Jinyao Mo

1.9k total citations
24 papers, 1.4k citations indexed

About

Jinyao Mo is a scholar working on Molecular Biology, Health, Toxicology and Mutagenesis and Environmental Chemistry. According to data from OpenAlex, Jinyao Mo has authored 24 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 6 papers in Health, Toxicology and Mutagenesis and 5 papers in Environmental Chemistry. Recurrent topics in Jinyao Mo's work include DNA Repair Mechanisms (6 papers), Arsenic contamination and mitigation (5 papers) and Heavy Metal Exposure and Toxicity (5 papers). Jinyao Mo is often cited by papers focused on DNA Repair Mechanisms (6 papers), Arsenic contamination and mitigation (5 papers) and Heavy Metal Exposure and Toxicity (5 papers). Jinyao Mo collaborates with scholars based in United States, China and Poland. Jinyao Mo's co-authors include Joseph A. Duncan, Ryujiro Hara, Aziz Sancar, Marietta Lee, Li Liu, Wei Chen, Gerard C. Blobe, Judy L. Mumford, Yajuan Xia and Hisaji Maki and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Jinyao Mo

23 papers receiving 1.4k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jinyao Mo United States 18 1.0k 428 164 161 138 24 1.4k
Prashant R. Nambiar United States 24 813 0.8× 502 1.2× 294 1.8× 545 3.4× 343 2.5× 55 2.0k
Yi Hu China 26 779 0.8× 615 1.4× 67 0.4× 154 1.0× 603 4.4× 61 1.6k
Yuanyuan Zheng China 17 1000 1.0× 683 1.6× 55 0.3× 317 2.0× 637 4.6× 40 1.9k
Jing Gong China 22 1.2k 1.2× 222 0.5× 190 1.2× 104 0.6× 787 5.7× 63 1.8k
Moses T. Bility United States 17 536 0.5× 219 0.5× 101 0.6× 84 0.5× 197 1.4× 30 1.3k
Martina Dorsch Germany 19 432 0.4× 327 0.8× 155 0.9× 143 0.9× 118 0.9× 42 1.2k
Domenico Frezza Italy 20 515 0.5× 253 0.6× 142 0.9× 59 0.4× 171 1.2× 56 1.1k
Xiaolin Luo China 23 722 0.7× 182 0.4× 146 0.9× 199 1.2× 210 1.5× 77 1.4k
Peter E. Phelan United States 11 349 0.3× 679 1.6× 62 0.4× 64 0.4× 176 1.3× 12 1.2k

Countries citing papers authored by Jinyao Mo

Since Specialization
Citations

This map shows the geographic impact of Jinyao Mo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jinyao Mo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jinyao Mo more than expected).

Fields of papers citing papers by Jinyao Mo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jinyao Mo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jinyao Mo. The network helps show where Jinyao Mo may publish in the future.

