James G. Lewis

2.1k total citations
33 papers, 1.7k citations indexed

About

James G. Lewis is a scholar working on Molecular Biology, Cancer Research and Behavioral Neuroscience. According to data from OpenAlex, James G. Lewis has authored 33 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 7 papers in Cancer Research and 5 papers in Behavioral Neuroscience. Recurrent topics in James G. Lewis's work include Stress Responses and Cortisol (5 papers), Carcinogens and Genotoxicity Assessment (5 papers) and Antioxidant Activity and Oxidative Stress (4 papers). James G. Lewis is often cited by papers focused on Stress Responses and Cortisol (5 papers), Carcinogens and Genotoxicity Assessment (5 papers) and Antioxidant Activity and Oxidative Stress (4 papers). James G. Lewis collaborates with scholars based in United States. James G. Lewis's co-authors include Dolph O. Adams, Edward C. Suarez, Ranga Krishnan, James A. Swenberg, Cynthia M. Kuhn, Kenneth H. Young, William E. Stewart, Salvatore V. Pizzo, Kishore M. Gadde and Michael J. Helms and has published in prestigious journals such as Nature, Environmental Health Perspectives and The American Journal of Medicine.

In The Last Decade

James G. Lewis

32 papers receiving 1.7k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
James G. Lewis United States 22 485 329 316 271 200 33 1.7k
Amàlia Lafuente Spain 28 910 1.9× 101 0.3× 167 0.5× 290 1.1× 96 0.5× 111 2.3k
Μάριος Μαρσέλος Greece 30 803 1.7× 199 0.6× 316 1.0× 135 0.5× 34 0.2× 120 2.8k
Juan Peng China 27 536 1.1× 116 0.4× 248 0.8× 135 0.5× 147 0.7× 61 2.0k
Piotr Czarny Poland 25 686 1.4× 200 0.6× 148 0.5× 381 1.4× 58 0.3× 64 1.6k
Robert Kinobe Australia 22 593 1.2× 249 0.8× 44 0.1× 139 0.5× 102 0.5× 55 2.3k
Lin He China 24 441 0.9× 65 0.2× 103 0.3× 113 0.4× 94 0.5× 67 1.5k
Mario Palermo Italy 26 369 0.8× 183 0.6× 60 0.2× 144 0.5× 86 0.4× 59 2.1k
Michel F. Rossier Switzerland 39 2.2k 4.5× 233 0.7× 153 0.5× 98 0.4× 604 3.0× 100 3.9k
Xiaofeng Jiang China 25 933 1.9× 113 0.3× 393 1.2× 233 0.9× 57 0.3× 91 2.0k
Dongmei Chen China 20 801 1.7× 59 0.2× 188 0.6× 73 0.3× 294 1.5× 55 1.7k

Countries citing papers authored by James G. Lewis

Since Specialization
Citations

This map shows the geographic impact of James G. Lewis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James G. Lewis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James G. Lewis more than expected).

Fields of papers citing papers by James G. Lewis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James G. Lewis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James G. Lewis. The network helps show where James G. Lewis may publish in the future.

