Jacob Hecksher‐Sørensen

7.1k total citations · 3 hit papers
55 papers, 4.7k citations indexed

About

Jacob Hecksher‐Sørensen is a scholar working on Molecular Biology, Surgery and Genetics. According to data from OpenAlex, Jacob Hecksher‐Sørensen has authored 55 papers receiving a total of 4.7k indexed citations (citations by other indexed papers that have themselves been cited), including 37 papers in Molecular Biology, 24 papers in Surgery and 18 papers in Genetics. Recurrent topics in Jacob Hecksher‐Sørensen's work include Pancreatic function and diabetes (22 papers), Diabetes and associated disorders (15 papers) and Congenital heart defects research (10 papers). Jacob Hecksher‐Sørensen is often cited by papers focused on Pancreatic function and diabetes (22 papers), Diabetes and associated disorders (15 papers) and Congenital heart defects research (10 papers). Jacob Hecksher‐Sørensen collaborates with scholars based in Denmark, United States and United Kingdom. Jacob Hecksher‐Sørensen's co-authors include Palle Serup, James Sharpe, Patrick Collombat, Ahmed Mansouri, Ulf Ahlgren, Paul Perry, Bill Hill, Allyson Ross, Duncan Davidson and Richard Baldock and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Clinical Investigation.

In The Last Decade

Jacob Hecksher‐Sørensen

54 papers receiving 4.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Optical Projection Tomography as a Tool for 3D Microscopy and Gene Expression Studies

2002 889 citations James Sharpe, Ulf Ahlgren et al. profile →
The arcuate nucleus mediates GLP-1 receptor agonist liraglutide-dependent weight loss

2014 702 citations Jacob Jelsing, Jacob Hecksher‐Sørensen et al. profile →
The GLP-1 Analogs Liraglutide and Semaglutide Reduce Atherosclerosis in ApoE−/− and LDLr−/− Mice by a Mechanism That Includes Inflammatory Pathways

2018 350 citations Robert Augustin, Jacob Hecksher‐Sørensen et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Jacob Hecksher‐Sørensen Denmark	27	2.2k	2.0k	1.7k	1.3k	407	55	4.7k
Heiko Lickert Germany	51	2.0k 0.9×	5.3k 2.7×	719 0.4×	1.7k 1.3×	187 0.5×	149	7.6k
Richard K.P. Benninger United States	32	1.5k 0.7×	1.3k 0.7×	697 0.4×	740 0.5×	414 1.0×	74	3.1k
Michele Solimena Germany	51	3.3k 1.5×	3.0k 1.5×	2.0k 1.2×	3.2k 2.3×	88 0.2×	136	8.5k
Lori Sussel United States	45	5.0k 2.3×	5.1k 2.6×	2.2k 1.4×	3.5k 2.6×	27 0.1×	104	9.4k
Mahlon D. Johnson United States	38	545 0.3×	1.7k 0.9×	321 0.2×	212 0.2×	147 0.4×	142	4.8k
Tune H. Pers Denmark	27	287 0.1×	1.5k 0.8×	187 0.1×	716 0.5×	76 0.2×	58	3.2k
Andreas F. Mack Germany	44	387 0.2×	2.6k 1.3×	238 0.1×	276 0.2×	74 0.2×	160	5.7k
John R. Fike United States	48	833 0.4×	2.4k 1.2×	84 0.1×	339 0.3×	123 0.3×	113	9.9k
Vincenzo Esposito Italy	48	1.3k 0.6×	1.2k 0.6×	1.2k 0.7×	265 0.2×	12 0.0×	248	6.7k
Marina Mione Italy	39	319 0.1×	4.1k 2.1×	128 0.1×	365 0.3×	359 0.9×	108	6.9k

Countries citing papers authored by Jacob Hecksher‐Sørensen

Since Specialization

Citations

This map shows the geographic impact of Jacob Hecksher‐Sørensen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jacob Hecksher‐Sørensen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jacob Hecksher‐Sørensen more than expected).

Fields of papers citing papers by Jacob Hecksher‐Sørensen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jacob Hecksher‐Sørensen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jacob Hecksher‐Sørensen. The network helps show where Jacob Hecksher‐Sørensen may publish in the future.

Co-authorship network of co-authors of Jacob Hecksher‐Sørensen

This figure shows the co-authorship network connecting the top 25 collaborators of Jacob Hecksher‐Sørensen. A scholar is included among the top collaborators of Jacob Hecksher‐Sørensen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Jacob Hecksher‐Sørensen. Jacob Hecksher‐Sørensen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Augustin, Robert, Anouk Oldenburger, Jens Markus Borghardt, et al.. (2025). Novel NPY2R agonist BI 1820237 provides synergistic anti-obesity efficacy when combined with the GCGR/GLP-1R dual agonist survodutide. Molecular Metabolism. 99. 102205–102205. 1 indexed citations

Dedic, Nina, Lien Wang, Éva Hajós‐Korcsok, et al.. (2024). TAAR1 agonists improve glycemic control, reduce body weight and modulate neurocircuits governing energy balance and feeding. Molecular Metabolism. 80. 101883–101883. 11 indexed citations

Fritzen, Andreas M., Jacob Lercke Skytte, Jens Lund, et al.. (2024). Atlas of exercise-induced brain activation in mice. Molecular Metabolism. 82. 101907–101907. 10 indexed citations

Barbut, Denise, William A. Kinney, Hsiao‐Huei Chen, et al.. (2024). A novel, centrally acting mammalian aminosterol, ENT ‐03, induces weight loss in obese and lean rodents. Diabetes Obesity and Metabolism. 26(12). 5701–5712.

