Issam Ben Sahra

2.2k total citations · 1 hit paper
7 papers, 1.9k citations indexed

About

Issam Ben Sahra is a scholar working on Molecular Biology, Surgery and Oncology. According to data from OpenAlex, Issam Ben Sahra has authored 7 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 4 papers in Surgery and 3 papers in Oncology. Recurrent topics in Issam Ben Sahra's work include Metabolism, Diabetes, and Cancer (5 papers), Pancreatic function and diabetes (4 papers) and Cancer, Hypoxia, and Metabolism (3 papers). Issam Ben Sahra is often cited by papers focused on Metabolism, Diabetes, and Cancer (5 papers), Pancreatic function and diabetes (4 papers) and Cancer, Hypoxia, and Metabolism (3 papers). Issam Ben Sahra collaborates with scholars based in France and United States. Issam Ben Sahra's co-authors include Frédéric Bost, Jean‐François Tanti, Y. Le Marchand‐Brustel, Patrick Auberger, Sophie Giorgetti‐Peraldi, Kathiane Laurent, Pascal Colosetti, Agnès Loubat, Pierre Gounon and Marcel Deckert and has published in prestigious journals such as PLoS ONE, Cancer Research and Oncogene.

In The Last Decade

Issam Ben Sahra

7 papers receiving 1.8k citations

Hit Papers

The antidiabetic drug metformin exerts an antitumoral eff... 2008 2026 2014 2020 2008 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Issam Ben Sahra France 7 1.6k 711 608 358 211 7 1.9k
Yiwei Lin China 22 1.8k 1.2× 692 1.0× 698 1.1× 144 0.4× 38 0.2× 42 2.4k
Myriem Boufraqech United States 23 1.1k 0.7× 366 0.5× 540 0.9× 186 0.5× 450 2.1× 45 1.8k
Toran Sanli Canada 10 769 0.5× 290 0.4× 328 0.5× 132 0.4× 61 0.3× 12 986
Georgios Kallifatidis United States 18 940 0.6× 646 0.9× 364 0.6× 148 0.4× 36 0.2× 26 1.7k
Jaekyoung Son South Korea 20 1.6k 1.0× 643 0.9× 1.3k 2.2× 175 0.5× 27 0.1× 31 2.4k
Maxime Denis Canada 22 931 0.6× 609 0.9× 285 0.5× 1.4k 4.0× 276 1.3× 27 2.0k
Sandra Guaita‐Esteruelas Spain 16 775 0.5× 388 0.5× 345 0.6× 130 0.4× 71 0.3× 25 1.2k
Arancha Cebrián Spain 22 647 0.4× 320 0.5× 249 0.4× 148 0.4× 279 1.3× 50 1.3k
Sunbin Ling China 19 790 0.5× 533 0.7× 402 0.7× 167 0.5× 31 0.1× 50 1.4k
Alessandra Leone Italy 22 934 0.6× 412 0.6× 312 0.5× 96 0.3× 37 0.2× 38 1.4k

Countries citing papers authored by Issam Ben Sahra

Since Specialization
Citations

This map shows the geographic impact of Issam Ben Sahra's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Issam Ben Sahra with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Issam Ben Sahra more than expected).

Fields of papers citing papers by Issam Ben Sahra

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Issam Ben Sahra. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Issam Ben Sahra. The network helps show where Issam Ben Sahra may publish in the future.

Co-authorship network of co-authors of Issam Ben Sahra

This figure shows the co-authorship network connecting the top 25 collaborators of Issam Ben Sahra. A scholar is included among the top collaborators of Issam Ben Sahra based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Issam Ben Sahra. Issam Ben Sahra is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Puissant, Alexandre, Maeva Dufies, Nina Fenouille, et al.. (2012). Imatinib triggers mesenchymal-like conversion of CML cells associated with increased aggressiveness. Journal of Molecular Cell Biology. 4(4). 207–220. 32 indexed citations
2.
Bost, Frédéric, Issam Ben Sahra, Y. Le Marchand‐Brustel, & Jean‐François Tanti. (2011). Metformin and cancer therapy. Current Opinion in Oncology. 24(1). 103–108. 74 indexed citations
3.
Sahra, Issam Ben, Kathiane Laurent, Sandy Giuliano, et al.. (2010). Targeting Cancer Cell Metabolism: The Combination of Metformin and 2-Deoxyglucose Induces p53-Dependent Apoptosis in Prostate Cancer Cells. Cancer Research. 70(6). 2465–2475. 434 indexed citations
4.
Sahra, Issam Ben, Y. Le Marchand‐Brustel, Jean‐François Tanti, & Frédéric Bost. (2010). Metformin in Cancer Therapy: A New Perspective for an Old Antidiabetic Drug?. Molecular Cancer Therapeutics. 9(5). 1092–1099. 414 indexed citations
5.
Sahra, Issam Ben, Jean‐François Tanti, & Frédéric Bost. (2010). The combination of metformin and 2 deoxyglucose inhibits autophagy and induces AMPK-dependent apoptosis in prostate cancer cells. Autophagy. 6(5). 670–671. 96 indexed citations
6.
Robert, Guillaume, Issam Ben Sahra, Alexandre Puissant, et al.. (2009). Acadesine Kills Chronic Myelogenous Leukemia (CML) Cells through PKC-Dependent Induction of Autophagic Cell Death. PLoS ONE. 4(11). e7889–e7889. 74 indexed citations
7.
Sahra, Issam Ben, Kathiane Laurent, Agnès Loubat, et al.. (2008). The antidiabetic drug metformin exerts an antitumoral effect in vitro and in vivo through a decrease of cyclin D1 level. Oncogene. 27(25). 3576–3586. 732 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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