Jin‐Wu Tsai

3.8k total citations
57 papers, 2.6k citations indexed

About

Jin‐Wu Tsai is a scholar working on Molecular Biology, Cell Biology and Genetics. According to data from OpenAlex, Jin‐Wu Tsai has authored 57 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Molecular Biology, 20 papers in Cell Biology and 17 papers in Genetics. Recurrent topics in Jin‐Wu Tsai's work include Neurogenesis and neuroplasticity mechanisms (14 papers), Microtubule and mitosis dynamics (10 papers) and Genetics and Neurodevelopmental Disorders (8 papers). Jin‐Wu Tsai is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (14 papers), Microtubule and mitosis dynamics (10 papers) and Genetics and Neurodevelopmental Disorders (8 papers). Jin‐Wu Tsai collaborates with scholars based in Taiwan, United States and Japan. Jin‐Wu Tsai's co-authors include Richard B. Vallee, Arnold R. Kriegstein, Xiaoqun Wang, K. Helen Bremner, Yu Chen, Janice H. Imai, Shahrnaz Kemal, Guang Yang, Wen‐Biao Gan and Lei Ma and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Jin‐Wu Tsai

53 papers receiving 2.6k citations

Peers

Jin‐Wu Tsai
Takeshi Kawauchi Japan
Teruyuki Tanaka Japan
Julian Ik‐Tsen Heng Australia
Tianzhi Shu United States
Yves Jossin Belgium
Michael Piper Australia
Daijiro Konno Japan
Pierre Billuart France
Tetsuji Mori Japan
Artur Kania Canada
Takeshi Kawauchi Japan
Jin‐Wu Tsai
Citations per year, relative to Jin‐Wu Tsai Jin‐Wu Tsai (= 1×) peers Takeshi Kawauchi

Countries citing papers authored by Jin‐Wu Tsai

Since Specialization
Citations

This map shows the geographic impact of Jin‐Wu Tsai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jin‐Wu Tsai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jin‐Wu Tsai more than expected).

Fields of papers citing papers by Jin‐Wu Tsai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jin‐Wu Tsai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jin‐Wu Tsai. The network helps show where Jin‐Wu Tsai may publish in the future.

Co-authorship network of co-authors of Jin‐Wu Tsai

This figure shows the co-authorship network connecting the top 25 collaborators of Jin‐Wu Tsai. A scholar is included among the top collaborators of Jin‐Wu Tsai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jin‐Wu Tsai. Jin‐Wu Tsai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhao, Hongjun, et al.. (2025). Functional defects in FOXG1 variants predict the severity of brain anomalies in FOXG1 syndrome. Molecular Psychiatry. 30(10). 4824–4835.
2.
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Li, Yueru, Yu‐Wen Cheng, Yuting Wang, et al.. (2024). Regulation of primary cilia disassembly through HUWE1-mediated TTBK2 degradation plays a crucial role in cerebellar development and medulloblastoma growth. Cell Death and Differentiation. 31(10). 1349–1361. 5 indexed citations
4.
Iemura, Kenji, Satoshi Miyashita, Mikio Hoshino, et al.. (2024). Kinesin-like motor protein KIF23 maintains neural stem and progenitor cell pools in the developing cortex. The EMBO Journal. 44(2). 331–355. 5 indexed citations
5.
Li, Yueru, et al.. (2024). Pathogenic SHQ1 variants result in disruptions to neuronal development and the dopaminergic pathway. Experimental Neurology. 382. 114968–114968.
6.
Tsai, Meng‐Han, Shih‐Ying Chen, Meng‐Ying Hsieh, et al.. (2023). A lissencephaly-associated BAIAP2 variant causes defects in neuronal migration during brain development. Development. 151(2). 1 indexed citations
7.
Lee, Kuang‐Yung, et al.. (2023). Muscleblind‐like 2 knockout shifts adducin 1 isoform expression and alters dendritic spine dynamics of cortical neurons during brain development. Neuropathology and Applied Neurobiology. 49(2). e12890–e12890. 5 indexed citations
8.
Liu, Yo‐Tsen, et al.. (2022). Novel lissencephaly‐associated DCX variants in the C‐terminal DCX domain affect microtubule binding and dynamics. Epilepsia. 63(5). 1253–1265. 8 indexed citations
9.
Guarnieri, Fabrizia Claudia, et al.. (2021). An Emerging Role of PRRT2 in Regulating Growth Cone Morphology. Cells. 10(10). 2666–2666. 3 indexed citations
10.
Takeda, Hiroki, Chun-Ying Huang, Ru Xiao, et al.. (2020). Efficient In Utero Gene Transfer to the Mammalian Inner Ears by the Synthetic Adeno-Associated Viral Vector Anc80L65. Molecular Therapy — Methods & Clinical Development. 18. 493–500. 21 indexed citations
11.
Tsai, Meng‐Han, et al.. (2020). Impairment in dynein-mediated nuclear translocation by BICD2 C-terminal truncation leads to neuronal migration defect and human brain malformation. Acta Neuropathologica Communications. 8(1). 106–106. 23 indexed citations
12.
Tsai, Meng‐Han, Mei‐Hsin Hsu, Wei‐Szu Liu, et al.. (2019). PRRT2 missense mutations cluster near C‐terminus and frequently lead to protein mislocalization. Epilepsia. 60(5). 807–817. 17 indexed citations
13.
Lin, Kon-Ping, et al.. (2019). Cellular secretion and cytotoxicity of transthyretin mutant proteins underlie late-onset amyloidosis and neurodegeneration. Cellular and Molecular Life Sciences. 77(7). 1421–1434. 13 indexed citations
14.
Zanini, Marco Antônio, Hua Yu, Audrey Mercier, et al.. (2019). Atoh1 Controls Primary Cilia Formation to Allow for SHH-Triggered Granule Neuron Progenitor Proliferation. Developmental Cell. 48(2). 184–199.e5. 50 indexed citations
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Liu, Chen, et al.. (2019). Multiple Functions of KBP in Neural Development Underlie Brain Anomalies in Goldberg-Shprintzen Syndrome. Frontiers in Molecular Neuroscience. 12. 265–265. 7 indexed citations
17.
Wang, Chun‐Hung, et al.. (2018). Gli2 modulates cell cycle re-entry through autophagy-mediated regulation of the length of primary cilia. Journal of Cell Science. 131(24). 17 indexed citations
18.
Hur, Sung Sik, Chia‐Ming Chang, Hsiao‐Hui Lee, et al.. (2018). Lis1 dysfunction leads to traction force reduction and cytoskeletal disorganization during cell migration. Biochemical and Biophysical Research Communications. 497(3). 869–875. 23 indexed citations
19.
Tsai, Jin‐Wu, Pei‐Ching Chang, Pei‐Lin Cheng, et al.. (2017). Ascl1 promotes tangential migration and confines migratory routes by induction of Ephb2 in the telencephalon. Scientific Reports. 7(1). 42895–42895. 6 indexed citations
20.
Vallee, Richard B. & Jin‐Wu Tsai. (2006). The cellular roles of the lissencephaly gene LIS1, and what they tell us aboutbrain development. Genes & Development. 20(11). 1384–1393. 114 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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