J. I. Last

520 total citations
8 papers, 360 citations indexed

About

J. I. Last is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, J. I. Last has authored 8 papers receiving a total of 360 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 3 papers in Oncology and 3 papers in Cancer Research. Recurrent topics in J. I. Last's work include DNA Repair Mechanisms (6 papers), Carcinogens and Genotoxicity Assessment (3 papers) and Cancer-related Molecular Pathways (2 papers). J. I. Last is often cited by papers focused on DNA Repair Mechanisms (6 papers), Carcinogens and Genotoxicity Assessment (3 papers) and Cancer-related Molecular Pathways (2 papers). J. I. Last collaborates with scholars based in United Kingdom, Netherlands and Ireland. J. I. Last's co-authors include A. Malcolm R. Taylor, P.J. Byrd, Corry M.R. Weemaes, M.M.M. Verhagen, Michèl A.A.P. Willemsen, Venkataramanan Srinivasan, Philip J. Byrd, J.A.P. Hiel, Annegien Broeks and Anne Reiman and has published in prestigious journals such as Neurology, British Journal of Cancer and Journal of Neurology Neurosurgery & Psychiatry.

In The Last Decade

J. I. Last

8 papers receiving 357 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
J. I. Last United Kingdom	7	293	119	95	89	47	8	360
M.M.M. Verhagen Netherlands	4	209 0.7×	81 0.7×	73 0.8×	69 0.8×	39 0.8×	4	281
Nienke J.H. van Os Netherlands	9	278 0.9×	117 1.0×	92 1.0×	94 1.1×	94 2.0×	16	427
Yasuyo Chochi Japan	10	227 0.8×	80 0.7×	81 0.9×	80 0.9×	40 0.9×	14	437
Szymon Janczar Poland	10	188 0.6×	44 0.4×	35 0.4×	54 0.6×	101 2.1×	29	338
Christina Manetopoulos Sweden	8	276 0.9×	75 0.6×	18 0.2×	110 1.2×	36 0.8×	8	377
Sana Sfar Tunisia	8	185 0.6×	117 1.0×	43 0.5×	72 0.8×	54 1.1×	12	318
A. Kidd United Kingdom	4	264 0.9×	105 0.9×	52 0.5×	118 1.3×	94 2.0×	4	320
Florian Lépinasse France	11	203 0.7×	30 0.3×	107 1.1×	142 1.6×	22 0.5×	12	394
April Nelson United States	5	329 1.1×	45 0.4×	73 0.8×	57 0.6×	23 0.5×	9	406
Morad Khayat Israel	7	118 0.4×	39 0.3×	48 0.5×	86 1.0×	40 0.9×	11	311

Countries citing papers authored by J. I. Last

Since Specialization

Citations

This map shows the geographic impact of J. I. Last's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by J. I. Last with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites J. I. Last more than expected).

Fields of papers citing papers by J. I. Last

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by J. I. Last. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by J. I. Last. The network helps show where J. I. Last may publish in the future.

Co-authorship network of co-authors of J. I. Last

This figure shows the co-authorship network connecting the top 25 collaborators of J. I. Last. A scholar is included among the top collaborators of J. I. Last based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with J. I. Last. J. I. Last is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Last, J. I., et al.. (2020). MFG-E8-derived peptide attenuates inflammation and injury after renal ischemia-reperfusion in mice. Heliyon. 6(12). e05794–e05794. 6 indexed citations

2.

Gurien, Steven D., Monowar Aziz, Naomi‐Liza Denning, et al.. (2020). An extracellular cold-inducible RNA-binding protein-derived small peptide targeting triggering receptor expressed on myeloid cells-1 attenuates hemorrhagic shock. The Journal of Trauma: Injury, Infection, and Critical Care. 88(6). 809–815. 11 indexed citations

3.

Taylor, A. Malcolm R., et al.. (2014). Ataxia telangiectasia: more variation at clinical and cellular levels. Clinical Genetics. 87(3). 199–208. 74 indexed citations

4.

Reiman, Anne, Venkataramanan Srinivasan, Giancarlo Barone, et al.. (2011). Lymphoid tumours and breast cancer in ataxia telangiectasia; substantial protective effect of residual ATM kinase activity against childhood tumours. British Journal of Cancer. 105(4). 586–591. 100 indexed citations

5.

Byrd, Philip J., Venkataramanan Srinivasan, J. I. Last, et al.. (2011). Severe reaction to radiotherapy for breast cancer as the presenting feature of ataxia telangiectasia. British Journal of Cancer. 106(2). 262–268. 32 indexed citations

6.

Cawley, Niamh, et al.. (2010). POG02 Variant ataxia telangiectasia in siblings with normal -fetoprotein levels. Journal of Neurology Neurosurgery & Psychiatry. 81(11). e48–e48. 3 indexed citations

7.

Verhagen, M.M.M., Wilson F. Abdo, Michèl A.A.P. Willemsen, et al.. (2009). Clinical spectrum of ataxia-telangiectasia in adulthood. Neurology. 73(6). 430–437. 108 indexed citations

8.

Hiel, J.A.P., Baziel G.M. van Engelen, Corry M.R. Weemaes, et al.. (2006). Distal spinal muscular atrophy as a major feature in adult-onset ataxia telangiectasia. Neurology. 67(2). 346–349. 26 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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