Jin Ock Kim

836 total citations
9 papers, 419 citations indexed

About

Jin Ock Kim is a scholar working on Molecular Biology, Cancer Research and Cell Biology. According to data from OpenAlex, Jin Ock Kim has authored 9 papers receiving a total of 419 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 3 papers in Cancer Research and 2 papers in Cell Biology. Recurrent topics in Jin Ock Kim's work include MicroRNA in disease regulation (3 papers), RNA regulation and disease (2 papers) and Circular RNAs in diseases (2 papers). Jin Ock Kim is often cited by papers focused on MicroRNA in disease regulation (3 papers), RNA regulation and disease (2 papers) and Circular RNAs in diseases (2 papers). Jin Ock Kim collaborates with scholars based in South Korea and United States. Jin Ock Kim's co-authors include Do Han Kim, Dong Woo Song, Eun Jeong Kwon, Jae Yong Ryu, Chunghee Cho, Hong Ki Song, Jong‐Sub Lee, Yong Sook Kim, Hyuck Hoon Kwon and Sungeun Kim and has published in prestigious journals such as PLoS ONE, Scientific Reports and Biochemical Journal.

In The Last Decade

Jin Ock Kim

9 papers receiving 411 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jin Ock Kim South Korea	9	295	144	54	50	48	9	419
Xiaowu Sheng China	10	330 1.1×	157 1.1×	18 0.3×	29 0.6×	26 0.5×	15	464
Miho Harada Japan	11	167 0.6×	102 0.7×	26 0.5×	35 0.7×	75 1.6×	27	368
Fabiao Zhang China	10	132 0.4×	82 0.6×	35 0.6×	42 0.8×	9 0.2×	23	331
Rei Nakano Japan	14	182 0.6×	115 0.8×	10 0.2×	73 1.5×	24 0.5×	27	417
Charlotte Tacheau France	10	205 0.7×	36 0.3×	11 0.2×	41 0.8×	77 1.6×	11	387
Eneda Hoxha United States	7	244 0.8×	51 0.4×	136 2.5×	64 1.3×	8 0.2×	11	426
Zhaofu Liao China	7	217 0.7×	95 0.7×	95 1.8×	24 0.5×	8 0.2×	12	326
Francesca Varrone Italy	7	440 1.5×	300 2.1×	58 1.1×	41 0.8×	4 0.1×	9	553
Samah Rekima France	10	171 0.6×	76 0.5×	20 0.4×	39 0.8×	4 0.1×	14	436

Countries citing papers authored by Jin Ock Kim

Since Specialization

Citations

This map shows the geographic impact of Jin Ock Kim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jin Ock Kim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jin Ock Kim more than expected).

Fields of papers citing papers by Jin Ock Kim

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jin Ock Kim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jin Ock Kim. The network helps show where Jin Ock Kim may publish in the future.

Co-authorship network of co-authors of Jin Ock Kim

This figure shows the co-authorship network connecting the top 25 collaborators of Jin Ock Kim. A scholar is included among the top collaborators of Jin Ock Kim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jin Ock Kim. Jin Ock Kim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

9 of 9 papers shown

Shin, Kyong‐Oh, Dae Hyun Ha, Jin Ock Kim, et al.. (2020). Exosomes from Human Adipose Tissue-Derived Mesenchymal Stem Cells Promote Epidermal Barrier Repair by Inducing de Novo Synthesis of Ceramides in Atopic Dermatitis. Cells. 9(3). 680–680. 147 indexed citations

Kim, Jin Ock, Tae-Yong Kim, Kyoung Jin Nho, et al.. (2018). A novel system-level approach using RNA-sequencing data identifies miR-30-5p and miR-142a-5p as key regulators of apoptosis in myocardial infarction. Scientific Reports. 8(1). 14638–14638. 18 indexed citations

Sreenivasaiah, Pradeep Kumar, Jin Ock Kim, Mi Young Lee, et al.. (2017). Tauroursodeoxycholic acid (TUDCA) attenuates pressure overload-induced cardiac remodeling by reducing endoplasmic reticulum stress. PLoS ONE. 12(4). e0176071–e0176071. 55 indexed citations

Huang, Mei, et al.. (2017). STIM2 regulates both intracellular Ca2+ distribution and Ca2+ movement in skeletal myotubes. Scientific Reports. 7(1). 17936–17936. 17 indexed citations

Lee, Jong‐Sub, et al.. (2017). miR-374 promotes myocardial hypertrophy by negatively regulating vascular endothelial growth factor receptor-1 signaling. BMB Reports. 50(4). 208–213. 16 indexed citations

Kim, Jin Ock, Eun Jeong Kwon, Dong Woo Song, Jong‐Sub Lee, & Do Han Kim. (2016). miR-185 inhibits endoplasmic reticulum stress-induced apoptosis by targeting Na⁺/H⁺exchanger-1 in the heart. BMB Reports. 49(4). 208–213. 29 indexed citations

Kim, Jin Ock, et al.. (2015). miR-185 Plays an Anti-Hypertrophic Role in the Heart via Multiple Targets in the Calcium-Signaling Pathways. PLoS ONE. 10(3). e0122509–e0122509. 60 indexed citations

Kim, Tae-Yong, et al.. (2014). Pressure-Overload Cardiac Hypertrophy Is Associated with Distinct Alternative Splicing Due to Altered Expression of Splicing Factors. Molecules and Cells. 37(1). 81–87. 16 indexed citations

Song, Dong Woo, Jae Yong Ryu, Jin Ock Kim, Eun Jeong Kwon, & Do Han Kim. (2013). The miR-19a/b family positively regulates cardiomyocyte hypertrophy by targeting atrogin-1 and MuRF-1. Biochemical Journal. 457(1). 151–162. 61 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact