-Like Growth Factor-I/Insulin Receptor Is Present in All Breast Cancer Subtypes and Is Related to Poor Survival. Cancer Research. 68(24). 10238–10246. 339 indexed citations

15.

Huang, Fei, Ann Greer, Warren Hurlburt, et al.. (2008). The Mechanisms of Differential Sensitivity to an Insulin-like Growth Factor-1 Receptor Inhibitor (BMS-536924) and Rationale for Combining with EGFR/HER2 Inhibitors. Cancer Research. 69(1). 161–170. 137 indexed citations

16.

Litzenburger, Beate C., Isere Kuiatse, Joan M. Carboni, et al.. (2008). BMS-536924 Reverses IGF-IR-Induced Transformation of Mammary Epithelial Cells and Causes Growth Inhibition and Polarization of MCF7 Cells. Clinical Cancer Research. 15(1). 226–237. 33 indexed citations

17.

Kim, Hyun Jung, Beate C. Litzenburger, Xiaojiang Cui, et al.. (2007). Constitutively Active Type I Insulin-Like Growth Factor Receptor Causes Transformation and Xenograft Growth of Immortalized Mammary Epithelial Cells and Is Accompanied by an Epithelial-to-Mesenchymal Transition Mediated by NF-κB and Snail. Molecular and Cellular Biology. 27(8). 3165–3175. 196 indexed citations

18.

Haluska, Paul, Joan M. Carboni, David A. Loegering, et al.. (2005). BMS-554417, an inhibitor of the insulin-like growth factor I receptor and insulin receptor, inhibits proliferation and induces mitochondrial pathway-mediated apoptosis in cancer cell lines. Cancer Research. 65. 1191–1191. 2 indexed citations

19.

Howell, Gillian, Lisa E. Humphrey, Barry L. Ziober, et al.. (1998). Regulation of Transforming Growth Factor α Expression in a Growth Factor-Independent Cell Line. Molecular and Cellular Biology. 18(1). 303–313. 16 indexed citations

20.

Ogihara, Satoshi, Joan M. Carboni, & John S. Condeelis. (1988). Electron microscopic localization of myosin II and ABP‐120 in the cortical actin matrix of Dictyostelium amoebae using IgG‐gold conjugates. Developmental Genetics. 9(4-5). 505–520. 28 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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