Jie Mo

717 total citations
26 papers, 473 citations indexed

About

Jie Mo is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Jie Mo has authored 26 papers receiving a total of 473 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 7 papers in Oncology and 6 papers in Cancer Research. Recurrent topics in Jie Mo's work include Ubiquitin and proteasome pathways (5 papers), Cancer Mechanisms and Therapy (3 papers) and Cancer, Hypoxia, and Metabolism (3 papers). Jie Mo is often cited by papers focused on Ubiquitin and proteasome pathways (5 papers), Cancer Mechanisms and Therapy (3 papers) and Cancer, Hypoxia, and Metabolism (3 papers). Jie Mo collaborates with scholars based in China, Taiwan and Germany. Jie Mo's co-authors include Bixiang Zhang, Jin Chen, Su Chen, Huifang Liang, Zhibin Liao, Pengcheng Li, Xiaoping Chen, Bixiang Zhang, Qiumeng Liu and Xiaoping Chen and has published in prestigious journals such as SHILAP Revista de lepidopterología, Hepatology and Oncogene.

In The Last Decade

Jie Mo

24 papers receiving 468 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jie Mo China 12 327 102 67 60 52 26 473
Laura Bachelot France 8 379 1.2× 129 1.3× 48 0.7× 79 1.3× 35 0.7× 10 543
Chien-Yu Chen United States 5 314 1.0× 157 1.5× 60 0.9× 86 1.4× 31 0.6× 7 475
Aktar Ali United States 8 251 0.8× 122 1.2× 98 1.5× 62 1.0× 34 0.7× 14 432
Xiaolan Chang China 15 316 1.0× 171 1.7× 49 0.7× 62 1.0× 24 0.5× 23 522
Ying Song China 14 303 0.9× 122 1.2× 50 0.7× 121 2.0× 36 0.7× 28 481
Dinesh Babu Uthaya Kumar United States 10 221 0.7× 93 0.9× 65 1.0× 58 1.0× 21 0.4× 21 411
Yunshin Jung United States 12 293 0.9× 69 0.7× 81 1.2× 47 0.8× 36 0.7× 19 493
Zhen Cai China 7 475 1.5× 149 1.5× 57 0.9× 81 1.4× 26 0.5× 9 593
Chen Shen China 12 537 1.6× 193 1.9× 58 0.9× 47 0.8× 56 1.1× 21 687

Countries citing papers authored by Jie Mo

Since Specialization
Citations

This map shows the geographic impact of Jie Mo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jie Mo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jie Mo more than expected).

Fields of papers citing papers by Jie Mo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jie Mo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jie Mo. The network helps show where Jie Mo may publish in the future.