Co-authorship network of co-authors of James G. Lewis

This figure shows the co-authorship network connecting the top 25 collaborators of James G. Lewis. A scholar is included among the top collaborators of James G. Lewis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James G. Lewis. James G. Lewis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Suarez, Edward C., Stephen H. Boyle, James G. Lewis, Russell P. Hall, & Kenneth H. Young. (2005). Increases in stimulated secretion of proinflammatory cytokines by blood monocytes following arousal of negative affect: The role of insulin resistance as moderator. Brain Behavior and Immunity. 20(4). 331–338. 39 indexed citations
3.
Boukhalfa, Hakim, James G. Lewis, & Alvin L. Crumbliss. (2004). Beryllium(II) binding to ATP and ADP: Potentiometric determination of the thermodynamic constants and implications for in vivo toxicity. BioMetals. 17(2). 105–109. 11 indexed citations
4.
Suarez, Edward C., James G. Lewis, Ranga Krishnan, & Kenneth H. Young. (2004). Enhanced expression of cytokines and chemokines by blood monocytes to in vitro lipopolysaccharide stimulation are associated with hostility and severity of depressive symptoms in healthy women. Psychoneuroendocrinology. 29(9). 1119–1128. 149 indexed citations
5.
Suarez, Edward C., Ranga Krishnan, & James G. Lewis. (2003). The Relation of Severity of Depressive Symptoms to Monocyte-Associated Proinflammatory Cytokines and Chemokines in Apparently Healthy Men. Psychosomatic Medicine. 65(3). 362–368. 166 indexed citations
6.
Suarez, Edward C., James G. Lewis, & Cynthia M. Kuhn. (2002). The relation of aggression, hostility, and anger to lipopolysaccharide-stimulated tumor necrosis factor (TNF)-α by blood monocytes from normal men. Brain Behavior and Immunity. 16(6). 675–684. 109 indexed citations
7.
Williams, Redford B., Douglas A. Marchuk, Kishore M. Gadde, et al.. (2002). Serotonin-Related Gene Polymorphisms and Central Nervous System Serotonin Function. Neuropsychopharmacology. 28(3). 533–541. 216 indexed citations
8.
Williams, Redford B., Douglas A. Marchuk, Kishore M. Gadde, et al.. (2001). Central Nervous System Serotonin Function and Cardiovascular Responses to Stress. Psychosomatic Medicine. 63(2). 300–305. 115 indexed citations
9.
Achanzar, William E., et al.. (2000). Cadmium Induces c-myc, p53, and c-jun Expression in Normal Human Prostate Epithelial Cells as a Prelude to Apoptosis. Toxicology and Applied Pharmacology. 164(3). 291–300. 92 indexed citations
10.
Gawdi, Govind, et al.. (1998). Alterations in Calcium Metabolism in Murine Macrophages by the Benzene Metabolite 1,4-Benzoquinone. Toxicology and Applied Pharmacology. 151(1). 1–8. 3 indexed citations
11.
Lewis, James G., et al.. (1995). Selective sensitivity of macrophages to cytotoxicity by inhibitors of macromolecular synthesis: induction of apoptosis. Journal of Leukocyte Biology. 57(4). 635–642. 14 indexed citations
12.
Adams, Dolph O., et al.. (1994). Effects of Benzene Metabolites on Receptor-Mediated Phagocytosis and Cytoskeletal Integrity in Mouse Peritoneal Macrophages. Toxicology and Applied Pharmacology. 126(2). 214–223. 12 indexed citations
13.
Lewis, James G., et al.. (1988). Toxic effects of benzene and benzene metabolites on mononuclear phagocytes. Toxicology and Applied Pharmacology. 92(2). 246–254. 34 indexed citations
14.
Lewis, James G. & Dolph O. Adams. (1988). Divalent cation requirements for mounting a respiratory burst in response to phorbol diesters by macrophages from SENCAR and C57BL/6 mice. Chemico-Biological Interactions. 66(1-2). 1–11. 3 indexed citations
15.
Lewis, James G. & Dolph O. Adams. (1987). Inflammation, oxidative DNA damage, and carcinogenesis.. Environmental Health Perspectives. 76. 19–27. 65 indexed citations
16.
Lewis, James G. & Dolph O. Adams. (1987). Early inflammatory changes in the skin of SENCAR and C57BL/6 mice following exposme to 12-O-tetradecanoylphorbol-13-acetat. Carcinogenesis. 8(7). 889–898. 49 indexed citations
17.
18.
Lewis, James G. & Dolph O. Adams. (1985). Induction of 5,6-ring-saturated thymine bases in NIH-3T3 cells by phorbol ester-stimulated macrophages: role of reactive oxygen intermediates.. PubMed. 45(3). 1270–5. 71 indexed citations
19.
Stead, Nancy W., Kenneth A. Bauer, Thomas R. Kinney, et al.. (1983). Venous thrombosis in a family with defective release of vascular plasminogen activator and elevated plasma factor VIII/von Willebrand's factor. The American Journal of Medicine. 74(1). 33–39. 77 indexed citations
20.
Swenberg, James A., et al.. (1983). Cell Specificity in DNA Damage and Repair. PubMed. 24. 605–617. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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