Jan Cendelín

1.0k total citations
56 papers, 748 citations indexed

About

Jan Cendelín is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Jan Cendelín has authored 56 papers receiving a total of 748 indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Cellular and Molecular Neuroscience, 29 papers in Molecular Biology and 20 papers in Neurology. Recurrent topics in Jan Cendelín's work include Neurogenesis and neuroplasticity mechanisms (19 papers), Genetic Neurodegenerative Diseases (16 papers) and Neuroscience and Neuropharmacology Research (15 papers). Jan Cendelín is often cited by papers focused on Neurogenesis and neuroplasticity mechanisms (19 papers), Genetic Neurodegenerative Diseases (16 papers) and Neuroscience and Neuropharmacology Research (15 papers). Jan Cendelín collaborates with scholars based in Czechia, Japan and France. Jan Cendelín's co-authors include F Vožeh, Jan Tůma, José M. Delgado‐García, Milena Králíčková, Pascal Hilber, Filip Tichánek, Zbyněk Tonar, Hiroshi Mitoma, Stéphane Besnard and Marie‐Laure Machado and has published in prestigious journals such as Journal of Neurophysiology, Scientific Reports and The FASEB Journal.

In The Last Decade

Jan Cendelín

53 papers receiving 743 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Jan Cendelín Czechia 15 429 325 257 168 116 56 748
Georgia Gunner United States 10 340 0.8× 256 0.8× 373 1.5× 203 1.2× 96 0.8× 14 1.0k
F Vožeh Czechia 13 303 0.7× 191 0.6× 172 0.7× 133 0.8× 59 0.5× 50 512
Barbara Di Benedetto Germany 19 386 0.9× 479 1.5× 251 1.0× 258 1.5× 69 0.6× 37 1.1k
Sinifunanya E. Nwaobi United States 8 442 1.0× 336 1.0× 286 1.1× 126 0.8× 112 1.0× 11 756
Luis Martínez‐Millán Spain 13 412 1.0× 262 0.8× 191 0.7× 191 1.1× 36 0.3× 36 703
Cristina Bertollini Italy 13 500 1.2× 263 0.8× 432 1.7× 105 0.6× 68 0.6× 15 1.2k
Daniel H. Mascó Argentina 18 350 0.8× 280 0.9× 85 0.3× 134 0.8× 86 0.7× 31 800
Martijn P. J. Dekkers Switzerland 11 362 0.8× 426 1.3× 115 0.4× 212 1.3× 43 0.4× 18 943
David González‐Forero Spain 16 431 1.0× 329 1.0× 105 0.4× 120 0.7× 103 0.9× 26 783
Stefanie Rauskolb Germany 8 672 1.6× 286 0.9× 101 0.4× 405 2.4× 73 0.6× 9 938

Countries citing papers authored by Jan Cendelín

Since Specialization
Citations

This map shows the geographic impact of Jan Cendelín's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jan Cendelín with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jan Cendelín more than expected).

Fields of papers citing papers by Jan Cendelín

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jan Cendelín. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jan Cendelín. The network helps show where Jan Cendelín may publish in the future.

