Janson W.T. Tse

414 total citations
8 papers, 260 citations indexed

About

Janson W.T. Tse is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Surgery. According to data from OpenAlex, Janson W.T. Tse has authored 8 papers receiving a total of 260 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 2 papers in Pathology and Forensic Medicine and 1 paper in Surgery. Recurrent topics in Janson W.T. Tse's work include Ubiquitin and proteasome pathways (2 papers), Histone Deacetylase Inhibitors Research (2 papers) and Cancer-related gene regulation (2 papers). Janson W.T. Tse is often cited by papers focused on Ubiquitin and proteasome pathways (2 papers), Histone Deacetylase Inhibitors Research (2 papers) and Cancer-related gene regulation (2 papers). Janson W.T. Tse collaborates with scholars based in Australia, United States and Norway. Janson W.T. Tse's co-authors include John M. Mariadason, Laura J. Jenkins, Fiona Chionh, Anderly C. Chüeh, Lars Tögel, Michael Büchert, Matthias Ernst, W. Douglas Fairlie, Stefan Thiem and Ian Y. Luk and has published in prestigious journals such as Clinical Cancer Research, Antioxidants and Redox Signaling and PROTEOMICS.

In The Last Decade

Janson W.T. Tse

8 papers receiving 259 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Janson W.T. Tse Australia	6	199	78	64	32	20	8	260
Vincent Yeung United States	5	206 1.0×	41 0.5×	98 1.5×	27 0.8×	20 1.0×	14	275
Alexander E. Kudinov United States	5	235 1.2×	94 1.2×	70 1.1×	17 0.5×	21 1.1×	6	314
Guoying Miao China	7	150 0.8×	51 0.7×	71 1.1×	23 0.7×	11 0.6×	12	230
George A. R. Wiggins New Zealand	8	166 0.8×	71 0.9×	64 1.0×	24 0.8×	23 1.1×	17	260
Xian-Zi Yang China	8	225 1.1×	95 1.2×	95 1.5×	21 0.7×	17 0.8×	8	320
Tanis Godwin New Zealand	8	173 0.9×	46 0.6×	82 1.3×	43 1.3×	34 1.7×	10	244
Jou‐Ho Shih Taiwan	8	222 1.1×	112 1.4×	77 1.2×	18 0.6×	11 0.6×	10	292
Antonella Di Costanzo Italy	10	249 1.3×	77 1.0×	68 1.1×	18 0.6×	8 0.4×	11	312
Xuetian Zhang China	8	242 1.2×	86 1.1×	66 1.0×	14 0.4×	17 0.8×	10	322
Mariko Nakano‐Okuno United States	7	171 0.9×	67 0.9×	46 0.7×	22 0.7×	8 0.4×	10	234

Countries citing papers authored by Janson W.T. Tse

Since Specialization

Citations

This map shows the geographic impact of Janson W.T. Tse's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Janson W.T. Tse with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Janson W.T. Tse more than expected).

Fields of papers citing papers by Janson W.T. Tse

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Janson W.T. Tse. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Janson W.T. Tse. The network helps show where Janson W.T. Tse may publish in the future.

Co-authorship network of co-authors of Janson W.T. Tse

This figure shows the co-authorship network connecting the top 25 collaborators of Janson W.T. Tse. A scholar is included among the top collaborators of Janson W.T. Tse based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Janson W.T. Tse. Janson W.T. Tse is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Afshar‐Sterle, Shoukat, Janson W.T. Tse, Stefanie Fischer, et al.. (2024). DCLK1 induces a pro-tumorigenic phenotype to drive gastric cancer progression. Science Signaling. 17(854). eabq4888–eabq4888. 3 indexed citations

2.

Tse, Janson W.T., et al.. (2023). A Mouse Model for the Rapid and Binomial Assessment of Putative WNT/β-Catenin Signalling Inhibitors. Biomedicines. 11(10). 2719–2719. 1 indexed citations

3.

Tse, Janson W.T., et al.. (2022). Onco-miR-21 Promotes Stat3-Dependent Gastric Cancer Progression. Cancers. 14(2). 264–264. 26 indexed citations

4.

Afshar‐Sterle, Shoukat, Alin Rai, Haoyun Fang, et al.. (2021). Cancer stem cell marker DCLK1 reprograms small extracellular vesicles toward migratory phenotype in gastric cancer cells. PROTEOMICS. 21(13-14). e2000098–e2000098. 20 indexed citations

5.

Chüeh, Anderly C., Janson W.T. Tse, Michael Dickinson, et al.. (2017). ATF3 Repression of BCL-XL Determines Apoptotic Sensitivity to HDAC Inhibitors across Tumor Types. Clinical Cancer Research. 23(18). 5573–5584. 44 indexed citations

6.

Tse, Janson W.T., Laura J. Jenkins, Fiona Chionh, & John M. Mariadason. (2017). Aberrant DNA Methylation in Colorectal Cancer: What Should We Target?. Trends in cancer. 3(10). 698–712. 87 indexed citations

7.

Chionh, Fiona, Hoanh Tran, Janson W.T. Tse, et al.. (2017). K‐Ras mutation and amplification status is predictive of resistance and high basal pAKT is predictive of sensitivity to everolimus in biliary tract cancer cell lines. Molecular Oncology. 11(9). 1130–1142. 17 indexed citations

8.

Chüeh, Anderly C., Janson W.T. Tse, Lars Tögel, & John M. Mariadason. (2014). Mechanisms of Histone Deacetylase Inhibitor-Regulated Gene Expression in Cancer Cells. Antioxidants and Redox Signaling. 23(1). 66–84. 62 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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