I B Weinstein

5.4k total citations
83 papers, 4.4k citations indexed

About

I B Weinstein is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, I B Weinstein has authored 83 papers receiving a total of 4.4k indexed citations (citations by other indexed papers that have themselves been cited), including 51 papers in Molecular Biology, 19 papers in Oncology and 14 papers in Cancer Research. Recurrent topics in I B Weinstein's work include Cancer-related Molecular Pathways (10 papers), Protein Kinase Regulation and GTPase Signaling (9 papers) and Plant biochemistry and biosynthesis (7 papers). I B Weinstein is often cited by papers focused on Cancer-related Molecular Pathways (10 papers), Protein Kinase Regulation and GTPase Signaling (9 papers) and Plant biochemistry and biosynthesis (7 papers). I B Weinstein collaborates with scholars based in United States, Myanmar and United Kingdom. I B Weinstein's co-authors include Paul B. Fisher, Lih‐Syng Lee, Hui Liu, Danhua Xiao, Masumi Suzui, Andrew K. Joe, Hiroshi Yamasaki, Wei Jiang, Paul M. Choi and Kam-Meng Tchou-Wong and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Circulation.

In The Last Decade

I B Weinstein

82 papers receiving 4.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
I B Weinstein United States	37	2.7k	1.2k	752	520	414	83	4.4k
Paul Amstad Switzerland	33	2.5k 0.9×	786 0.7×	852 1.1×	322 0.6×	309 0.7×	55	4.6k
Barbara S. Beckman United States	38	2.7k 1.0×	866 0.7×	825 1.1×	459 0.9×	279 0.7×	116	4.6k
John S. Bertram United States	36	3.5k 1.3×	690 0.6×	1.1k 1.4×	624 1.2×	205 0.5×	84	6.1k
Donald L. Hill United States	38	3.0k 1.1×	821 0.7×	554 0.7×	590 1.1×	270 0.7×	167	4.8k
Jill C. Pelling United States	39	2.9k 1.1×	931 0.8×	800 1.1×	721 1.4×	629 1.5×	80	4.6k
Mauro Piantelli Italy	44	2.7k 1.0×	1.6k 1.4×	767 1.0×	577 1.1×	650 1.6×	180	6.3k
G. Tim Bowden United States	33	2.3k 0.9×	828 0.7×	930 1.2×	321 0.6×	225 0.5×	91	3.9k
William T. Melvin United Kingdom	39	2.2k 0.8×	1.7k 1.5×	1.5k 2.0×	551 1.1×	234 0.6×	94	5.2k
James H. Wyche United States	27	2.3k 0.8×	817 0.7×	373 0.5×	256 0.5×	362 0.9×	83	3.2k
Jun‐ichi Sawada Japan	44	3.4k 1.3×	1.9k 1.6×	401 0.5×	589 1.1×	240 0.6×	205	6.8k

Countries citing papers authored by I B Weinstein

Since Specialization

Citations

This map shows the geographic impact of I B Weinstein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by I B Weinstein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites I B Weinstein more than expected).

Fields of papers citing papers by I B Weinstein

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by I B Weinstein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by I B Weinstein. The network helps show where I B Weinstein may publish in the future.

Co-authorship network of co-authors of I B Weinstein

This figure shows the co-authorship network connecting the top 25 collaborators of I B Weinstein. A scholar is included among the top collaborators of I B Weinstein based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with I B Weinstein. I B Weinstein is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Yoon, Angela J., Jing Shen, Regina M. Santella, et al.. (2007). Activated Checkpoint Kinase 2 Expression and Risk for Oral Squamous Cell Carcinoma. Cancer Epidemiology Biomarkers & Prevention. 16(12). 2768–2772. 12 indexed citations

Shimizu, Masahito, et al.. (2005). EGCG inhibits activation of HER3 and expression of cyclooxygenase-2 in human colon cancer cells.. PubMed. 5(1). 69–78. 83 indexed citations

Slosberg, Eric D., Margaret C. Eggo, Chi Kong Ching, et al.. (2000). Overexpression of protein kinase C-β1 isoenzyme suppresses indomethacin-induced apoptosis in gastric epithelial cells. Gastroenterology. 118(3). 507–514. 46 indexed citations