Co-authorship network of co-authors of Jan Cendelín

This figure shows the co-authorship network connecting the top 25 collaborators of Jan Cendelín. A scholar is included among the top collaborators of Jan Cendelín based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jan Cendelín. Jan Cendelín is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Babuška, Václav, et al.. (2024). Quantification of Solid Embryonic Cerebellar Graft Volume in a Degenerative Ataxia Model. The Cerebellum. 23(5). 1811–1823.
2.
Tichánek, Filip, et al.. (2023). Experimental Treatment with Edaravone in a Mouse Model of Spinocerebellar Ataxia 1. International Journal of Molecular Sciences. 24(13). 10689–10689. 2 indexed citations
3.
Tichánek, Filip, et al.. (2020). Forced activity and environmental enrichment mildly improve manifestation of rapid cerebellar degeneration in mice. Behavioural Brain Research. 401. 113060–113060. 6 indexed citations
4.
Tichánek, Filip, et al.. (2020). Hippocampal mitochondrial dysfunction and psychiatric-relevant behavioral deficits in spinocerebellar ataxia 1 mouse model. Scientific Reports. 10(1). 5418–5418. 21 indexed citations
5.
Tichánek, Filip, et al.. (2020). Abnormalities in the cerebellar levels of trophic factors BDNF and GDNF in pcd and lurcher cerebellar mutant mice. Neuroscience Letters. 725. 134870–134870. 10 indexed citations
6.
Lorivel, Thomas, Jan Cendelín, & Pascal Hilber. (2020). Familiarization effects on the behavioral disinhibition of the cerebellar Lurcher mutant mice: use of the innovative Dual Maze. Behavioural Brain Research. 398. 112972–112972. 7 indexed citations
7.
Cendelín, Jan, et al.. (2018). Long-Term Development of Embryonic Cerebellar Grafts in Two Strains of Lurcher Mice. The Cerebellum. 17(4). 428–437. 7 indexed citations
8.
Hilber, Pascal, et al.. (2018). Cooperation of the vestibular and cerebellar networks in anxiety disorders and depression. Progress in Neuro-Psychopharmacology and Biological Psychiatry. 89. 310–321. 54 indexed citations
9.
Cendelín, Jan, Hiroshi Mitoma, & Mario Manto. (2017). Neurotransplantation Therapy and Cerebellar Reserve. CNS & Neurological Disorders - Drug Targets. 17(3). 172–183. 11 indexed citations
10.
Cendelín, Jan. (2016). Experimental neurotransplantation treatment for hereditary cerebellar ataxias. PubMed. 3(1). 7–7. 5 indexed citations
11.
Cendelín, Jan, et al.. (2016). Smaller Absolute Quantities but Greater Relative Densities of Microvessels Are Associated with Cerebellar Degeneration in Lurcher Mice. Frontiers in Neuroanatomy. 10. 35–35. 14 indexed citations
12.
Tůma, Jan, et al.. (2015). Mutation-related differences in exploratory, spatial, and depressive-like behavior in pcd and Lurcher cerebellar mutant mice. Frontiers in Behavioral Neuroscience. 9. 116–116. 25 indexed citations
13.
Cendelín, Jan. (2015). Transplantation and Stem Cell Therapy for Cerebellar Degenerations. The Cerebellum. 15(1). 48–50. 13 indexed citations
14.
Cendelín, Jan, et al.. (2013). Effect of dimethyl sulfoxide in cerebellar mutant Lurcher mice. Neuroscience Letters. 543. 142–145. 10 indexed citations
15.
Cendelín, Jan, et al.. (2012). Long-term survival of solid embryonic cerebellar grafts in Lurcher mice. Neuroscience Letters. 515(1). 23–27. 12 indexed citations
16.
Babuška, Václav, Vlastimil Kulda, Martin Pešta, et al.. (2010). Characterization of P19 cells during retinoic acid induced differentiation.. PubMed. 111(4). 289–99. 7 indexed citations
17.
Cendelín, Jan, et al.. (2010). Changes of motor abilities during ontogenetic development in Lurcher mutant mice. Neuroscience. 168(3). 646–651. 5 indexed citations
18.
Cendelín, Jan, et al.. (2009). A Preliminary Study of Solid Embryonic Cerebellar Graft Survival in Adult B6CBA Lurcher Mutant and Wild Type Mice. The Anatomical Record. 292(12). 1986–1992. 12 indexed citations
19.
20.
Cendelín, Jan, et al.. (2005). Purkinje cell loss affects differentially the execution, acquisition and prepulse inhibition of skeletal and facial motor responses in Lurcher mice. European Journal of Neuroscience. 21(4). 979–988. 73 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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