In‐Chul Park

1.1k total citations
30 papers, 926 citations indexed

About

In‐Chul Park is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, In‐Chul Park has authored 30 papers receiving a total of 926 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 10 papers in Oncology and 7 papers in Cancer Research. Recurrent topics in In‐Chul Park's work include Cell death mechanisms and regulation (5 papers), Retinoids in leukemia and cellular processes (5 papers) and Ubiquitin and proteasome pathways (5 papers). In‐Chul Park is often cited by papers focused on Cell death mechanisms and regulation (5 papers), Retinoids in leukemia and cellular processes (5 papers) and Ubiquitin and proteasome pathways (5 papers). In‐Chul Park collaborates with scholars based in South Korea, Ethiopia and Japan. In‐Chul Park's co-authors include Su‐Jae Lee, Seok-Il Hong, Myung Jin Park, Yun‐Sil Lee, Hee Yong Chung, Jin Kyung Lee, Chang Hun Rhee, Young-Hee Kang, Moon-Taek Park and Sungkwan An and has published in prestigious journals such as Journal of Biological Chemistry, Cancer Research and Scientific Reports.

In The Last Decade

In‐Chul Park

30 papers receiving 910 citations

Peers

In‐Chul Park
In-geun Ryoo South Korea
Umesh T. Sankpal United States
Lei Bi China
Chul-Koo Cho South Korea
Pui–Kei Wu United States
Gavin Y. Wang United States
In-geun Ryoo South Korea
In‐Chul Park
Citations per year, relative to In‐Chul Park In‐Chul Park (= 1×) peers In-geun Ryoo

Countries citing papers authored by In‐Chul Park

Since Specialization
Citations

This map shows the geographic impact of In‐Chul Park's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by In‐Chul Park with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites In‐Chul Park more than expected).

Fields of papers citing papers by In‐Chul Park

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by In‐Chul Park. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by In‐Chul Park. The network helps show where In‐Chul Park may publish in the future.

