J. Stephen Fink
- Cellular and Molecular Neuroscience top 0.5%
- Molecular Biology top 2%
- Physiology top 0.05%
- Neurology top 2%
- Immunology top 5%
- Co-authors
- Jiang‐Fan ChenMichael A. SchwarzschildAlexia E. PollackGerard P. SmithRichard H. GoodmanRosario MoratallaRobert A. PeterfreundAviva J. Symes
- Topics
- Adenosine and Purinergic Signaling (20 papers)Neurotransmitter Receptor Influence on Behavior (17 papers)Receptor Mechanisms and Signaling (17 papers)
- Partner nations
- United StatesPolandJapan
In The Last Decade
J. Stephen Fink
73 papers receiving 6.1k citations
Hit Papers
Peers
Comparison fields: 5 of 116
- Cellular and Molecular Neuroscience 2.7k
- Molecular Biology 2.6k
- Physiology 2.2k
- Neurology 701
- Immunology 618
Countries citing papers authored by J. Stephen Fink
This map shows the geographic impact of J. Stephen Fink's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. Stephen Fink with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. Stephen Fink more than expected).
Fields of papers citing papers by J. Stephen Fink
This network shows the impact of papers produced by J. Stephen Fink. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. Stephen Fink. The network helps show where J. Stephen Fink may publish in the future.
Co-authorship network of co-authors of J. Stephen Fink
This figure shows the co-authorship network connecting the top 25 collaborators of J. Stephen Fink. A scholar is included among the top collaborators of J. Stephen Fink based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. Stephen Fink. J. Stephen Fink is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 30 | |
| 2 | 156 | |
| 3 | 194 | |
| 4 | 28 | |
| 5 | 108 | |
| 6 | 31 | |
| 7 | 51 | |
| 8 | 15 | |
| 9 | 5 | |
| 10 | 58 | |
| 11 | 85 | |
| 12 | 42 | |
| 13 | 13 | |
| 14 | 18 | |
| 15 | 61 | |
| 16 | 61 | |
| 17 | 25 | |
| 18 | 5 | |
| 19 | 122 | |
| 20 | 54 |
About J. Stephen Fink
J. Stephen Fink is a scholar working on Physiology, Cellular and Molecular Neuroscience and Molecular Biology, having authored 73 papers that have together received 6.3k indexed citations. Recurring topics across this work include Adenosine and Purinergic Signaling (20 papers), Neurotransmitter Receptor Influence on Behavior (17 papers) and Receptor Mechanisms and Signaling (17 papers). The work is most often cited by research in Physiology (2.2k citations), Cellular and Molecular Neuroscience (2.7k citations) and Endocrine and Autonomic Systems (483 citations). J. Stephen Fink has collaborated with scholars based in United States, Poland and Japan. Frequent co-authors include Jiang‐Fan Chen, Michael A. Schwarzschild, Alexia E. Pollack, Gerard P. Smith, Richard H. Goodman, Rosario Moratalla, Robert A. Peterfreund, Aviva J. Symes, David G. Standaert and Jinmin Zhu. Their work appears in journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.