J. Thomas Pento

793 total citations
68 papers, 607 citations indexed

About

J. Thomas Pento is a scholar working on Molecular Biology, Genetics and Oncology. According to data from OpenAlex, J. Thomas Pento has authored 68 papers receiving a total of 607 indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 24 papers in Genetics and 12 papers in Oncology. Recurrent topics in J. Thomas Pento's work include Estrogen and related hormone effects (20 papers), Fibroblast Growth Factor Research (13 papers) and Kruppel-like factors research (10 papers). J. Thomas Pento is often cited by papers focused on Estrogen and related hormone effects (20 papers), Fibroblast Growth Factor Research (13 papers) and Kruppel-like factors research (10 papers). J. Thomas Pento collaborates with scholars based in United States, Kuwait and France. J. Thomas Pento's co-authors include Xiaoping Zang, Daniel J. Brackett, Eric W. Howard, S. M. A. Abidi, Megan R. Lerner, Ana M. Tari, Seymour M. Glick, M. Margaret King, Avir Kagan and K. Meyer and has published in prestigious journals such as Endocrinology, Journal of Medicinal Chemistry and American Journal of Physiology-Endocrinology and Metabolism.

In The Last Decade

J. Thomas Pento

68 papers receiving 588 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
J. Thomas Pento United States 14 300 142 124 68 52 68 607
Α. Φωτίου United Kingdom 14 339 1.1× 208 1.5× 99 0.8× 97 1.4× 40 0.8× 23 755
Sonia Hovsépian France 14 276 0.9× 66 0.5× 109 0.9× 60 0.9× 33 0.6× 33 539
Dany Rouillard France 14 387 1.3× 287 2.0× 46 0.4× 93 1.4× 33 0.6× 21 702
Jae Won Soh United States 12 437 1.5× 258 1.8× 89 0.7× 87 1.3× 50 1.0× 12 749
K. Akai Japan 13 377 1.3× 51 0.4× 51 0.4× 28 0.4× 47 0.9× 20 727
Anne John United Arab Emirates 14 397 1.3× 140 1.0× 139 1.1× 66 1.0× 18 0.3× 48 841
M. Asamoto Japan 13 547 1.8× 60 0.4× 96 0.8× 143 2.1× 18 0.3× 26 842
Toshiaki Segawa Japan 9 359 1.2× 81 0.6× 42 0.3× 71 1.0× 23 0.4× 9 621
Shabana Shabbeer United States 13 829 2.8× 233 1.6× 109 0.9× 73 1.1× 28 0.5× 18 1.1k
N. Martel France 15 505 1.7× 227 1.6× 60 0.5× 144 2.1× 16 0.3× 30 838

Countries citing papers authored by J. Thomas Pento

Since Specialization
Citations

This map shows the geographic impact of J. Thomas Pento's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Thomas Pento with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Thomas Pento more than expected).

Fields of papers citing papers by J. Thomas Pento

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. Thomas Pento. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Thomas Pento. The network helps show where J. Thomas Pento may publish in the future.

