Jamie H. Rose

1.2k total citations
14 papers, 852 citations indexed

About

Jamie H. Rose is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Behavioral Neuroscience. According to data from OpenAlex, Jamie H. Rose has authored 14 papers receiving a total of 852 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Cellular and Molecular Neuroscience, 10 papers in Molecular Biology and 2 papers in Behavioral Neuroscience. Recurrent topics in Jamie H. Rose's work include Neurotransmitter Receptor Influence on Behavior (10 papers), Receptor Mechanisms and Signaling (10 papers) and Neuroscience and Neuropharmacology Research (7 papers). Jamie H. Rose is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (10 papers), Receptor Mechanisms and Signaling (10 papers) and Neuroscience and Neuropharmacology Research (7 papers). Jamie H. Rose collaborates with scholars based in United States. Jamie H. Rose's co-authors include Sara R. Jones, Anushree N. Karkhanis, Rong Chen, Jason L. Locke, J. Konstantopoulos, Mark J. Ferris, Jeffrey L. Weiner, Rodrigo A. España, Brian A. McCool and Marcelo F. Lopez and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and The FASEB Journal.

In The Last Decade

Jamie H. Rose

14 papers receiving 849 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jamie H. Rose United States	11	629	379	196	151	126	14	852
Anna Molander Sweden	17	991 1.6×	605 1.6×	197 1.0×	187 1.2×	102 0.8×	21	1.4k
Michel M. M. Verheij Netherlands	18	555 0.9×	338 0.9×	199 1.0×	93 0.6×	94 0.7×	48	922
Linda M. Rorick‐Kehn United States	14	636 1.0×	421 1.1×	187 1.0×	131 0.9×	98 0.8×	14	866
Esi Domi Italy	14	462 0.7×	278 0.7×	152 0.8×	116 0.8×	127 1.0×	34	773
Ilga Misane Sweden	17	622 1.0×	394 1.0×	286 1.5×	80 0.5×	145 1.2×	21	945
Sonja J. Stutz United States	20	720 1.1×	442 1.2×	109 0.6×	116 0.8×	72 0.6×	29	1.1k
Thierry Jolas France	18	849 1.3×	488 1.3×	222 1.1×	240 1.6×	95 0.8×	24	1.2k
Chiara Ruzza Italy	19	921 1.5×	546 1.4×	365 1.9×	257 1.7×	126 1.0×	61	1.1k
Sami Ben Hamida United States	23	932 1.5×	582 1.5×	269 1.4×	127 0.8×	56 0.4×	43	1.3k
Kerstin A. Ford United States	15	782 1.2×	429 1.1×	223 1.1×	116 0.8×	63 0.5×	15	1.0k

Countries citing papers authored by Jamie H. Rose

Since Specialization

Citations

This map shows the geographic impact of Jamie H. Rose's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jamie H. Rose with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jamie H. Rose more than expected).

Fields of papers citing papers by Jamie H. Rose

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jamie H. Rose. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jamie H. Rose. The network helps show where Jamie H. Rose may publish in the future.

Co-authorship network of co-authors of Jamie H. Rose

This figure shows the co-authorship network connecting the top 25 collaborators of Jamie H. Rose. A scholar is included among the top collaborators of Jamie H. Rose based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jamie H. Rose. Jamie H. Rose is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Holleran, Katherine M., et al.. (2020). Organic cation transporter 3 and the dopamine transporter differentially regulate catecholamine uptake in the basolateral amygdala and nucleus accumbens. European Journal of Neuroscience. 52(11). 4546–4562. 17 indexed citations

Kash, Thomas L., Antoniette M. Maldonado‐Devincci, Kristen E. Pleil, et al.. (2020). Effects of chronic ethanol exposure on neuronal function in the prefrontal cortex and extended amygdala. UNC Libraries. 2 indexed citations

Brust, Tarsis F., Jenny Morgenweck, Susy A. Kim, et al.. (2017). A G Protein‐Biased Ligand of the Kappa Opioid Receptor is Antinociceptive and Antipruritic But Does Not Cause Sedation or Dysphoria. The FASEB Journal. 31(S1). 2 indexed citations

Karkhanis, Anushree N., Jamie H. Rose, Jeffrey L. Weiner, & Sara R. Jones. (2016). Early-Life Social Isolation Stress Increases Kappa Opioid Receptor Responsiveness and Downregulates the Dopamine System. Neuropsychopharmacology. 41(9). 2263–2274. 96 indexed citations

Brust, Tarsis F., Jenny Morgenweck, Susy A. Kim, et al.. (2016). Biased agonists of the kappa opioid receptor suppress pain and itch without causing sedation or dysphoria. Science Signaling. 9(456). ra117–ra117. 177 indexed citations

Karkhanis, Anushree N., et al.. (2016). Switch from excitatory to inhibitory actions of ethanol on dopamine levels after chronic exposure: Role of kappa opioid receptors. Neuropharmacology. 110(Pt A). 190–197. 54 indexed citations

Martin, Thomas J., et al.. (2016). (361) A novel G-protein biased kappa opioid that displays analgesic but not dysphoric actions in the rat. Journal of Pain. 17(4). S65–S65. 1 indexed citations

Pleil, Kristen E., Emily G. Lowery‐Gionta, Nicole A. Crowley, et al.. (2015). Effects of chronic ethanol exposure on neuronal function in the prefrontal cortex and extended amygdala. Neuropharmacology. 99. 735–749. 125 indexed citations

Rose, Jamie H., Anushree N. Karkhanis, Rong Chen, et al.. (2015). Supersensitive Kappa Opioid Receptors Promotes Ethanol Withdrawal-Related Behaviors and Reduce Dopamine Signaling in the Nucleus Accumbens. The International Journal of Neuropsychopharmacology. 19(5). pyv127–pyv127. 110 indexed citations

10.

Ferris, Mark J., Erin S. Calipari, Jamie H. Rose, et al.. (2015). A Single Amphetamine Infusion Reverses Deficits in Dopamine Nerve-Terminal Function Caused by a History of Cocaine Self-Administration. Neuropsychopharmacology. 40(8). 1826–1836. 20 indexed citations

11.

Karkhanis, Anushree N., et al.. (2015). Chronic intermittent ethanol exposure reduces presynaptic dopamine neurotransmission in the mouse nucleus accumbens. Drug and Alcohol Dependence. 150. 24–30. 67 indexed citations

12.

Yorgason, Jordan T., Jamie H. Rose, J. Michael McIntosh, Mark J. Ferris, & Sara R. Jones. (2014). Greater ethanol inhibition of presynaptic dopamine release in C57BL/6J than DBA/2J mice: Role of nicotinic acetylcholine receptors. Neuroscience. 284. 854–864. 18 indexed citations

13.

Ferris, Mark J., Rodrigo A. España, Jason L. Locke, et al.. (2014). Dopamine transporters govern diurnal variation in extracellular dopamine tone. Proceedings of the National Academy of Sciences. 111(26). E2751–9. 139 indexed citations

14.

Rose, Jamie H., Erin S. Calipari, Tiffany A. Mathews, & Sara R. Jones. (2013). Greater Ethanol-Induced Locomotor Activation in DBA/2J versus C57BL/6J Mice Is Not Predicted by Presynaptic Striatal Dopamine Dynamics. PLoS ONE. 8(12). e83852–e83852. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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