J Rotta

646 total citations
53 papers, 524 citations indexed

About

J Rotta is a scholar working on Public Health, Environmental and Occupational Health, Molecular Biology and Epidemiology. According to data from OpenAlex, J Rotta has authored 53 papers receiving a total of 524 indexed citations (citations by other indexed papers that have themselves been cited), including 34 papers in Public Health, Environmental and Occupational Health, 16 papers in Molecular Biology and 15 papers in Epidemiology. Recurrent topics in J Rotta's work include Streptococcal Infections and Treatments (34 papers), Neonatal and Maternal Infections (15 papers) and Pneumonia and Respiratory Infections (6 papers). J Rotta is often cited by papers focused on Streptococcal Infections and Treatments (34 papers), Neonatal and Maternal Infections (15 papers) and Pneumonia and Respiratory Infections (6 papers). J Rotta collaborates with scholars based in Czechia, Vietnam and United States. J Rotta's co-authors include Richard M. Krause, Henry Lackland, Rebecca C. Lancefield, H Raŝková, K. Mašek, Walter W. Karakawa, E Tikhomirov, K Raska, Aziz El Kholy and M. Zaoral and has published in prestigious journals such as The Journal of Experimental Medicine, The Journal of Physiology and Journal of Bacteriology.

In The Last Decade

J Rotta

49 papers receiving 422 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
J Rotta Czechia 12 287 170 148 112 68 53 524
Rachel Haft United States 9 146 0.5× 74 0.4× 120 0.8× 101 0.9× 29 0.4× 11 376
M. Hastings United Kingdom 10 235 0.8× 34 0.2× 171 1.2× 92 0.8× 104 1.5× 14 505
John H. Wilkins United States 9 261 0.9× 85 0.5× 57 0.4× 102 0.9× 15 0.2× 18 870
J. K. S. Chia United States 13 59 0.2× 169 1.0× 121 0.8× 78 0.7× 139 2.0× 25 655
Kazuyo Fujita Japan 17 218 0.8× 42 0.2× 180 1.2× 240 2.1× 33 0.5× 57 764
Christine M. Sanfilippo United States 18 164 0.6× 126 0.7× 167 1.1× 174 1.6× 80 1.2× 44 892
Lara M. Jacobson United States 10 248 0.9× 166 1.0× 117 0.8× 42 0.4× 24 0.4× 15 719
Chi‐Jen Lee United States 16 73 0.3× 61 0.4× 343 2.3× 175 1.6× 110 1.6× 44 906
Paulo Câmara Marques Pereira Brazil 14 105 0.4× 167 1.0× 281 1.9× 93 0.8× 57 0.8× 60 666
Gérard Praz Switzerland 11 97 0.3× 193 1.1× 232 1.6× 255 2.3× 29 0.4× 23 759

Countries citing papers authored by J Rotta

Since Specialization
Citations

This map shows the geographic impact of J Rotta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J Rotta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J Rotta more than expected).

Fields of papers citing papers by J Rotta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J Rotta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J Rotta. The network helps show where J Rotta may publish in the future.