Roostalu, Urmas, Casper G. Salinas, Jacob Lercke Skytte, et al.. (2023). Uncovering CNS access of lipidated exendin-4 analogues by quantitative whole-brain 3D light sheet imaging. Neuropharmacology. 238. 109637–109637. 16 indexed citations

Dedic, Nina, Philip Jones, Éva Hajós‐Korcsok, et al.. (2023). TAAR1 Agonist Ulotaront Improves Glycemic Control and Reduces Body Weight in Rodent Models of Diabetes, Obesity, and Iatrogenic Weight Gain. CNS Spectrums. 28(2). 259–259. 1 indexed citations

Roostalu, Urmas, Henrik H. Hansen, Thomas A. Lutz, et al.. (2021). Whole‐brain mapping of amylin‐induced neuronal activity in receptor activity–modifying protein 1/3 knockout mice. European Journal of Neuroscience. 54(1). 4154–4166. 13 indexed citations

Hansen, Henrik H., Urmas Roostalu, Jacob Lercke Skytte, et al.. (2021). Whole-brain activation signatures of weight-lowering drugs. Molecular Metabolism. 47. 101171–101171. 39 indexed citations

Østergaard, Mette V., Jacob Lercke Skytte, Urmas Roostalu, et al.. (2020). Automated Image Analyses of Glomerular Hypertrophy in a Mouse Model of Diabetic Nephropathy. Kidney360. 1(6). 469–479. 16 indexed citations

10.

Wismann, Pernille, Christina Rye Underwood, Nikolaj Kulahin, et al.. (2013). Real-time trafficking and signaling of the glucagon-like peptide-1 receptor. Molecular and Cellular Endocrinology. 382(2). 938–949. 125 indexed citations

11.

Fomina‐Yadlin, Dina, Stefan Kubicek, Deepika Walpita, et al.. (2010). Small-molecule inducers of insulin expression in pancreatic α-cells. Proceedings of the National Academy of Sciences. 107(34). 15099–15104. 58 indexed citations

12.

Jensen, Jan, et al.. (2010). The transcriptional activity of Neurog3 affects migration and differentiation of ectopic endocrine cells in chicken endoderm. Developmental Dynamics. 239(7). 1950–1966. 8 indexed citations

13.

Wang, Sui, Jacob Hecksher‐Sørensen, Yanwen Xu, et al.. (2008). Myt1 and Ngn3 form a feed-forward expression loop to promote endocrine islet cell differentiation. Developmental Biology. 317(2). 531–540. 72 indexed citations

14.

Ahnfelt‐Rønne, Jonas, et al.. (2007). Preservation of proliferating pancreatic progenitor cells by Delta-Notch signaling in the embryonic chicken pancreas. BMC Developmental Biology. 7(1). 63–63. 36 indexed citations

15.

Sharpe, James, Robert P. Watson, Jacob Hecksher‐Sørensen, et al.. (2006). Spleen versus pancreas: strict control of organ interrelationship revealed by analyses of Bapx1 ^−/− mice. Genes & Development. 20(16). 2208–2213. 53 indexed citations

16.

Hecksher‐Sørensen, Jacob, Robert P. Watson, Laura A. Lettice, et al.. (2004). The splanchnic mesodermal plate directs spleen and pancreatic laterality,and is regulated by Bapx1/Nkx3.2. Development. 131(19). 4665–4675. 86 indexed citations

17.

Collombat, Patrick, Ahmed Mansouri, Jacob Hecksher‐Sørensen, et al.. (2003). Opposing actions of Arx and Pax4 in endocrine pancreas development. Genes & Development. 17(20). 2591–2603. 427 indexed citations

18.

Hecksher‐Sørensen, Jacob & James Sharpe. (2001). 3D confocal reconstruction of gene expression in mouse. Mechanisms of Development. 100(1). 59–63. 30 indexed citations

19.

Lettice, Laura A., Jacob Hecksher‐Sørensen, & Robert E. Hill. (1999). The dominant hemimelia mutation uncouples epithelial-mesenchymal interactions and disrupts anterior mesenchyme formation in mouse hindlimbs. Development. 126(21). 4729–4736. 15 indexed citations

20.

Sharpe, James, Laura A. Lettice, Jacob Hecksher‐Sørensen, et al.. (1999). Identification of Sonic hedgehog as a candidate gene responsible for the polydactylous mouse mutant Sasquatch. Current Biology. 9(2). 97–S1. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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