Jeremy Mills

1.4k total citations
47 papers, 1.1k citations indexed

About

Jeremy Mills is a scholar working on Molecular Biology, Cancer Research and Hematology. According to data from OpenAlex, Jeremy Mills has authored 47 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 9 papers in Cancer Research and 7 papers in Hematology. Recurrent topics in Jeremy Mills's work include Carcinogens and Genotoxicity Assessment (7 papers), Hemoglobinopathies and Related Disorders (5 papers) and Pharmacogenetics and Drug Metabolism (4 papers). Jeremy Mills is often cited by papers focused on Carcinogens and Genotoxicity Assessment (7 papers), Hemoglobinopathies and Related Disorders (5 papers) and Pharmacogenetics and Drug Metabolism (4 papers). Jeremy Mills collaborates with scholars based in United Kingdom, United States and Saudi Arabia. Jeremy Mills's co-authors include Randy L. Jirtle, Melvin E. Andersen, Ravi S. Chari, Alexander Dodoo, William Kudzi, Ivan Boyer, Linda S. Birnbaum, M.N. Gould, William F. Greenlee and Angus T. De Souza and has published in prestigious journals such as Journal of Neuroscience, Oncogene and Annals of Surgery.

In The Last Decade

Jeremy Mills

42 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jeremy Mills United Kingdom 17 432 309 225 223 209 47 1.1k
Nadine Dragin France 22 394 0.9× 263 0.9× 324 1.4× 216 1.0× 280 1.3× 32 1.6k
Martin C. Dyroff United States 15 418 1.0× 323 1.0× 105 0.5× 190 0.9× 153 0.7× 29 1.0k
Carl L. Alden United States 22 397 0.9× 441 1.4× 276 1.2× 175 0.8× 180 0.9× 65 1.4k
Jaspreet S. Sidhu United States 21 879 2.0× 208 0.7× 144 0.6× 256 1.1× 476 2.3× 34 1.6k
Xinsheng Gu China 16 472 1.1× 183 0.6× 60 0.3× 324 1.5× 310 1.5× 31 1.1k
Adrian J. Fretland United States 22 1.1k 2.6× 690 2.2× 97 0.4× 376 1.7× 437 2.1× 52 2.1k
Nghia Nguyen United States 19 589 1.4× 144 0.5× 107 0.5× 382 1.7× 436 2.1× 31 1.4k
Peter Münzel Germany 22 507 1.2× 232 0.8× 176 0.8× 397 1.8× 553 2.6× 38 1.3k
Gang Luo China 17 468 1.1× 94 0.3× 37 0.2× 176 0.8× 324 1.6× 60 1.3k
T.C. Orton United Kingdom 16 633 1.5× 271 0.9× 205 0.9× 274 1.2× 292 1.4× 40 1.4k

Countries citing papers authored by Jeremy Mills

Since Specialization
Citations

This map shows the geographic impact of Jeremy Mills's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jeremy Mills with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jeremy Mills more than expected).

Fields of papers citing papers by Jeremy Mills

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jeremy Mills. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jeremy Mills. The network helps show where Jeremy Mills may publish in the future.

