Jo Vanoevelen

2.3k total citations
45 papers, 1.8k citations indexed

About

Jo Vanoevelen is a scholar working on Molecular Biology, Cell Biology and Genetics. According to data from OpenAlex, Jo Vanoevelen has authored 45 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Molecular Biology, 19 papers in Cell Biology and 12 papers in Genetics. Recurrent topics in Jo Vanoevelen's work include Cellular transport and secretion (13 papers), Genetic and rare skin diseases. (9 papers) and Mitochondrial Function and Pathology (8 papers). Jo Vanoevelen is often cited by papers focused on Cellular transport and secretion (13 papers), Genetic and rare skin diseases. (9 papers) and Mitochondrial Function and Pathology (8 papers). Jo Vanoevelen collaborates with scholars based in Belgium, Netherlands and United States. Jo Vanoevelen's co-authors include Frank Wuytack, Luc Raeymaekers, Ludwig Missiaen, Leonard Dode, Kurt Van Baelen, Jan B. Parys, Peter Vangheluwe, Geert Callewaert, Ilse Vandecaetsbeek and L. Raeymaekers and has published in prestigious journals such as Chemical Reviews, Journal of Biological Chemistry and Journal of Neuroscience.

In The Last Decade

Jo Vanoevelen

45 papers receiving 1.8k citations

Peers

Jo Vanoevelen

GENET

CMN

Radha Ayyagari United States

Leonard Dode Belgium

Hiroyuki Okamoto Japan

Aaron G. Smith Australia

Maija H. Zile United States

Udo Hasler Switzerland

Nicolás Montalbetti United States

Anastasia Andringa United States

Michael N. Badminton United Kingdom

Gregory W. Aponte United States

Radha Ayyagari United States

Jo Vanoevelen

2.8k ×2.3

477 ×0.8

455 ×1.4

GENET

322 ×1.3

CMN

160 ×0.9

Citations per year, relative to Jo Vanoevelen Jo Vanoevelen (= 1×) peers Radha Ayyagari

Countries citing papers authored by Jo Vanoevelen

Since Specialization

Citations

This map shows the geographic impact of Jo Vanoevelen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jo Vanoevelen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jo Vanoevelen more than expected).

Fields of papers citing papers by Jo Vanoevelen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jo Vanoevelen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jo Vanoevelen. The network helps show where Jo Vanoevelen may publish in the future.

Co-authorship network of co-authors of Jo Vanoevelen

This figure shows the co-authorship network connecting the top 25 collaborators of Jo Vanoevelen. A scholar is included among the top collaborators of Jo Vanoevelen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jo Vanoevelen. Jo Vanoevelen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Bekke, Rachel M.A. ter, Sergio Conti, Godelieve R.F. Claes, et al.. (2023). Juvenile-onset multifocal atrial arrhythmias, atrial standstill and compound heterozygosity of genetic variants in TAF1A: sentinel event for evolving dilated cardiomyopathy—a case report. European Heart Journal - Case Reports. 7(6). ytad255–ytad255. 1 indexed citations

Vanoevelen, Jo, Jörgen Bierau, Erik‐Jan Kamsteeg, et al.. (2021). DTYMK is essential for genome integrity and neuronal survival. Acta Neuropathologica. 143(2). 245–262. 11 indexed citations

Kapsokalyvas, Dimitrios, Jo Vanoevelen, Miranda Nabben, et al.. (2021). Multiview deconvolution approximation multiphoton microscopy of tissues and zebrafish larvae. Scientific Reports. 11(1). 10160–10160. 5 indexed citations

Chen, Jialin, Filip Pamula, Ilse Vandecaetsbeek, et al.. (2019). An N-terminal Ca2+-binding motif regulates the secretory pathway Ca2+/Mn2+-transport ATPase SPCA1. Journal of Biological Chemistry. 294(19). 7878–7891. 15 indexed citations

Vanoevelen, Jo, et al.. (2019). A zebrafish model to study small-fiber neuropathy reveals a potential role for GDAP1. Mitochondrion. 47. 273–281. 9 indexed citations

Otten, Auke B.C., Monique M. Gerrits, Janneke G. J. Hoeijmakers, et al.. (2018). Expression of pathogenic SCN9A mutations in the zebrafish: A model to study small-fiber neuropathy. Experimental Neurology. 311. 257–264. 17 indexed citations

