Jake A. Kloeber

1.7k total citations
22 papers, 514 citations indexed

About

Jake A. Kloeber is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Jake A. Kloeber has authored 22 papers receiving a total of 514 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 9 papers in Oncology and 4 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Jake A. Kloeber's work include DNA Repair Mechanisms (10 papers), CRISPR and Genetic Engineering (4 papers) and Asthma and respiratory diseases (4 papers). Jake A. Kloeber is often cited by papers focused on DNA Repair Mechanisms (10 papers), CRISPR and Genetic Engineering (4 papers) and Asthma and respiratory diseases (4 papers). Jake A. Kloeber collaborates with scholars based in United States, China and Hong Kong. Jake A. Kloeber's co-authors include Zhenkun Lou, Fei Zhao, Wootae Kim, Sergei I. Ochkur, Alfred D. Doyle, Jinzhou Huang, Elizabeth A. Jacobsen, Kelly P. Shim, Ming Gao and Nancy A. Lee and has published in prestigious journals such as Nature, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Jake A. Kloeber

18 papers receiving 513 citations

Peers

Jake A. Kloeber
Y Basaki Japan
Natalie Briones United States
F. Guddo Italy
Klas Strååt Sweden
Jaroslav Žák United States
Masao Naruke Japan
Stefan Thiem Australia
Mi-Yeon Jung South Korea
Shay Hantisteanu Israel
Y Basaki Japan
Jake A. Kloeber
Citations per year, relative to Jake A. Kloeber Jake A. Kloeber (= 1×) peers Y Basaki

Countries citing papers authored by Jake A. Kloeber

Since Specialization
Citations

This map shows the geographic impact of Jake A. Kloeber's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jake A. Kloeber with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jake A. Kloeber more than expected).

Fields of papers citing papers by Jake A. Kloeber

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jake A. Kloeber. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jake A. Kloeber. The network helps show where Jake A. Kloeber may publish in the future.

Co-authorship network of co-authors of Jake A. Kloeber

This figure shows the co-authorship network connecting the top 25 collaborators of Jake A. Kloeber. A scholar is included among the top collaborators of Jake A. Kloeber based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jake A. Kloeber. Jake A. Kloeber is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kloeber, Jake A., Michael G. Haddock, Yasamin Sharifzadeh, et al.. (2026). Clinical Evidence on Particle Radiation, DNA Damage Response Inhibitors, and Immunotherapy for Mismatch Repair-Proficient Rectal Cancer. Cancers. 18(4). 652–652.
2.
Ma, Jiasen, Nicholas B. Remmes, Jake A. Kloeber, et al.. (2025). Linear energy transfer optimized proton therapy for rectal cancer. Radiotherapy and Oncology. 207. 110850–110850. 1 indexed citations
3.
Kloeber, Jake A., Bin Chen, Guangchao Sun, et al.. (2025). KCTD10 is a sensor for co-directional transcription–replication conflicts. Nature. 648(8092). 210–219.
4.
Han, Yiqun, Qin Zhou, Bin Chen, et al.. (2025). Poly ADP Ribose Polymerase Inhibitors Potentiate Proton Therapy End-of-Range Effects by Accelerating Replication Forks and Promoting Transcription Conflict. International Journal of Radiation Oncology*Biology*Physics. 124(2). 451–464.
5.
Kloeber, Jake A., et al.. (2024). Proton Stereotactic Body Radiotherapy for Liver Metastases From Malignant Pancreatic Insulinoma. JCEM Case Reports. 2(10). luae175–luae175.
6.
Hou, Jing, Huanyao Gao, Qi Hu, et al.. (2023). SUMOylation of HNRNPA2B1 modulates RPA dynamics during unperturbed replication and genotoxic stress responses. Molecular Cell. 83(4). 539–555.e7. 21 indexed citations
7.
Gao, Ming, Wenya Feng, Hongyang Huang, et al.. (2022). m6A demethylation of cytidine deaminase APOBEC3B mRNA orchestrates arsenic-induced mutagenesis. Journal of Biological Chemistry. 298(2). 101563–101563. 20 indexed citations
8.
Gao, Ming, Min Deng, Hongyang Huang, et al.. (2022). The deubiquitinase USP7 regulates oxidative stress through stabilization of HO-1. Oncogene. 41(33). 4018–4027. 24 indexed citations
9.
Kloeber, Jake A. & Zhenkun Lou. (2021). Critical DNA damaging pathways in tumorigenesis. Seminars in Cancer Biology. 85. 164–184. 12 indexed citations
10.
Kim, Wootae, Fei Zhao, Huanyao Gao, et al.. (2021). USP13 regulates the replication stress response by deubiquitinating TopBP1. DNA repair. 100. 103063–103063. 17 indexed citations
11.
Zhu, Qian, Jinzhou Huang, Hongyang Huang, et al.. (2021). RNF19A-mediated ubiquitination of BARD1 prevents BRCA1/BARD1-dependent homologous recombination. Nature Communications. 12(1). 16 indexed citations
12.
Frede, Julia, Praveen Anand, Ricardo A. Pinto, et al.. (2021). Dynamic transcriptional reprogramming leads to immunotherapeutic vulnerabilities in myeloma. Nature Cell Biology. 23(11). 1199–1211. 26 indexed citations
13.
Gao, Ming, Guijie Guo, Jinzhou Huang, et al.. (2020). USP52 regulates DNA end resection and chemosensitivity through removing inhibitory ubiquitination from CtIP. Nature Communications. 11(1). 5362–5362. 25 indexed citations
14.
Zhao, Fei, Wootae Kim, Jake A. Kloeber, & Zhenkun Lou. (2020). DNA end resection and its role in DNA replication and DSB repair choice in mammalian cells. Experimental & Molecular Medicine. 52(10). 1705–1714. 91 indexed citations
15.
Yan, Yuanliang, Zhijie Xu, Jinzhou Huang, et al.. (2020). The deubiquitinase USP36 Regulates DNA replication stress and confers therapeutic resistance through PrimPol stabilization. Nucleic Acids Research. 48(22). 12711–12726. 35 indexed citations
16.
Jeganathan, Karthik B., Jake A. Kloeber, Brian A. Davies, et al.. (2020). FoxM1 insufficiency hyperactivates Ect2–RhoA–mDia1 signaling to drive cancer. Nature Cancer. 1(10). 1010–1024. 4 indexed citations
17.
Doyle, Alfred D., Manali Mukherjee, William E. LeSuer, et al.. (2019). Eosinophil-derived IL-13 promotes emphysema. European Respiratory Journal. 53(5). 1801291–1801291. 59 indexed citations
18.
Frede, Julia, Praveen Anand, Tushara Vijaykumar, et al.. (2019). Defining the differentiation states of multiple myeloma at single cell resolution – Identifying opportunities for immunotherapy. Clinical Lymphoma Myeloma & Leukemia. 19(10). e65–e65. 1 indexed citations
19.
Lee, James J., Cheryl Protheroe, Huijun Luo, et al.. (2014). Eosinophil-dependent skin innervation and itching following contact toxicant exposure in mice. Journal of Allergy and Clinical Immunology. 135(2). 477–487.e1. 28 indexed citations
20.
Doyle, Alfred D., Elizabeth A. Jacobsen, Sergei I. Ochkur, et al.. (2013). Homologous recombination into the eosinophil peroxidase locus generates a strain of mice expressing Cre recombinase exclusively in eosinophils. Journal of Leukocyte Biology. 94(1). 17–24. 70 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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