Co-authorship network of co-authors of J Rotta

This figure shows the co-authorship network connecting the top 25 collaborators of J Rotta. A scholar is included among the top collaborators of J Rotta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J Rotta. J Rotta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Majeed, H. A., et al.. (1991). Human heart sarcolemmal sheath antibodies in children with non-suppurative sequelae of group A streptococcal infections: a follow up study.. Annals of the Rheumatic Diseases. 50(11). 752–754. 1 indexed citations
2.
Rotta, J, et al.. (1989). Prospective Study of Pharyngitis: Clinical Diagnosis and Microbiological Profile. Zentralblatt für Bakteriologie. 271(4). 532–542. 4 indexed citations
4.
Ježek, Jan, et al.. (1988). Muramyl-dipeptide analogues: Synthesis and biological activities. Collection of Czechoslovak Chemical Communications. 53(11). 2897–2906. 3 indexed citations
5.
Rotta, J, et al.. (1988). The rheumatogenic and nephritogenic strains of the group A streptococcus: the Kuwait experience.. PubMed. 101(847 Pt 2). 398–401. 6 indexed citations
6.
Lavický, J., et al.. (1986). Changes of pharmacokinetics of Trimethoprim after pretreatment with streptococcal Peptidoglycan. European Journal of Drug Metabolism and Pharmacokinetics. 11(1). 17–22. 8 indexed citations
7.
Majeed, H. A., et al.. (1986). The Concurrent Associations of Group A Streptococcal Serotypes in Children with Acute Rheumatic Fever or Pharyngitis-Associated Glomerulonephritis and their Families in Kuwait. Zentralblatt für Bakteriologie Mikrobiologie und Hygiene Series A Medical Microbiology Infectious Diseases Virology Parasitology. 262(3). 346–356. 5 indexed citations
8.
Pekárek, J, et al.. (1985). Biological activity of synthetic subunits of streptococcus peptidoglycan. III. Relationship of subunit and analogue structure to adjuvant activity in cell-mediated immunity.. PubMed. 53(5). 260–4. 1 indexed citations
9.
Lavický, J., et al.. (1983). The influence of streptococcal peptidoglycane on neonate pharmacokinetics of trimethoprim. Ghent University Academic Bibliography (Ghent University). 122. 1374–1377. 1 indexed citations
10.
Rotta, J. (1983). Pathogenesis of rheumatic fever: a review. Annals of Tropical Paediatrics. 3(1). 1–8. 2 indexed citations
11.
Rotta, J, et al.. (1978). Biological characteristics of peptidoglycans of group A streptococcus and some other bacterial species. II. Immunological mechanisms involved in thrombocytolysis.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 22(4). 435–41. 4 indexed citations
12.
Jelínková, J, et al.. (1978). Nine-year study of streptococcal infections in a sample of the general population.. PubMed. 22(2). 162–76. 3 indexed citations
13.
Paegelow, I., et al.. (1973). Hemodynamic Effects of Group A Streptococcus Mucopeptide. Experimental Biology and Medicine. 144(3). 1020–1024. 5 indexed citations
14.
Rotta, J, et al.. (1971). NEW APPROACHES FOR THE LABORATORY RECOGNITION OF M TYPES OF GROUP A STREPTOCOCCI. The Journal of Experimental Medicine. 134(5). 1298–1315. 122 indexed citations
15.
Rotta, J, et al.. (1969). BIOLOGICAL PROPERTIES OF CELL WALL MUCOPEPTIDE OF HEMOLYTIC STREPTOCOCCI. The Journal of Experimental Medicine. 130(1). 31–47. 21 indexed citations
16.
Jelínková, J, et al.. (1967). Some manifestations of a relationship between group A and L streptococci. Preliminary report.. PubMed. 11(3). 353–8. 2 indexed citations
17.
Rotta, J, et al.. (1965). ENHANCED RESISTANCE TO STREPTOCOCCAL INFECTION INDUCED IN MICE BY CELL WALL MUCOPEPTIDE. The Journal of Experimental Medicine. 122(5). 877–890. 22 indexed citations
18.
Raska, K & J Rotta. (1963). NON-SPECIFIC RESISTANCE OF MICE TO STREPTOCOCCAL INFECTION INDUCED BY A STREPTOCOCCI OR THEIR CELLULAR COMPONENTS.. PubMed. 7. 319–26. 3 indexed citations
19.
Raska, K & J Rotta. (1956). [Persistence of streptococci of Group A after intranasal infection].. PubMed. 19(3). 356–61. 1 indexed citations
20.
Raska, K & J Rotta. (1956). Die Persistenz von Streptokokken der Gruppe A nach intranasaler Infektion. Pathobiology. 19(3). 356–361. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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