J. Terrig Thomas

1.7k total citations
15 papers, 1.4k citations indexed

About

J. Terrig Thomas is a scholar working on Molecular Biology, Genetics and Rheumatology. According to data from OpenAlex, J. Terrig Thomas has authored 15 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 7 papers in Genetics and 2 papers in Rheumatology. Recurrent topics in J. Terrig Thomas's work include TGF-β signaling in diseases (8 papers), Developmental Biology and Gene Regulation (6 papers) and Wnt/β-catenin signaling in development and cancer (4 papers). J. Terrig Thomas is often cited by papers focused on TGF-β signaling in diseases (8 papers), Developmental Biology and Gene Regulation (6 papers) and Wnt/β-catenin signaling in development and cancer (4 papers). J. Terrig Thomas collaborates with scholars based in United States, Belgium and Australia. J. Terrig Thomas's co-authors include Frank P. Luyten, Malcolm Moos, Keming Lin, J Cervenka, Maurício Camargo, Bang H. Hoang, Christine A. Kozak, Shu‐Chun Chang, Nicholas J. P. Ryba and Slobodan Vukičević and has published in prestigious journals such as Journal of Biological Chemistry, Nature Genetics and The Journal of Cell Biology.

In The Last Decade

J. Terrig Thomas

15 papers receiving 1.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
J. Terrig Thomas United States	11	866	476	319	224	143	15	1.4k
Kuber T. Sampath United States	18	1.3k 1.5×	338 0.7×	285 0.9×	234 1.0×	96 0.7×	30	1.9k
Dina Lewinson Israel	21	542 0.6×	291 0.6×	265 0.8×	157 0.7×	110 0.8×	60	1.3k
H Stöß Germany	17	483 0.6×	945 2.0×	374 1.2×	261 1.2×	113 0.8×	49	1.6k
Marina D’Angelo United States	16	492 0.6×	320 0.7×	202 0.6×	91 0.4×	130 0.9×	20	1.1k
Noboru Yamaji Japan	9	860 1.0×	177 0.4×	158 0.5×	311 1.4×	84 0.6×	15	1.4k
Cristin M. Ferguson United States	18	605 0.7×	723 1.5×	141 0.4×	511 2.3×	178 1.2×	32	1.6k
Michael Ulrich‐Vinther Denmark	18	375 0.4×	363 0.8×	207 0.6×	676 3.0×	133 0.9×	22	1.3k
Motomi Enomoto‐Iwamoto United States	18	786 0.9×	1.2k 2.5×	290 0.9×	332 1.5×	41 0.3×	21	1.9k
Akitoshi Jikko Japan	18	481 0.6×	436 0.9×	88 0.3×	198 0.9×	119 0.8×	30	1.2k
S C Ghivizzani United States	12	465 0.5×	234 0.5×	343 1.1×	172 0.8×	76 0.5×	15	877

Countries citing papers authored by J. Terrig Thomas

Since Specialization

Citations

This map shows the geographic impact of J. Terrig Thomas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Terrig Thomas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Terrig Thomas more than expected).

Fields of papers citing papers by J. Terrig Thomas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. Terrig Thomas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Terrig Thomas. The network helps show where J. Terrig Thomas may publish in the future.

Co-authorship network of co-authors of J. Terrig Thomas

This figure shows the co-authorship network connecting the top 25 collaborators of J. Terrig Thomas. A scholar is included among the top collaborators of J. Terrig Thomas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. Terrig Thomas. J. Terrig Thomas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Thomas, J. Terrig, et al.. (2017). SMOC can act as both an antagonist and an expander of BMP signaling. eLife. 6. 22 indexed citations

Thomas, J. Terrig, et al.. (2016). SMOC Binds to Pro-EGF, but Does Not Induce Erk Phosphorylation via the EGFR. PLoS ONE. 11(4). e0154294–e0154294. 3 indexed citations

Thomas, J. Terrig, et al.. (2013). Cartilage repair and replacement in the knee: a regulatory perspective. Trends in biotechnology. 31(12). 665–667. 8 indexed citations

Sentürker, Sema, et al.. (2012). A Homolog of Subtilisin-Like Proprotein Convertase 7 Is Essential to Anterior Neural Development in Xenopus. PLoS ONE. 7(6). e39380–e39380. 11 indexed citations

Thomas, J. Terrig, et al.. (2009). Xenopus SMOC-1 Inhibits Bone Morphogenetic Protein Signaling Downstream of Receptor Binding and Is Essential for Postgastrulation Development in Xenopus. Journal of Biological Chemistry. 284(28). 18994–19005. 31 indexed citations

Wang, Wei, Eboneé N. Butler, Vic Veguilla, et al.. (2008). Establishment of retroviral pseudotypes with influenza hemagglutinins from H1, H3, and H5 subtypes for sensitive and specific detection of neutralizing antibodies. Journal of Virological Methods. 153(2). 111–119. 85 indexed citations

Thomas, J. Terrig & Malcolm Moos. (2007). Vg1 has specific processing requirements that restrict its action to body axis patterning centers. Developmental Biology. 310(1). 129–139. 2 indexed citations

Lories, Rik, Astrid D. Bakker, Przemko Tylżanowski, et al.. (2007). Articular cartilage and biomechanical properties of the long bones in Frzb‐knockout mice. Arthritis & Rheumatism. 56(12). 4095–4103. 228 indexed citations

Thomas, J. Terrig, et al.. (2006). CDMP1/GDF5 Has Specific Processing Requirements That Restrict Its Action to Joint Surfaces. Journal of Biological Chemistry. 281(36). 26725–26733. 14 indexed citations

10.

Tylżanowski, Przemko, Wouter Bossuyt, J. Terrig Thomas, & Frank P. Luyten. (2004). Characterization of Frzb‐Cre transgenic mouse. genesis. 40(4). 200–204. 7 indexed citations

11.

Everman, David B., Cynthia F. Bartels, Yue Yang, et al.. (2002). The mutational spectrum of brachydactyly type C. American Journal of Medical Genetics. 112(3). 291–296. 65 indexed citations

12.

Tsumaki, Noriyuki, Kazuhiro Tanaka, Eri Arikawa‐Hirasawa, et al.. (1999). Role of CDMP-1 in Skeletal Morphogenesis: Promotion of Mesenchymal Cell Recruitment and Chondrocyte Differentiation. The Journal of Cell Biology. 144(1). 161–173. 154 indexed citations

13.

Hoang, Bang H., J. Terrig Thomas, Fadi W. Abdul‐Karim, et al.. (1998). Expression pattern of twoFrizzled-related genes,Frzb-1 andSfrp-1, during mouse embryogenesis suggests a role for modulating action ofWnt family members. Developmental Dynamics. 212(3). 364–372. 81 indexed citations

14.

Thomas, J. Terrig, et al.. (1996). A human chondrodysplasia due to a mutation in a TGF-β superfamily member. Nature Genetics. 12(3). 315–317. 331 indexed citations

15.

Chang, Shu‐Chun, Bang H. Hoang, J. Terrig Thomas, et al.. (1994). Cartilage-derived morphogenetic proteins. New members of the transforming growth factor-beta superfamily predominantly expressed in long bones during human embryonic development.. Journal of Biological Chemistry. 269(45). 28227–28234. 346 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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