Irina Kholodnyuk

706 total citations
18 papers, 278 citations indexed

About

Irina Kholodnyuk is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Irina Kholodnyuk has authored 18 papers receiving a total of 278 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 6 papers in Oncology and 6 papers in Cancer Research. Recurrent topics in Irina Kholodnyuk's work include Cancer Genomics and Diagnostics (6 papers), Genomic variations and chromosomal abnormalities (4 papers) and Immune Cell Function and Interaction (3 papers). Irina Kholodnyuk is often cited by papers focused on Cancer Genomics and Diagnostics (6 papers), Genomic variations and chromosomal abnormalities (4 papers) and Immune Cell Function and Interaction (3 papers). Irina Kholodnyuk collaborates with scholars based in Sweden, Ukraine and United States. Irina Kholodnyuk's co-authors include George Klein, Stephan Imreh, Eugene R. Zabarovsky, Eric J. Stanbridge, Anna Szeles, Maria Kost‐Alimova, Hajnalka Kiss, Ying Yang, В. И. Кашуба and Elena Kashuba and has published in prestigious journals such as Proceedings of the National Academy of Sciences, International Journal of Molecular Sciences and Virology.

In The Last Decade

Irina Kholodnyuk

18 papers receiving 274 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Irina Kholodnyuk Sweden 10 195 83 64 60 46 18 278
Michael Mullokandov United States 8 368 1.9× 180 2.2× 70 1.1× 72 1.2× 45 1.0× 11 485
Krzysztof Mędrek Poland 8 150 0.8× 127 1.5× 95 1.5× 74 1.2× 46 1.0× 11 273
Svenja Leible Germany 7 366 1.9× 48 0.6× 88 1.4× 64 1.1× 54 1.2× 10 509
Ludmila Fedorova Sweden 11 289 1.5× 110 1.3× 138 2.2× 70 1.2× 17 0.4× 16 415
Gergana Bounova Netherlands 7 307 1.6× 32 0.4× 80 1.3× 95 1.6× 46 1.0× 7 390
Toshinori Ide Japan 7 229 1.2× 72 0.9× 60 0.9× 23 0.4× 16 0.3× 9 504
Jacqueline Dickson United Kingdom 6 358 1.8× 78 0.9× 50 0.8× 53 0.9× 11 0.2× 7 426
Eva Hilgenfeld Germany 10 194 1.0× 92 1.1× 66 1.0× 44 0.7× 34 0.7× 13 318
Teerawit Supakorndej United States 5 318 1.6× 30 0.4× 63 1.0× 25 0.4× 21 0.5× 6 463
Mika Bessho United States 10 344 1.8× 96 1.2× 143 2.2× 77 1.3× 21 0.5× 15 414

Countries citing papers authored by Irina Kholodnyuk

Since Specialization
Citations

This map shows the geographic impact of Irina Kholodnyuk's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Irina Kholodnyuk with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Irina Kholodnyuk more than expected).

Fields of papers citing papers by Irina Kholodnyuk

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Irina Kholodnyuk. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Irina Kholodnyuk. The network helps show where Irina Kholodnyuk may publish in the future.

