James E. Hopper

5.4k total citations
102 papers, 4.3k citations indexed

About

James E. Hopper is a scholar working on Molecular Biology, Nephrology and Biomedical Engineering. According to data from OpenAlex, James E. Hopper has authored 102 papers receiving a total of 4.3k indexed citations (citations by other indexed papers that have themselves been cited), including 67 papers in Molecular Biology, 21 papers in Nephrology and 21 papers in Biomedical Engineering. Recurrent topics in James E. Hopper's work include Fungal and yeast genetics research (42 papers), Biofuel production and bioconversion (21 papers) and Renal Diseases and Glomerulopathies (19 papers). James E. Hopper is often cited by papers focused on Fungal and yeast genetics research (42 papers), Biofuel production and bioconversion (21 papers) and Renal Diseases and Glomerulopathies (19 papers). James E. Hopper collaborates with scholars based in United States, India and Cyprus. James E. Hopper's co-authors include S. A. Johnston, Claude G. Biava, T Torchia, Paike Jayadeva Bhat, Lawrence M. Mylin, Gang Peng, Lucy B. Rowe, Robert W. Hamilton, Keith A. Bostian and Donald J. Tipper and has published in prestigious journals such as New England Journal of Medicine, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

James E. Hopper

100 papers receiving 3.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
James E. Hopper United States 39 3.2k 557 537 506 390 102 4.3k
Qian Yin United States 32 3.3k 1.0× 201 0.4× 147 0.3× 87 0.2× 128 0.3× 72 4.7k
Brian Wong United States 45 4.6k 1.4× 42 0.1× 334 0.6× 156 0.3× 233 0.6× 92 8.6k
Patrick Van Deŕ Smissen Belgium 31 1.9k 0.6× 130 0.2× 79 0.1× 149 0.3× 456 1.2× 69 3.7k
Kenneth Jacobs United States 20 992 0.3× 77 0.1× 261 0.5× 138 0.3× 292 0.7× 37 3.0k
Yupeng Wang China 15 5.9k 1.8× 998 1.8× 52 0.1× 516 1.0× 107 0.3× 20 7.2k
William J. Kaiser United States 41 6.3k 2.0× 191 0.3× 142 0.3× 40 0.1× 298 0.8× 64 8.7k
Jose G. Teodoro Canada 29 2.0k 0.6× 99 0.2× 84 0.2× 81 0.2× 1.0k 2.7× 47 3.4k
Edward C. Franklin United States 35 2.4k 0.7× 225 0.4× 60 0.1× 33 0.1× 418 1.1× 123 4.7k
Huabin He China 12 3.2k 1.0× 522 0.9× 46 0.1× 281 0.6× 77 0.2× 18 4.2k
Yongliang Zhang Singapore 31 1.9k 0.6× 167 0.3× 52 0.1× 170 0.3× 150 0.4× 87 3.5k

Countries citing papers authored by James E. Hopper

Since Specialization
Citations

This map shows the geographic impact of James E. Hopper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James E. Hopper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James E. Hopper more than expected).

Fields of papers citing papers by James E. Hopper

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James E. Hopper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James E. Hopper. The network helps show where James E. Hopper may publish in the future.