Yao, Yao, Yuichiro� Doki, Wei Jiang, et al.. (2000). Cloning and Characterization of DIP1, a Novel Protein That Is Related to the Id Family of Proteins. Experimental Cell Research. 257(1). 22–32. 17 indexed citations

Weinstein, I B. (2000). Disorders in cell circuitry during multistage carcinogenesis: the role of homeostasis. Carcinogenesis. 21(5). 857–864. 214 indexed citations

Slosberg, Eric D., Michael G. Klein, Yao Yao, et al.. (1999). The α isoform of protein kinase C mediates phorbol ester-induced growth inhibition and p21cip1 induction in HC11 mammary epithelial cells. Oncogene. 18(48). 6658–6666. 29 indexed citations

Goluboff, Erik T., Ahmad Shabsigh, I B Weinstein, et al.. (1999). Exisulind (sulindac sulfone) suppresses growth of human prostate cancer in a nude mouse xenograft model by increasing apoptosis. Urology. 53(2). 440–445. 114 indexed citations

Luo, Jianhua, et al.. (1995). The phorbol ester TPA markedly enhances the binding of calcium to the regulatory domain of protein kinase C β1 in the presence of phosphatidylserine. Carcinogenesis. 16(4). 897–905. 4 indexed citations

Han, Edward K., Alessandro Sgambato, Wei Jiang, et al.. (1995). Stable overexpression of cyclin D1 in a human mammary epithelial cell line prolongs the S-phase and inhibits growth.. PubMed. 10(5). 953–61. 81 indexed citations

10.

Hei, Tom K., et al.. (1994). Effects of increased expression of protein kinase C on radiation-induced cell transformation. Carcinogenesis. 15(2). 365–370. 9 indexed citations

11.

Nomoto, Koji, Masami Morotomi, Masami Miyake, et al.. (1994). The effects of bile acids on phospholipase c activity in extracts of normal human colon mucosa and primary colon tumors. Molecular Carcinogenesis. 9(2). 87–94. 17 indexed citations

12.

Hoshina, Sadayori, Marius Ueffing, & I B Weinstein. (1990). Growth factor‐induced DNA synthesis in cells that overproduce protein kinase C. Journal of Cellular Physiology. 145(2). 262–267. 23 indexed citations

13.

Hsieh, Ling‐Ling & I B Weinstein. (1990). Factors influencing the expression of endogenous retrovirus‐like sequences in rat 6 cells. Molecular Carcinogenesis. 3(6). 344–349. 10 indexed citations

14.

Weinstein, I B, G A Cook, & Murray Heimberg. (1986). Regulation by oestrogen of carnitine palmitoyltransferase in hepatic mitochondria. Biochemical Journal. 237(2). 593–596. 30 indexed citations

15.

Liskamp, Rob M. J., et al.. (1985). Cellular uptake and localization of fluorescent derivatives of phorbol ester tumor promoters. Biochemical and Biophysical Research Communications. 131(2). 920–927. 32 indexed citations

16.

Neugut, Alfred I., et al.. (1978). Effects of asbestos on epithelioid cell lines. Environmental Research. 17(2). 256–265. 20 indexed citations

17.

Fisher, Paul B., I B Weinstein, David P. Eisenberg, & H S Ginsberg. (1978). Interactions between adenovirus, a tumor promoter, and chemical carcinogens in transformation of rat embryo cell cultures.. Proceedings of the National Academy of Sciences. 75(5). 2311–2314. 81 indexed citations

18.

Weinstein, I B, et al.. (1975). Growth and structural properties of epithelial cell cultures established from normal rat liver and chemically induced hepatomas.. PubMed. 35(1). 253–63. 89 indexed citations

19.

Weinstein, I B & E F Osserman. (1964). AMINO ACID INCORPORATION INTO PROTEIN BY A CELL FREE SYSTEM FROM PLASMA CELL TUMORS.. PubMed. 20. 932–6. 3 indexed citations

20.

Kabakow, B, et al.. (1959). A clinical and metabolic study of metastatic carcinoid. The American Journal of Medicine. 26(4). 636–645. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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