Co-authorship network of co-authors of Jinyao Mo

This figure shows the co-authorship network connecting the top 25 collaborators of Jinyao Mo. A scholar is included among the top collaborators of Jinyao Mo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jinyao Mo. Jinyao Mo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Duncan, Joseph A., Alex Petrucelli, Lu Zhang, et al.. (2020). NLRC3, a Member of the NLR Family of Proteins, Is a Negative Regulator of Innate Immune Signaling Induced by the DNA Sensor STING. UNC Libraries. 2 indexed citations
5.
Li, Xin, Meng Deng, Alex Petrucelli, et al.. (2019). Viral DNA Binding to NLRC3, an Inhibitory Nucleic Acid Sensor, Unleashes STING, a Cyclic Dinucleotide Receptor that Activates Type I Interferon. Immunity. 50(3). 591–599.e6. 66 indexed citations
6.
Feng, Hui, Erik M. Lenarcic, Daisuke Yamane, et al.. (2017). NLRX1 promotes immediate IRF1-directed antiviral responses by limiting dsRNA-activated translational inhibition mediated by PKR. Nature Immunology. 18(12). 1299–1309. 71 indexed citations
7.
Yan, Jun, Feng Yan, Zhijun Li, et al.. (2014). The 3M Complex Maintains Microtubule and Genome Integrity. Molecular Cell. 54(5). 791–804. 66 indexed citations
8.
Zhang, Lu, Jinyao Mo, Karen V. Swanson, et al.. (2014). NLRC3, a Member of the NLR Family of Proteins, Is a Negative Regulator of Innate Immune Signaling Induced by the DNA Sensor STING. Immunity. 40(3). 329–341. 225 indexed citations
9.
Mo, Jinyao & Joseph A. Duncan. (2013). Assessing ATP Binding and Hydrolysis by NLR Proteins. Methods in molecular biology. 1040. 153–168. 9 indexed citations
10.
Mo, Jinyao, Joseph P. Boyle, Christopher B. Howard, et al.. (2012). Pathogen Sensing by Nucleotide-binding Oligomerization Domain-containing Protein 2 (NOD2) Is Mediated by Direct Binding to Muramyl Dipeptide and ATP. Journal of Biological Chemistry. 287(27). 23057–23067. 134 indexed citations
11.
Mo, Jinyao, Yajuan Xia, Timothy J. Wade, et al.. (2011). Altered Gene Expression by Low-Dose Arsenic Exposure in Humans and Cultured Cardiomyocytes: Assessment by Real-Time PCR Arrays. International Journal of Environmental Research and Public Health. 8(6). 2090–2108. 29 indexed citations
12.
Mo, Jinyao, et al.. (2008). Elevated Human Telomerase Reverse Transcriptase Gene Expression in Blood Cells Associated with Chronic Arsenic Exposure in Inner Mongolia, China. Environmental Health Perspectives. 117(3). 354–360. 31 indexed citations
13.
Mo, Jinyao, Yajuan Xia, Timothy J. Wade, et al.. (2006). Chronic Arsenic Exposure and Oxidative Stress: OGG1 Expression and Arsenic Exposure, Nail Selenium, and Skin Hyperkeratosis in Inner Mongolia. Environmental Health Perspectives. 114(6). 835–841. 83 indexed citations
14.
Chen, Wei, Kellye C. Kirkbride, Tam How, et al.. (2003). ß-Arrestin 2 Mediates Endocytosis of Type III TGF-ß Receptor and Down-Regulation of Its Signaling. Science. 301(5638). 1394–1397. 207 indexed citations
15.
Mo, Jinyao, Shijing Fang, Wei Chen, & Gerard C. Blobe. (2002). Regulation of ALK-1 Signaling by the Nuclear Receptor LXRβ. Journal of Biological Chemistry. 277(52). 50788–50794. 25 indexed citations
16.
Hara, Ryujiro, Jinyao Mo, & Aziz Sancar. (2000). DNA Damage in the Nucleosome Core Is Refractory to Repair by Human Excision Nuclease. Molecular and Cellular Biology. 20(24). 9173–9181. 151 indexed citations
17.
Liu, Li, Jinyao Mo, Esther Rodríguez‐Belmonte, & Marietta Lee. (2000). Identification of a Fourth Subunit of Mammalian DNA Polymerase δ. Journal of Biological Chemistry. 275(25). 18739–18744. 91 indexed citations
18.
Wu, Sheng‐Ming, et al.. (1998). Characterization of the p125 Subunit of Human DNA Polymerase δ and Its Deletion Mutants. Journal of Biological Chemistry. 273(16). 9561–9569. 23 indexed citations
19.
Mo, Jinyao & Roel M. Schaaper. (1996). Fidelity and Error Specificity of the α Catalytic Subunit of Escherichia coli DNA Polymerase III. Journal of Biological Chemistry. 271(31). 18947–18953. 39 indexed citations
20.
Mo, Jinyao, et al.. (1991). Mutational specificity of the dnaE173 mutator associated with a defect in the catalytic subunit of DNA polymerase III of Escherichia coli. Journal of Molecular Biology. 222(4). 925–936. 72 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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