James S. Bean

635 total citations
16 papers, 414 citations indexed

About

James S. Bean is a scholar working on Molecular Biology, Cancer Research and Surgery. According to data from OpenAlex, James S. Bean has authored 16 papers receiving a total of 414 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Cancer Research and 3 papers in Surgery. Recurrent topics in James S. Bean's work include Cancer, Lipids, and Metabolism (3 papers), Cerebrovascular and Carotid Artery Diseases (3 papers) and Estrogen and related hormone effects (3 papers). James S. Bean is often cited by papers focused on Cancer, Lipids, and Metabolism (3 papers), Cerebrovascular and Carotid Artery Diseases (3 papers) and Estrogen and related hormone effects (3 papers). James S. Bean collaborates with scholars based in United States, Brazil and Egypt. James S. Bean's co-authors include Raymond F. Kauffman, Karen M. Zimmerman, Raymond F. Brown, Mitchell I. Steinberg, William R. Bensch, Mark D. Rekhter, George J. Cullinan, Xiao Lu, Ghassan S. Kassab and Joseph V. Haas and has published in prestigious journals such as PLoS ONE, The Journal of Clinical Endocrinology & Metabolism and Journal of Medicinal Chemistry.

In The Last Decade

James S. Bean

16 papers receiving 395 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
James S. Bean United States 11 161 111 94 75 74 16 414
Yoji Shibayama United States 13 183 1.1× 86 0.8× 51 0.5× 46 0.6× 36 0.5× 23 818
Jens‐Kerim Ergüden Germany 6 147 0.9× 170 1.5× 65 0.7× 29 0.4× 54 0.7× 8 423
Tadeu L. Montagnoli Brazil 12 127 0.8× 56 0.5× 110 1.2× 40 0.5× 44 0.6× 21 396
Nathalie Delesque-Touchard France 7 197 1.2× 65 0.6× 166 1.8× 28 0.4× 115 1.6× 7 502
Karen M. Zimmerman United States 8 108 0.7× 104 0.9× 109 1.2× 15 0.2× 109 1.5× 11 368
B Sepehrnia United States 11 212 1.3× 195 1.8× 62 0.7× 134 1.8× 153 2.1× 14 552
Shoji Kume Japan 14 185 1.1× 29 0.3× 92 1.0× 75 1.0× 82 1.1× 25 571
Maria Escobar United States 8 184 1.1× 34 0.3× 32 0.3× 71 0.9× 29 0.4× 10 399
H.A.P. Pols Netherlands 11 199 1.2× 207 1.9× 68 0.7× 121 1.6× 17 0.2× 18 465
James A. Koehn United States 14 262 1.6× 62 0.6× 155 1.6× 30 0.4× 40 0.5× 24 668

Countries citing papers authored by James S. Bean

Since Specialization
Citations

This map shows the geographic impact of James S. Bean's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James S. Bean with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James S. Bean more than expected).

Fields of papers citing papers by James S. Bean

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James S. Bean. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James S. Bean. The network helps show where James S. Bean may publish in the future.