Co-authorship network of co-authors of James E. Hopper

This figure shows the co-authorship network connecting the top 25 collaborators of James E. Hopper. A scholar is included among the top collaborators of James E. Hopper based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James E. Hopper. James E. Hopper is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Jiang, Feng‐Lei, et al.. (2009). Gene Activation by Dissociation of an Inhibitor from a Transcriptional Activation Domain. Molecular and Cellular Biology. 29(20). 5604–5610. 34 indexed citations
2.
Diep, Cuong Q., et al.. (2008). Genetic Evidence for Sites of Interaction Between the Gal3 and Gal80 Proteins of the Saccharomyces cerevisiae GAL Gene Switch. Genetics. 178(2). 725–736. 14 indexed citations
3.
Mylin, Lawrence M. & James E. Hopper. (2003). Inducible Expression Cassettes in Yeast: GAL4. Humana Press eBooks. 62. 131–148. 1 indexed citations
4.
Levinson, Howard, James E. Hopper, & H. Paul Ehrlich. (2002). Overexpression of integrin αv promotes human osteosarcoma cell populated collagen lattice contraction and cell migration. Journal of Cellular Physiology. 193(2). 219–224. 15 indexed citations
5.
Hueston, William J., et al.. (2000). Why Are Antibiotics Prescribed for Patients With Acute Bronchitis? A Postintervention Analysis. The Journal of the American Board of Family Medicine. 13(6). 398–402. 12 indexed citations
7.
Long, Roy, Lawrence M. Mylin, & James E. Hopper. (1991). GAL11 (SPT13), a Transcriptional Regulator of Diverse Yeast Genes, Affects the Phosphorylation State of GAL4, a Highly Specific Transcriptional Activator. Molecular and Cellular Biology. 11(4). 2311–2314. 9 indexed citations
8.
Mylin, Lawrence M., et al.. (1991). Sequence conservation in theSaccharomycesandKluveromycesGAL11 transcription acvivators suggests functional domains. Nucleic Acids Research. 19(19). 5345–5350. 18 indexed citations
9.
Hopper, James E., et al.. (1990). Transcription of a Yeast Phosphoglucomutase Isozyme Gene Is Galactose Inducible and Glucose Repressible. Molecular and Cellular Biology. 10(4). 1415–1422. 56 indexed citations
10.
Sumner-Smith, Martin, et al.. (1985). Analysis of the inducible MEL1 gene of Saccharomyces carlsbergensis and its secreted product, alpha-galactosidase (melibiase). Gene. 36(3). 333–340. 57 indexed citations
11.
Hamilton, Robert W., et al.. (1984). Regulation of Basal and Induced Levels of the MEL1 Transcript in Saccharomyces cerevisiae. Molecular and Cellular Biology. 4(7). 1238–1245. 20 indexed citations
12.
Hopper, James E., Claude G. Biava, & Wu-Hao Tu. (1981). Membranous nephropathy: high-dose alternate-day therapy with prednisone.. PubMed. 135(1). 1–8. 9 indexed citations
13.
Bostian, Keith A., James E. Hopper, David T. Rogers, & Donald J. Tipper. (1980). Translational analysis of the killer-associated virus-like particle dsRNA genome of S. cerevisiae: M dsRNA encodes toxin. Cell. 19(2). 403–414. 132 indexed citations
14.
Shearn, Martin A., James E. Hopper, & Claude G. Biava. (1980). Membranous lupus nephropathy initially seen as idiopathic membranous nephropathy. Possible diagnostic value of tubular reticular structures.. PubMed. 140(11). 1521–3. 24 indexed citations
15.
Perlman, Daniel & James E. Hopper. (1979). Constitutive synthesis of the GAL4 protein, a galactose pathway regulator in saccharomyces cerevisiae. Cell. 16(1). 89–95. 105 indexed citations
17.
Hopper, James E. & S. J. Peloquin. (1976). Analysis of stylar self-incompatibility competence by use of heat induced inactivation. Theoretical and Applied Genetics. 47(6). 291–297. 3 indexed citations
18.
Rosenau, Werner, et al.. (1970). KIDNEY TRANSPLANTATION FOR RAPIDLY PROGRESSIVE GLOMERULONEPHRITIS. The Lancet. 296(7665). 180–182. 15 indexed citations
19.
Hopper, James E., et al.. (1963). Creatinine clearance in clinical medicine.. PubMed. 98. 121–8. 2 indexed citations
20.
Hopper, James E., et al.. (1961). Renal lesions in pregnancy. Clinical observations and light and electron microscopic findings.. PubMed. 17. 271–93. 37 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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