Co-authorship network of co-authors of J. Thomas Pento

This figure shows the co-authorship network connecting the top 25 collaborators of J. Thomas Pento. A scholar is included among the top collaborators of J. Thomas Pento based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. Thomas Pento. J. Thomas Pento is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Pento, J. Thomas. (2017). Monoclonal Antibodies for the Treatment of Cancer. Anticancer Research. 37(11). 5935–5939. 73 indexed citations
2.
Harrison, Roger G., et al.. (2009). Selective Growth Inhibition of Cancer Cells by <i>L</i>-Methioninase-Containing Fusion Protein Targeted to the Urokinase Receptor. Pharmacology. 84(5). 271–275. 6 indexed citations
3.
Hackett, John C., Zili Xiao, Xiaoping Zang, et al.. (2008). Development of keratinocyte growth factor receptor tyrosine kinase inhibitors for the treatment of cancer.. PubMed. 27(6B). 3801–6. 10 indexed citations
4.
Zang, Xiaoping, et al.. (2006). A Comparison of KGF Receptor Expression in Various Types of Human Cancer.. PubMed. 3(6). 369–372. 3 indexed citations
5.
Zang, Xiaoping, et al.. (2006). Effect of Antiestrogens on EGF-Mediated Movement of Human Breast Cancer Cells. Pharmacology. 79(2). 93–96. 1 indexed citations
6.
Zang, Xiaoping, et al.. (2004). Keratinocyte Growth Factor-Mediated Pattern of Gene Expression in Breast Cancer Cells.. PubMed. 1(4). 339–344. 4 indexed citations
7.
Zang, Xiaoping, et al.. (2004). KGF-induced motility of breast cancer cells is dependent on Grb2 and Erk1,2. Clinical & Experimental Metastasis. 21(5). 437–443. 21 indexed citations
8.
Zang, Xiaoping, et al.. (2003). Measurement of beta-galactosidase tissue levels in a tumor cellxenograft model. Methods and Findings in Experimental and Clinical Pharmacology. 25(9). 713–713. 19 indexed citations
9.
Pento, J. Thomas, et al.. (2000). Influence of Antiestrogens on NIH-3T3-Fibroblast-Induced Motility of Breast Cancer Cells. Chemotherapy. 47(1). 56–69. 10 indexed citations
10.
Pento, J. Thomas, et al.. (2000). Keratinocyte growth factor-induced motility of breast cancer cells. Clinical & Experimental Metastasis. 18(7). 573–580. 32 indexed citations
11.
Pento, J. Thomas, et al.. (1997). Antitumor activity of a novel antiestrogen (Analog II) on human breast cancer cells. Anti-Cancer Drugs. 8(10). 964–973. 9 indexed citations
12.
Abidi, S. M. A., et al.. (1997). Differential influence of antiestrogens on the in vitro release of gelatinases (type IV collagenases) by invasive and non-invasive breast cancer cells. Clinical & Experimental Metastasis. 15(4). 432–439. 24 indexed citations
13.
Pento, J. Thomas, et al.. (1996). Antitumor mechanism of action of a cyclopropyl antiestrogen (compound 7b) on human breast cancer cells in culture. Cancer Chemotherapy and Pharmacology. 38(3). 238–244. 7 indexed citations
14.
15.
Pento, J. Thomas, et al.. (1993). The influence of a novel cyclopropyl antiestrogen (compound 7a) on human breast cancer cells in culture. Breast Cancer Research and Treatment. 25(3). 225–233. 3 indexed citations
16.
Pento, J. Thomas, et al.. (1991). Antiproliferative activity of a series of novel cyclopropyl antiestrogens on MCF-7 human breast cancer cells in culture. Anti-Cancer Drugs. 2(5). 487–494. 6 indexed citations
17.
Day, Billy W., et al.. (1991). Synthesis and biological evaluation of a series of 1,1-dichloro-2,2,3-triarylcyclopropanes as pure antiestrogens. Journal of Medicinal Chemistry. 34(2). 842–851. 24 indexed citations
18.
Pento, J. Thomas, et al.. (1988). Biological Evaluation of Novel Cyclopropyl Analogues of Stilbene, Stilbenediol, and Phenanthrene for Estrogenic and Antiestrogenic Activity. Journal of Pharmaceutical Sciences. 77(2). 120–125. 5 indexed citations
19.
Pento, J. Thomas, et al.. (1988). Time-dependent deterioration of active transport in duodenal segments of rat intestine. Journal of Pharmacological Methods. 20(1). 9–14. 8 indexed citations
20.
Pento, J. Thomas. (1973). INFLUENCE OF WHOLE-BODY IRRADIATION ON CALCIUM TRANSPORT IN RAT SMALL INTESTINE. Proceedings of the Oklahoma Academy of Science. 53. 57–60. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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