Jannis Meents

903 total citations
20 papers, 565 citations indexed

About

Jannis Meents is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Jannis Meents has authored 20 papers receiving a total of 565 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 7 papers in Cellular and Molecular Neuroscience and 6 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Jannis Meents's work include Ion channel regulation and function (9 papers), Cardiac electrophysiology and arrhythmias (6 papers) and Pain Mechanisms and Treatments (5 papers). Jannis Meents is often cited by papers focused on Ion channel regulation and function (9 papers), Cardiac electrophysiology and arrhythmias (6 papers) and Pain Mechanisms and Treatments (5 papers). Jannis Meents collaborates with scholars based in Germany, United Kingdom and Austria. Jannis Meents's co-authors include Michael J. M. Fischer, Lars Neeb, Uwe Reuter, Cosmin I. Ciotu, Peter A. McNaughton, Angelika Lampert, Ralf Hausmann, Petra Hautvast, Günther Schmalzing and Jan Hoffmann and has published in prestigious journals such as Journal of Biological Chemistry, SHILAP Revista de lepidopterología and PLoS ONE.

In The Last Decade

Jannis Meents

20 papers receiving 560 citations

Peers

Jannis Meents
Nisha Vastani United Kingdom
Jannis Meents
Citations per year, relative to Jannis Meents Jannis Meents (= 1×) peers Nisha Vastani

Countries citing papers authored by Jannis Meents

Since Specialization
Citations

This map shows the geographic impact of Jannis Meents's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jannis Meents with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jannis Meents more than expected).

Fields of papers citing papers by Jannis Meents

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jannis Meents. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jannis Meents. The network helps show where Jannis Meents may publish in the future.

Co-authorship network of co-authors of Jannis Meents

This figure shows the co-authorship network connecting the top 25 collaborators of Jannis Meents. A scholar is included among the top collaborators of Jannis Meents based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jannis Meents. Jannis Meents is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Erickson, Andelain, Petra Hautvast, Sebastian Gießelmann, et al.. (2021). The difficulty to model Huntington’s disease in vitro using striatal medium spiny neurons differentiated from human induced pluripotent stem cells. Scientific Reports. 11(1). 6934–6934. 17 indexed citations
2.
Zhang, Jin, Jannis Meents, Andelain Erickson, et al.. (2021). The Potential Effect of Nav1.8 in Autism Spectrum Disorder: Evidence From a Congenital Case With Compound Heterozygous SCN10A Mutations. Frontiers in Molecular Neuroscience. 14. 709228–709228. 2 indexed citations
3.
Meents, Jannis, Maike F. Dohrn, Andrea Maier, et al.. (2021). Assessing the impact of pain-linked Nav1.7 variants: An example of two variants with no biophysical effect. Channels. 15(1). 208–228. 6 indexed citations
4.
Rodrigues, G. C., et al.. (2021). Towards a Wireless System that Can Monitor the Encapsulation of mm-sized Active Implants in vivo for Bioelectronic Medicine. Publikationsdatenbank der Fraunhofer-Gesellschaft (Fraunhofer-Gesellschaft). 981–984. 3 indexed citations
5.
Hausmann, Ralf, Silvia Detro‐Dassen, Petra Hautvast, et al.. (2020). Uncoupling sodium channel dimers restores the phenotype of a pain‐linked Na v 1.7 channel mutation. British Journal of Pharmacology. 177(19). 4481–4496. 22 indexed citations
6.
Hautvast, Petra, et al.. (2020). Phosphorylation of a chronic pain mutation in the voltage-gated sodium channel Nav1.7 increases voltage sensitivity. Journal of Biological Chemistry. 296(3). 100227–100227. 11 indexed citations
7.
Neacsu, Cristian, Andrias O. O’Reilly, Ralf Hausmann, et al.. (2020). Dataset of electrophysiological patch-clamp recordings of the effect of the compounds deltamethrin, ATx-II and β4-peptide on human cardiac Nav1.5 sodium channel gating properties. SHILAP Revista de lepidopterología. 31. 105844–105844. 2 indexed citations
8.
Neacsu, Cristian, Andrias O. O’Reilly, Ralf Hausmann, et al.. (2020). Mechanism underlying hooked resurgent-like tail currents induced by an insecticide in human cardiac Nav1.5. Toxicology and Applied Pharmacology. 397. 115010–115010. 4 indexed citations
9.
Urban, Nicole, Michael Schaefer, Achim Kless, et al.. (2019). Identification of aurintricarboxylic acid as a potent allosteric antagonist of P2X1 and P2X3 receptors. Neuropharmacology. 158. 107749–107749. 44 indexed citations
10.
Ciotu, Cosmin I., Christoforos Tsantoulas, Jannis Meents, et al.. (2019). Noncanonical Ion Channel Behaviour in Pain. International Journal of Molecular Sciences. 20(18). 4572–4572. 7 indexed citations
11.
Meents, Jannis, Martin Hampl, Petra Hautvast, et al.. (2018). Loss-of-function of Nav1.8/D1639N linked to human pain can be rescued by lidocaine. Pflügers Archiv - European Journal of Physiology. 470(12). 1787–1801. 12 indexed citations
12.
Schmidt, Diana, Esther Eberhardt, Inge Petter Kleggetveit, et al.. (2018). Pain relief in a neuropathy patient by lacosamide: Proof of principle of clinical translation from patient-specific iPS cell-derived nociceptors. EBioMedicine. 39. 401–408. 69 indexed citations
13.
Meents, Jannis, et al.. (2018). β1 subunit stabilises sodium channel Nav1.7 against mechanical stress. The Journal of Physiology. 596(12). 2433–2445. 10 indexed citations
14.
Meents, Jannis, Cosmin I. Ciotu, & Michael J. M. Fischer. (2018). TRPA1: a molecular view. Journal of Neurophysiology. 121(2). 427–443. 114 indexed citations
15.
Meents, Jannis, et al.. (2018). The opioid oxycodone use‐dependently inhibits the cardiac sodium channel NaV1.5. British Journal of Pharmacology. 175(14). 3007–3020. 15 indexed citations
16.
Meents, Jannis, Michael J. M. Fischer, & Peter A. McNaughton. (2017). Sensitization of TRPA1 by Protein Kinase A. PLoS ONE. 12(1). e0170097–e0170097. 42 indexed citations
17.
Meents, Jannis, Michael J. M. Fischer, & Peter A. McNaughton. (2016). Agonist‐induced sensitisation of the irritant receptor ion channel TRPA1. The Journal of Physiology. 594(22). 6643–6660. 30 indexed citations
18.
Meents, Jannis, Jan Hoffmann, Sandra R. Chaplan, et al.. (2015). Two TRPV1 receptor antagonists are effective in two different experimental models of migraine. The Journal of Headache and Pain. 16(1). 57–57. 33 indexed citations
19.
Meents, Jannis, Lars Neeb, & Uwe Reuter. (2010). TRPV1 in migraine pathophysiology. Trends in Molecular Medicine. 16(4). 153–159. 91 indexed citations
20.
Neeb, Lars, Jannis Meents, & Uwe Reuter. (2010). 5-HT1F Receptor Agonists: A New Treatment Option for Migraine Attacks?. Neurotherapeutics. 7(2). 176–182. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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