Co-authorship network of co-authors of Jie Mo

This figure shows the co-authorship network connecting the top 25 collaborators of Jie Mo. A scholar is included among the top collaborators of Jie Mo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jie Mo. Jie Mo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mo, Jie, Su Chen, Qiumeng Liu, et al.. (2025). Targeting FAM134B-DDX3X axis inhibiting AKT signaling in hepatocellular carcinoma. Cell Death and Disease. 16(1). 797–797.
2.
Wang, Weijian, Jie Mo, Su Chen, et al.. (2025). NEK7-induced phosphorylation of EGFR on serine 1070 drives the acquired lenvatinib resistance in hepatocellular carcinoma. Hepatology. 1 indexed citations
3.
Mo, Jie, Su Chen, Pengcheng Li, et al.. (2024). CKAP4 in hepatocellular carcinoma: competitive RETREG1/FAM134B binding, reticulophagy regulation, and cancer progression. Autophagy. 21(4). 840–859. 3 indexed citations
4.
Chen, Su, et al.. (2024). Integrinβ-1 in disorders and cancers: molecular mechanisms and therapeutic targets. Cell Communication and Signaling. 22(1). 71–71. 32 indexed citations
5.
Chen, Su, et al.. (2024). SP1‐activated USP27X‐AS1 promotes hepatocellular carcinoma progression via USP7‐mediated AKT stabilisation. Clinical and Translational Medicine. 14(1). e1563–e1563. 6 indexed citations
6.
Liu, Qiumeng, Hailing Liu, Jie Mo, et al.. (2024). Disrupting CCDC137-mediated LZTS2 and β-TrCP interaction in the nucleus inhibits hepatocellular carcinoma development via β-catenin and AKT. Cell Death and Differentiation. 32(1). 134–148. 3 indexed citations
7.
Yuan, Chaoyi, Guan-nan Jin, Pengcheng Li, et al.. (2023). Tubular cell transcriptional intermediary factor 1γ deficiency exacerbates kidney injury-induced tubular cell polyploidy and fibrosis. Kidney International. 104(4). 769–786. 4 indexed citations
9.
Chen, Jin, Qiumeng Liu, Jie Mo, et al.. (2023). USP11 potentiates HGF/AKT signaling and drives metastasis in hepatocellular carcinoma. Oncogene. 43(2). 123–135. 12 indexed citations
10.
Yang, Zhenhua, Qiumeng Liu, Lei Xu, et al.. (2023). The Role of Nuclear Receptors in the Pathogenesis and Treatment of Non-alcoholic Fatty Liver Disease. International Journal of Biological Sciences. 20(1). 113–126. 23 indexed citations
11.
Chen, Jin, Hanhua Dong, Qiumeng Liu, et al.. (2022). MTDH-stabilized DDX17 promotes tumor initiation and progression through interacting with YB1 to induce EGFR transcription in Hepatocellular Carcinoma. Oncogene. 42(3). 169–183. 13 indexed citations
12.
Chen, Su, Weijian Wang, Jie Mo, et al.. (2022). Long noncoding RNA HOXC-AS3 interacts with CDK2 to promote proliferation in hepatocellular carcinoma. Biomarker Research. 10(1). 65–65. 6 indexed citations
13.
Mo, Jie, et al.. (2022). The RNA m6A writer WTAP in diseases: structure, roles, and mechanisms. Cell Death and Disease. 13(10). 852–852. 87 indexed citations
14.
Liu, Yachong, Zhibin Liao, Jie Mo, et al.. (2022). The role of YAP1 in liver cancer stem cells: proven and potential mechanisms. Biomarker Research. 10(1). 42–42. 13 indexed citations
15.
Mo, Jie, Huifang Liang, Su Chen, et al.. (2021). DDX3X: structure, physiologic functions and cancer. Molecular Cancer. 20(1). 38–38. 148 indexed citations
16.
Mo, Jie, Jin Chen, & Bixiang Zhang. (2020). Critical roles of FAM134B in ER-phagy and diseases. Cell Death and Disease. 11(11). 983–983. 45 indexed citations
17.
Chen, Jin, Qiumeng Liu, Long Zhang, et al.. (2019). DEPTOR induces a partial epithelial-to-mesenchymal transition and metastasis via autocrine TGFβ1 signaling and is associated with poor prognosis in hepatocellular carcinoma. Journal of Experimental & Clinical Cancer Research. 38(1). 273–273. 22 indexed citations
18.
Mo, Jie, et al.. (2015). Effect of lit-moxa stimulation of Guanyuan (CV 4) acupoint on levels of lactic acid and super-oxide dismutase in skeletal muscle of rats. Journal of Traditional Chinese Medicine. 35(2). 234–237. 3 indexed citations
19.
Chen, Huan, Jie Mo, Li Luo, et al.. (2014). Effects of Preventive Acupuncture and Moxibustion on Fat Accumulation, Blood Lipid, and Uterus E2 of Menopause Rats. Evidence-based Complementary and Alternative Medicine. 2014(1). 621975–621975. 3 indexed citations
20.
Zhai, Jinghui, et al.. (2010). Changes of HSP70, HSP70 mRNA, SOD, NOS in uterus of natural climacteric rats and the intervention of preventive moxibustion on Guanyuan point.. Zhonghua zhongyiyao zazhi. 25(1). 46–49. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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