Co-authorship network of co-authors of Jeremy Mills

This figure shows the co-authorship network connecting the top 25 collaborators of Jeremy Mills. A scholar is included among the top collaborators of Jeremy Mills based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jeremy Mills. Jeremy Mills is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Mohamed, Magdi Awadalla, Tilal Elsaman, Abozer Y. Elderdery, et al.. (2024). Unveiling the Anticancer Potential: Computational Exploration of Nitrogenated Derivatives of (+)-Pancratistatin as Topoisomerase I Inhibitors. International Journal of Molecular Sciences. 25(19). 10779–10779. 4 indexed citations
4.
Elderdery, Abozer Y., et al.. (2022). Impact of Methionine Synthase Reductase Polymorphisms in Chronic Myeloid Leukemia Patients. Genes. 13(10). 1729–1729. 3 indexed citations
5.
Elderdery, Abozer Y., et al.. (2019). Detection of Genetic polymorphisms of Methylene tetrahydrofolate reductase among Sudanese patients with chronic myeloid leukemia. 13(4). 1325–1329. 2 indexed citations
6.
Daak, Ahmed, Abozer Y. Elderdery, Jeremy Mills, et al.. (2015). Omega 3 (n − 3) fatty acids down-regulate nuclear factor-kappa B (NF-κB) gene and blood cell adhesion molecule expression in patients with homozygous sickle cell disease. Blood Cells Molecules and Diseases. 55(1). 48–55. 43 indexed citations
7.
Elderdery, Abozer Y., et al.. (2014). First recorded case of haemoglobin SC in sudan. 2(1). 2–2.
8.
Elderdery, Abozer Y., et al.. (2012). BRCA 1 and 2 Mutations in Sudanese Secondary School Girls with Known Breast Cancer in Their Families. International Journal of Health Sciences. 6(1). 63–71. 8 indexed citations
9.
Elderdery, Abozer Y., et al.. (2011). Tribal distribution of haemoglobinopathies in a Sudanese patient population. 2(4). 31–37. 10 indexed citations
10.
Kudzi, William, Alexander Dodoo, & Jeremy Mills. (2009). Characterisation of CYP2C8, CYP2C9 and CYP2C19 polymorphisms in a Ghanaian population. BMC Medical Genetics. 10(1). 124–124. 72 indexed citations
11.
Arkle, S., et al.. (2006). Regulation and induction of CYP3A11, CYP3A13 and CYP3A25 in C57BL/6J mouse liver. Archives of Biochemistry and Biophysics. 457(1). 105–110. 36 indexed citations
12.
Andersen, Melvin E., et al.. (1997). A Multicompartment Geometric Model of the Liver in Relation to Regional Induction of Cytochrome P450s. Toxicology and Applied Pharmacology. 144(1). 135–144. 41 indexed citations
13.
Mansbach, Jonathan M., Jeremy Mills, Ivan Boyer, et al.. (1996). SHORT COMMUNICATION: Phenobarbital selectively promotes initiated cells with reduced TGFβ receptor levels. Carcinogenesis. 17(1). 171–174. 26 indexed citations
14.
Moser, Glenda J., Douglas C. Wolf, Andrew M. Standeven, et al.. (1996). Cell Proliferation and regulation of negative growth factors in mouseliver foci. Carcinogenesis. 17(9). 1835–1840. 19 indexed citations
15.
Chari, Ravi S., Feng‐Ming Kong, Jeremy Mills, et al.. (1995). Transforming Growth Factor-Beta Receptors and Mannose 6-Phosphate/Insulin-Like Growth Factor-II Receptor Expression in Human Hepatocellular Carcinoma. Annals of Surgery. 222(2). 171–178. 65 indexed citations
16.
Mills, Jeremy, et al.. (1993). The Dynamically Reconfigurable Assembly System: Implementation Issues. 27. 1 indexed citations
17.
Mills, Jeremy & Melvin E. Andersen. (1993). Dioxin hepatic carcinogenesis: Biologically motivated modeling and risk assessment. Toxicology Letters. 68(1-2). 177–189. 18 indexed citations
18.
Andersen, Melvin E., Jeremy Mills, Michael L. Gargas, et al.. (1993). Modeling Receptor‐Mediated Processes with Dioxin: Implications for Pharmacokinetics and Risk Assessment. Risk Analysis. 13(1). 25–36. 108 indexed citations
19.
Mills, Jeremy, Michael L. Gargas, & Melvin E. Andersen. (1992). Biological and physiological factors involved in disposition of dioxin and related compounds. Chemosphere. 25(1-2). 3–6. 3 indexed citations
20.
Mills, Jeremy, N. P. Bishun, Ronald W. Raven, & D. Williams. (1974). The effect of estradiol and progesterone on various gynecological tissues in culture. Journal of Surgical Oncology. 6(6). 471–479. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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