Co-authorship network of co-authors of In‐Chul Park

This figure shows the co-authorship network connecting the top 25 collaborators of In‐Chul Park. A scholar is included among the top collaborators of In‐Chul Park based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with In‐Chul Park. In‐Chul Park is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kim, Se‐Lim, et al.. (2025). Iron overload enhances the susceptibility to cysteine deprivation-induced ferroptosis in non-small cell lung cancer cells. Medical Oncology. 42(6). 201–201. 2 indexed citations
2.
Ye, Sang‐Kyu, et al.. (2021). Correction to: Inhibition of mTORC1 through ATF4-induced REDD1 and Sestrin2 expression by Metformin. BMC Cancer. 21(1). 881–881. 2 indexed citations
3.
Kim, Byung‐Hak, Bo Ruem Yoon, Eun Kyoung Kim, et al.. (2016). Alleviation of collagen-induced arthritis by the benzoxathiole derivative BOT-4-one in mice: Implication of the Th1- and Th17-cell-mediated immune responses. Biochemical Pharmacology. 110-111. 47–57. 18 indexed citations
4.
Yun, Hong Shik, Jeong‐Hwa Baek, J. H. Yim, et al.. (2016). Radiotherapy diagnostic biomarkers in radioresistant human H460 lung cancer stem-like cells. Cancer Biology & Therapy. 17(2). 208–218. 32 indexed citations
5.
Park, Jin‐Ah, et al.. (2016). Inhibition of JNK-mediated autophagy enhances NSCLC cell sensitivity to mTORC1/2 inhibitors. Scientific Reports. 6(1). 28945–28945. 19 indexed citations
6.
Choi, Yunseon, Sang Taek Oh, Min Ji Ko, et al.. (2016). Targeting ODC1 inhibits tumor growth through reduction of lipid metabolism in human hepatocellular carcinoma. Biochemical and Biophysical Research Communications. 478(4). 1674–1681. 29 indexed citations
7.
Yim, J. H., Hong Shik Yun, Su‐Jae Lee, et al.. (2015). Radiosensitizing effect of PSMC5, a 19S proteasome ATPase, in H460 lung cancer cells. Biochemical and Biophysical Research Communications. 469(1). 94–100. 11 indexed citations
8.
Park, Jin‐Ah, Jin Hee Kim, Bora Kim, et al.. (2015). Combined effects of EGFR tyrosine kinase inhibitors and vATPase inhibitors in NSCLC cells. Toxicology and Applied Pharmacology. 287(1). 17–25. 8 indexed citations
9.
Woo, Sang Hyeok, Sang Taek Oh, Taeboo Choe, et al.. (2015). Melatonin enhances arsenic trioxide-induced cell death via sustained upregulation of Redd1 expression in breast cancer cells. Molecular and Cellular Endocrinology. 422. 64–73. 57 indexed citations
10.
Park, Jin‐Ah, Jin Hee Kim, Bora Kim, et al.. (2015). Heme Oxygenase-1 Determines the Differential Response of Breast Cancer and Normal Cells to Piperlongumine. Molecules and Cells. 38(4). 327–335. 59 indexed citations
11.
Lee, Yun-Han, Jin‐Ah Park, Jin Hee Kim, et al.. (2014). Blockage of Stat3 enhances the sensitivity of NSCLC cells to PI3K/mTOR inhibition. Biochemical and Biophysical Research Communications. 444(4). 502–508. 23 indexed citations
12.
Yoon, Changhwan, Min-Jung Kim, Rae-Kwon Kim, et al.. (2012). PTTG1 Oncogene Promotes Tumor Malignancy via Epithelial to Mesenchymal Transition and Expansion of Cancer Stem Cell Population. Journal of Biological Chemistry. 287(23). 19516–19527. 81 indexed citations
13.
Suh, Kyung‐Suk, Seon Rang Woo, In‐Chul Park, et al.. (2011). Integrated Analysis of Prognostic Gene Expression Profiles from Hepatitis B Virus-Positive Hepatocellular Carcinoma and Adjacent Liver Tissue. Annals of Surgical Oncology. 19(S3). 328–338. 9 indexed citations
14.
Woo, Seon Rang, Jeong-Eun Park, Jaemin Jeong, et al.. (2011). Cells with dysfunctional telomeres are susceptible to reactive oxygen species hydrogen peroxide via generation of multichromosomal fusions and chromosomal fragments bearing telomeres. Biochemical and Biophysical Research Communications. 417(1). 204–210. 4 indexed citations
16.
Park, In‐Chul, et al.. (2008). Ultrasonographic evaluation of renal dimension and resistive index in clinically healthy Korean domestic short-hair cats. Journal of Veterinary Science. 9(4). 415–415. 28 indexed citations
17.
Woo, Sang Hyeok, Myung‐Jin Park, Sungkwan An, et al.. (2005). Diarsenic and tetraarsenic oxide inhibit cell cycle progression and bFGF‐ and VEGF‐induced proliferation of human endothelial cells. Journal of Cellular Biochemistry. 95(1). 120–130. 29 indexed citations
18.
Park, In‐Chul, Myung Jin Park, Seok-Il Hong, et al.. (2002). Enhancement of radiation response in human cervical cancer cells in vitro and in vivo by arsenic trioxide (As2O3). FEBS Letters. 519(1-3). 195–200. 55 indexed citations
19.
Park, In‐Chul. (2000). Mitomycin C induces apoptosis in a caspases-dependent and Fas/CD95-independent manner in human gastric adenocarcinoma cells. Cancer Letters. 158(2). 125–132. 29 indexed citations
20.
Park, In‐Chul, et al.. (1998). Increased susceptibility of the c-Myc overexpressing cell line, SNU-16, to TNF-alpha. Cancer Letters. 125(1-2). 17–23. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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