James E. Koch

701 total citations
19 papers, 558 citations indexed

About

James E. Koch is a scholar working on Cellular and Molecular Neuroscience, Endocrine and Autonomic Systems and Molecular Biology. According to data from OpenAlex, James E. Koch has authored 19 papers receiving a total of 558 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Cellular and Molecular Neuroscience, 6 papers in Endocrine and Autonomic Systems and 5 papers in Molecular Biology. Recurrent topics in James E. Koch's work include Neurotransmitter Receptor Influence on Behavior (10 papers), Neuropeptides and Animal Physiology (8 papers) and Regulation of Appetite and Obesity (6 papers). James E. Koch is often cited by papers focused on Neurotransmitter Receptor Influence on Behavior (10 papers), Neuropeptides and Animal Physiology (8 papers) and Regulation of Appetite and Obesity (6 papers). James E. Koch collaborates with scholars based in United States, United Kingdom and France. James E. Koch's co-authors include Richard J. Bodnar, Richard J. Bodnar, Michael J. Glass, Sarah F. Leibowitz, Madeline L. Cooper, Mary Osbakken, Mark I. Friedman, Hong Ji, Barry Hahn and Gavril W. Pasternak and has published in prestigious journals such as Brain Research, Neuroscience and American Journal of Physiology-Regulatory, Integrative and Comparative Physiology.

In The Last Decade

James E. Koch

19 papers receiving 548 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
James E. Koch United States 15 358 217 186 155 106 19 558
Robert A. Del Vecchio United States 10 227 0.6× 120 0.6× 86 0.5× 183 1.2× 169 1.6× 14 522
Becky T. Davies United States 12 225 0.6× 170 0.8× 97 0.5× 91 0.6× 81 0.8× 19 455
Annie Morien United States 10 177 0.5× 165 0.8× 70 0.4× 70 0.5× 69 0.7× 13 394
P.J. Wellman United States 13 194 0.5× 192 0.9× 100 0.5× 84 0.5× 144 1.4× 32 466
Magdaléna Šustková-Fišerová Czechia 13 175 0.5× 188 0.9× 57 0.3× 101 0.7× 148 1.4× 21 450
Rainer O. Heurich United States 5 237 0.7× 397 1.8× 40 0.2× 260 1.7× 103 1.0× 6 635
Victoria Mendizábal Argentina 10 399 1.1× 60 0.3× 322 1.7× 208 1.3× 112 1.1× 17 631
Roger P. Dilts United States 14 534 1.5× 58 0.3× 56 0.3× 355 2.3× 138 1.3× 17 780
A. Aracil-Fernández Spain 12 492 1.4× 89 0.4× 528 2.8× 84 0.5× 107 1.0× 17 717

Countries citing papers authored by James E. Koch

Since Specialization
Citations

This map shows the geographic impact of James E. Koch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James E. Koch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James E. Koch more than expected).

Fields of papers citing papers by James E. Koch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James E. Koch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James E. Koch. The network helps show where James E. Koch may publish in the future.