Kamps, Rick, Radek Szklarczyk, Tom E. J. Theunissen, et al.. (2018). Genetic defects in mtDNA-encoded protein translation cause pediatric, mitochondrial cardiomyopathy with early-onset brain disease. European Journal of Human Genetics. 26(4). 537–551. 22 indexed citations

Paulussen, Aimée, Anja Steyls, Jo Vanoevelen, et al.. (2016). Rare novel variants in the ZIC3 gene cause X-linked heterotaxy. European Journal of Human Genetics. 24(12). 1783–1791. 16 indexed citations

Otten, Auke B.C., Tom E. J. Theunissen, Josien G. Derhaag, et al.. (2016). Differences in Strength and Timing of the mtDNA Bottleneck between Zebrafish Germline and Non-germline Cells. Cell Reports. 16(3). 622–630. 41 indexed citations

10.

Theunissen, Tom E. J., Radek Szklarczyk, Mike Gerards, et al.. (2016). Specific MRI Abnormalities Reveal Severe Perrault Syndrome due to CLPP Defects. Frontiers in Neurology. 7. 203–203. 21 indexed citations

11.

Hellebrekers, Debby M.E.I., Jo Vanoevelen, Rick Kamps, et al.. (2015). Pathogenic CWF19L1 variants as a novel cause of autosomal recessive cerebellar ataxia and atrophy. European Journal of Human Genetics. 24(4). 619–622. 18 indexed citations

12.

Vandecaetsbeek, Ilse, et al.. (2011). The Ca2+ Pumps of the Endoplasmic Reticulum and Golgi Apparatus. Cold Spring Harbor Perspectives in Biology. 3(5). a004184–a004184. 171 indexed citations

13.

Missiaen, Ludwig, Leonard Dode, Jo Vanoevelen, Luc Raeymaekers, & Frank Wuytack. (2006). Calcium in the Golgi apparatus. Cell Calcium. 41(5). 405–416. 88 indexed citations

14.

Vanoevelen, Jo, Leonard Dode, Kurt Van Baelen, et al.. (2005). The Secretory Pathway Ca2+/Mn2+-ATPase 2 Is a Golgi-localized Pump with High Affinity for Ca2+ Ions. Journal of Biological Chemistry. 280(24). 22800–22808. 105 indexed citations

15.

Baelen, Kurt Van, Leonard Dode, Jo Vanoevelen, et al.. (2004). The Ca2+/Mn2+ pumps in the Golgi apparatus. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1742(1-3). 103–112. 106 indexed citations

16.

Missiaen, Ludwig, Karel Van Acker, Karen Van Baelen, et al.. (2004). Calcium release from the Golgi apparatus and the endoplasmic reticulum in HeLa cells stably expressing targeted aequorin to these compartments. Cell Calcium. 36(6). 479–487. 47 indexed citations

17.

Baelen, Kurt Van, Jo Vanoevelen, Geert Callewaert, et al.. (2003). The contribution of the SPCA1 Ca2+ pump to the Ca2+ accumulation in the Golgi apparatus of HeLa cells assessed via RNA-mediated interference. Biochemical and Biophysical Research Communications. 306(2). 430–436. 75 indexed citations

18.

Fairclough, Rebecca J., Leonard Dode, Jo Vanoevelen, et al.. (2003). Effect of Hailey-Hailey Disease Mutations on the Function of a New Variant of Human Secretory Pathway Ca2+/Mn2+-ATPase (hSPCA1). Journal of Biological Chemistry. 278(27). 24721–24730. 78 indexed citations

19.

Missiaen, Ludwig, Jo Vanoevelen, Luc Raeymaekers, et al.. (2002). Ca2+ signals in Pmr1-GFP-expressing COS-1 cells with functional endoplasmic reticulum. Biochemical and Biophysical Research Communications. 294(2). 249–253. 17 indexed citations

20.

Baelen, Kurt Van, Jo Vanoevelen, Ludwig Missiaen, Luc Raeymaekers, & Frank Wuytack. (2001). The Golgi PMR1 P-type ATPase of Caenorhabditis elegans. Journal of Biological Chemistry. 276(14). 10683–10691. 76 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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