Co-authorship network of co-authors of James S. Bean

This figure shows the co-authorship network connecting the top 25 collaborators of James S. Bean. A scholar is included among the top collaborators of James S. Bean based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James S. Bean. James S. Bean is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Dey, Asim, James S. Bean, Francis S. Willard, et al.. (2020). Selective Phosphodiesterase 1 Inhibitor BTTQ Reduces Blood Pressure in Spontaneously Hypertensive and Dahl Salt Sensitive Rats: Role of Peripheral Vasodilation. Frontiers in Physiology. 11. 543727–543727. 6 indexed citations
2.
Luz, J.G., Matthew W. Carson, Bradley Condon, et al.. (2015). Indole Glucocorticoid Receptor Antagonists Active in a Model of Dyslipidemia Act via a Unique Association with an Agonist Binding Site.. Journal of Medicinal Chemistry. 58(16). 6607–6618. 33 indexed citations
3.
4.
Conway, Richard G., Eyassu Chernet, Robert J. Benschop, et al.. (2012). Glucose Metabolic Trapping in Mouse Arteries: Nonradioactive Assay of Atherosclerotic Plaque Inflammation Applicable to Drug Discovery. PLoS ONE. 7(11). e50349–e50349. 7 indexed citations
5.
Lu, Xiao, James S. Bean, Ghassan S. Kassab, & Mark D. Rekhter. (2011). Protein Kinase C inhibition ameliorates functional endothelial insulin resistance and Vascular Smooth Muscle Cell hypersensitivity to insulin in diabetic hypertensive rats. Cardiovascular Diabetology. 10(1). 48–48. 22 indexed citations
6.
Wang, Jian, Yu Lan, He Wang, et al.. (2007). Identification and Characterization of Hamster Stearoyl‐CoA Desaturase Isoforms. Lipids. 43(3). 197–205. 4 indexed citations
7.
8.
Xu, Yanping, Xiaodong Wang, R. C. Thompson, et al.. (2003). Design and Synthesis of a Potent and Selective Triazolone-Based Peroxisome Proliferator-Activated Receptor α Agonist. Journal of Medicinal Chemistry. 46(24). 5121–5124. 52 indexed citations
9.
Kauffman, Raymond F., et al.. (2000). Raloxifene and Estrogen Inhibit Neointimal Thickening After Balloon Injury in the Carotid Artery of Male and Ovariectomized Female Rats. Journal of Cardiovascular Pharmacology. 36(4). 459–465. 18 indexed citations
10.
Bensch, William R., Robert A. Gadski, James S. Bean, et al.. (1999). Effects of LY295427, a Low-Density Lipoprotein (LDL) Receptor Up-Regulator, on LDL Receptor Gene Transcription and Cholesterol Metabolism in Normal and Hypercholesterolemic Hamsters. Journal of Pharmacology and Experimental Therapeutics. 289(1). 85–92. 14 indexed citations
11.
Kauffman, Raymond F., William R. Bensch, Roger E. Roudebush, et al.. (1997). Hypocholesterolemic Activity of Raloxifene (LY139481): Pharmacological Characterization as a Selective Estrogen Receptor Modulator. Journal of Pharmacology and Experimental Therapeutics. 280(1). 146–153. 48 indexed citations
12.
Bean, James S., C. P. Dell, Steven Williams, et al.. (1996). Inhibition of vascular smooth muscle cell proliferation and arterial intimal thickening by a novel antiproliferative naphthopyran.. Journal of Pharmacology and Experimental Therapeutics. 278(3). 1452–1459. 21 indexed citations
13.
Kauffman, Raymond F., et al.. (1993). Vascular serotonin 5-hydroxytryptamine2 receptor blockade by LY215840: lack of effect upon neointima formation in balloon-injured rat carotid arteries.. Journal of Pharmacology and Experimental Therapeutics. 266(1). 450–455. 1 indexed citations
14.
Kauffman, Raymond F., James S. Bean, Karen M. Zimmerman, Raymond F. Brown, & Mitchell I. Steinberg. (1991). Losartan, a nonpeptide angiotensin II (Ang II) receptor antagonist, inhibits neointima formation following balloon injury to rat carotid arteries. Life Sciences. 49(25). PL223–PL228. 104 indexed citations
15.
Bendele, A.M., James S. Bean, & James F. Hulman. (1991). Passive Role of Articular Chondrocytes in the Pathogenesis of Acute Meniscectomy-induced Cartilage Degeneration. Veterinary Pathology. 28(3). 207–215. 14 indexed citations
16.
Keele, Doman K., et al.. (1962). Immunologic Reactions to Human Chorionic Gonadotropin. The Journal of Clinical Endocrinology & Metabolism. 22(3). 287–299. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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