Co-authorship network of co-authors of James E. Koch

This figure shows the co-authorship network connecting the top 25 collaborators of James E. Koch. A scholar is included among the top collaborators of James E. Koch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James E. Koch. James E. Koch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Lam, Suzanne, Anne‐Sophie Hérard, James E. Koch, et al.. (2019). Encephalopathy induced by Alzheimer brain inoculation in a non-human primate. Acta Neuropathologica Communications. 7(1). 126–126. 34 indexed citations
2.
Koch, James E., et al.. (2003). Effects of the cannabinoid antagonists AM281 and AM630 on deprivation-induced intake in Lewis rats. Brain Research. 967(1-2). 290–292. 49 indexed citations
3.
Friedman, Mark I., et al.. (2002). High-fat diet prevents eating response and attenuates liver ATP decline in rats given 2,5-anhydro-d-mannitol. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 282(3). R710–R714. 11 indexed citations
4.
Koch, James E., et al.. (2001). Δ9-Tetrahydrocannabinol Stimulates Palatable Food Intake in Lewis Rats: Effects of Peripheral and Central Administration. Nutritional Neuroscience. 4(3). 179–187. 44 indexed citations
5.
Koch, James E.. (2001). Δ9-THC stimulates food intake in Lewis rats: effects on chow, high-fat and sweet high-fat diets. Pharmacology Biochemistry and Behavior. 68(3). 539–543. 106 indexed citations
6.
Koch, James E., Hong Ji, Mary Osbakken, & Mark I. Friedman. (1998). Temporal relationships between eating behavior and liver adenine nucleotides in rats treated with 2,5-AM. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 274(3). R610–R617. 25 indexed citations
7.
Bodnar, Richard J., Michael J. Glass, & James E. Koch. (1995). Analysis of central opioid receptor subtype antagonism of hypotonic and hypertonic saline intake in water-deprived rats. Brain Research Bulletin. 36(3). 293–300. 14 indexed citations
8.
Koch, James E., Michael J. Glass, Madeline L. Cooper, & Richard J. Bodnar. (1995). Alterations in deprivation, glucoprivic and sucrose intake following general, mu and kappa opioid antagonists in the hypothalamic paraventricular nucleus of rats. Neuroscience. 66(4). 951–957. 45 indexed citations
9.
Koch, James E., et al.. (1994). Naltrexone, dopamine receptor agonists and antagonists, and food intake in rats: 2. 2-deoxy-d-glucose. Pharmacology Biochemistry and Behavior. 49(1). 205–211. 14 indexed citations
10.
Dougherty, Thomas J., et al.. (1994). Naltrexone, serotonin receptor subtype antagonists, and carbohydrate intake in rats. Pharmacology Biochemistry and Behavior. 48(1). 193–201. 9 indexed citations
11.
Koch, James E., et al.. (1994). Naltrexone, dopamine receptor agonists and antagonists, and food intake in rats: 1. Food deprivation. Pharmacology Biochemistry and Behavior. 49(1). 197–204. 17 indexed citations
12.
Hahn, Barry, et al.. (1994). Differential modulation of angiotensin II and hypertonic saline-induced drinking by opioid receptor subtype antagonists in rats. Brain Research. 635(1-2). 203–210. 18 indexed citations
13.
Koch, James E. & Richard J. Bodnar. (1994). Selective alterations in macronutrient intake of food-deprived or glucoprivic rats by centrally-administered opioid receptor subtype antagonists in rats. Brain Research. 657(1-2). 191–201. 46 indexed citations
14.
Koch, James E., et al.. (1993). Central opioid receptor subtype antagonists differentially reduce intake of saccharin and maltose dextrin solutions in rats. Brain Research. 618(2). 261–270. 65 indexed citations
15.
Koch, James E. & Richard J. Bodnar. (1993). Involvement of mu1 and mu2 opioid receptor subtypes in tail-pinch feeding in rats. Physiology & Behavior. 53(3). 603–605. 18 indexed citations
16.
Koch, James E., et al.. (1992). Naltrexone, serotonin receptor subtype antagonists, and glucoprivic intake: 2. Insulin. Pharmacology Biochemistry and Behavior. 42(4). 671–680. 6 indexed citations
17.
Koch, James E., et al.. (1992). Naloxone benzoylhydrazone, a κ3 opioid agonist, stimulates food intake in rats. Brain Research. 581(2). 311–314. 19 indexed citations
18.
Koch, James E., Lindsay B. Hough, & Richard J. Bodnar. (1992). Potentiation of 2-deoxy-D-glucose antinociception, but not hyperphagia by zolantidine, a Histamine (H2) receptor antagonist. Pharmacology Biochemistry and Behavior. 41(2). 371–376. 3 indexed citations
19.
Koch, James E., et al.. (1992). Naltrexone, serotonin receptor subtype antagonists, and glucoprivic intake: 1.2-deoxy-D-glucose. Pharmacology Biochemistry and Behavior. 42(4). 661–670. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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