Co-authorship network of co-authors of Irina Kholodnyuk

This figure shows the co-authorship network connecting the top 25 collaborators of Irina Kholodnyuk. A scholar is included among the top collaborators of Irina Kholodnyuk based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Irina Kholodnyuk. Irina Kholodnyuk is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Ehlin‐Henriksson, Barbro, et al.. (2022). Expression of the Chemokine Receptor CCR1 in Burkitt Lymphoma Cell Lines Is Linked to the CD10-Negative Cell Phenotype and Co-Expression of the EBV Latent Genes EBNA2, LMP1, and LMP2. International Journal of Molecular Sciences. 23(7). 3434–3434. 3 indexed citations
2.
Kholodnyuk, Irina, et al.. (2020). Chemokine Receptors CCR1 and CCR2 on Peripheral Blood Mononuclear Cells of Newly Diagnosed Patients with the CD38-Positive Chronic Lymphocytic Leukemia. Journal of Clinical Medicine. 9(7). 2312–2312. 1 indexed citations
6.
Kholodnyuk, Irina, et al.. (2017). THE TGF-BETA — SMAD PATHWAY IS INACTIVATED IN CRONIC LYMPHOCYTIC LEUKEMIA CELLS. Experimental Oncology. 39(4). 286–290. 12 indexed citations
7.
Yurchenko, Maria, et al.. (2013). Overexpression of MRPS18-2 in Cancer Cell Lines Results in Appearance of Multinucleated Cells. Acta Naturae. 5(1). 85–89. 4 indexed citations
8.
Kost‐Alimova, Maria, Ludmila Fedorova, Ying Yang, et al.. (2007). Mandatory chromosomal segment balance in aneuploid tumor cells. BMC Cancer. 7(1). 21–21. 6 indexed citations
9.
Yang, Ying, Jingfeng Li, Anna Szeles, et al.. (2003). Consistent downregulation of human lactoferrin gene, in the common eliminated region 1 on 3p21.3, following tumor growth in severe combined immunodeficient (SCID) mice. Cancer Letters. 191(2). 155–164. 16 indexed citations
10.
Yang, Ying, Maria Kost‐Alimova, Sigurður Ingvarsson, et al.. (2001). Similar regions of human chromosome 3 are eliminated from or retained in human/human and human/mouse microcell hybrids during tumor growth in severe combined immunodeficient (SCID) mice. Proceedings of the National Academy of Sciences. 98(3). 1136–1141. 26 indexed citations
11.
Yang, Ying, Hajnalka Kiss, Maria Kost‐Alimova, et al.. (1999). A 1-Mb PAC Contig Spanning the Common Eliminated Region 1 (CER1) in Microcell Hybrid-Derived SCID Tumors. Genomics. 62(2). 147–155. 25 indexed citations
12.
Imreh, Stephan, Maria Kost‐Alimova, Irina Kholodnyuk, et al.. (1997). Differential elimination of 3p and retention of 3q segments in human/mouse microcell hybrids during tumor growth. Genes Chromosomes and Cancer. 20(3). 224–233. 26 indexed citations
13.
Szeles, Anna, Ying Yang, Irina Kholodnyuk, et al.. (1997). Human/mouse microcell hybrid based elimination test reduces the putative tumor suppressor region at 3p21.3 to 1.6 cM. Genes Chromosomes and Cancer. 20(4). 329–336. 1 indexed citations
14.
Kholodnyuk, Irina, В. И. Кашуба, Anna Szeles, et al.. (1997). A 3p21.3 region is preferentially eliminated from human chromosome 3/mouse microcell hybrids during tumor growth in SCID mice. Genes Chromosomes and Cancer. 18(3). 200–211. 34 indexed citations
15.
Szeles, Anna, Ying Yang, Irina Kholodnyuk, et al.. (1997). Human/mouse microcell hybrid based elimination test reduces the putative tumor suppressor region at 3p21.3 to 1.6 cM. Genes Chromosomes and Cancer. 20(4). 329–336. 29 indexed citations
16.
Zabarovsky, Eugene R., В. И. Кашуба, Rinat Gizatullin, et al.. (1996). NotI jumping and linking clones as a tool for genome mapping and analysis of chromosome rearrangements in different tumors.. PubMed. 20(1). 1–10. 6 indexed citations
17.
Zabarovsky, Eugene R., Rando Allikmets, Irina Kholodnyuk, et al.. (1994). Construction of Representative NotI Linking Libraries Specific for the Total Human Genome and for Human Chromosome 3. Genomics. 20(2). 312–316. 23 indexed citations
18.
Imreh, Stephan, et al.. (1994). Nonrandom loss of human chromosome 3 fragments from mouse‐human microcell hybrids following progressive growth in SCID mice. Genes Chromosomes and Cancer. 11(4). 237–245. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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