James Pilling
Impact in
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- Pluripotent Stem Cells Research
- Wnt/β-catenin signaling in development and cancer
- Bone Metabolism and Diseases
Papers in
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- Microtubule and mitosis dynamics 2
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- CRISPR and Genetic Engineering 3
- Wnt/β-catenin signaling in development and cancer 3
- Protein Degradation and Inhibitors 2
- Co-authors
- C ArcherAmy PointonThierry DorvalYinhai WangChristopher E. PollardPatrick OʼSheaMichael L. SullivanEdward Ainscow
- Journals
- Molecular Cancer Therapeutics (3 papers)SLAS DISCOVERY (2 papers)Clinical Cancer Research (1 paper)Toxicological Sciences (1 paper)Expert Opinion on Drug Safety (1 paper)
- Partner nations
- United KingdomSingaporeUnited States
In The Last Decade
James Pilling
14 papers receiving 348 citations
Peers
Comparison fields: 5 of 68
- Molecular Biology 215
- Biomaterials 37
- Cell Biology 40
- Oncology 60
- Biophysics 12
Countries citing papers authored by James Pilling
This map shows the geographic impact of James Pilling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James Pilling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James Pilling more than expected).
Fields of papers citing papers by James Pilling
This network shows the impact of papers produced by James Pilling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James Pilling. The network helps show where James Pilling may publish in the future.
Co-authorship network
The 25 scholars most cited alongside James Pilling, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2024 | 0 | |
| 2 | 2021 | 1 | |
| 3 | 2021 | 1 | |
| 4 | 2020 | 1 | |
| 5 | 2019 | 16 | |
| 6 | 2018 | 99 | |
| 7 | 2017 | 29 | |
| 8 | 2017 | 24 | |
| 9 | 2016 | 56 | |
| 10 | 2016 | 18 | |
| 11 | 2013 | 19 | |
| 12 | 2010 | 8 | |
| 13 | 2010 | 11 | |
| 14 | 2010 | 56 | |
| 15 | 2008 | 16 |
About James Pilling
James Pilling is a scholar working on Cell Biology, Molecular Biology, Oncology, Biochemistry and Cancer Research, having authored 15 papers that have together received 355 indexed citations. Recurring topics across this work include CRISPR and Genetic Engineering (3 papers), Wnt/β-catenin signaling in development and cancer (3 papers), Cancer, Hypoxia, and Metabolism (2 papers), Microtubule and mitosis dynamics (2 papers), Protein Degradation and Inhibitors (2 papers), Neuroscience and Neural Engineering (2 papers), Biotechnology and Related Fields (2 papers) and Cardiac electrophysiology and arrhythmias (2 papers). The work is most often cited by research in Molecular Biology (215 citations), Biomaterials (37 citations), Cell Biology (40 citations), Oncology (60 citations) and Biophysics (12 citations). James Pilling has collaborated with scholars based in United Kingdom, Singapore and United States. Frequent co-authors include C Archer, Amy Pointon, Thierry Dorval, Yinhai Wang, Christopher E. Pollard, Patrick OʼShea, Michael L. Sullivan, Edward Ainscow, Ilaria Bellantuono and Lucksy Kottam. Their work appears in journals such as Molecular Cancer Therapeutics, SLAS DISCOVERY, Clinical Cancer Research, Toxicological Sciences and Expert Opinion on Drug Safety.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.