Hung‐Ming Lam

3.8k total citations · 1 hit paper
57 papers, 2.5k citations indexed

About

Hung‐Ming Lam is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Oncology. According to data from OpenAlex, Hung‐Ming Lam has authored 57 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Molecular Biology, 24 papers in Pulmonary and Respiratory Medicine and 15 papers in Oncology. Recurrent topics in Hung‐Ming Lam's work include Prostate Cancer Treatment and Research (22 papers), Bladder and Urothelial Cancer Treatments (10 papers) and Estrogen and related hormone effects (8 papers). Hung‐Ming Lam is often cited by papers focused on Prostate Cancer Treatment and Research (22 papers), Bladder and Urothelial Cancer Treatments (10 papers) and Estrogen and related hormone effects (8 papers). Hung‐Ming Lam collaborates with scholars based in United States, Macao and Canada. Hung‐Ming Lam's co-authors include Colm Morrissey, Robert L. Vessella, Venu Menon, Alfonso T. Perez, Faı̈ez Zannad, George L. Bakris, P. Fleck, William B. White, Eva Corey and Craig Wilson and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Circulation.

In The Last Decade

Hung‐Ming Lam

55 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Heart failure and mortality outcomes in patients with type 2 diabetes taking alogliptin versus placebo in EXAMINE: a multicentre, randomised, double-blind trial

2015 551 citations Hung‐Ming Lam et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Hung‐Ming Lam United States	24	1.1k	770	766	677	494	57	2.5k
Anne T. Reutens Australia	26	1.5k 1.4×	574 0.7×	309 0.4×	688 1.0×	222 0.4×	38	2.9k
D M Peehl United States	23	1.3k 1.2×	471 0.6×	826 1.1×	740 1.1×	495 1.0×	33	3.0k
Shiu‐Ru Lin Taiwan	32	1.1k 1.0×	1.1k 1.5×	485 0.6×	209 0.3×	818 1.7×	105	2.7k
Roberta Malaguarnera Italy	32	1.6k 1.5×	792 1.0×	164 0.2×	1.2k 1.8×	686 1.4×	72	3.2k
Alison F. Munro United Kingdom	26	850 0.8×	855 1.1×	285 0.4×	183 0.3×	557 1.1×	54	2.2k
Soo Ok Lee United States	36	1.2k 1.1×	969 1.3×	935 1.2×	281 0.4×	692 1.4×	62	2.8k
David Mills United States	17	1.6k 1.5×	1.6k 2.1×	1.2k 1.6×	1.6k 2.4×	889 1.8×	27	4.3k
Andelko Hrzenjak Austria	32	1.4k 1.3×	374 0.5×	326 0.4×	205 0.3×	604 1.2×	63	2.5k
Mitsuteru Kitade Japan	32	1.2k 1.1×	522 0.7×	192 0.3×	464 0.7×	527 1.1×	88	3.2k
Ken‐Ichiro Yoshida Japan	28	924 0.8×	593 0.8×	769 1.0×	273 0.4×	229 0.5×	118	2.4k

Countries citing papers authored by Hung‐Ming Lam

Since Specialization

Citations

This map shows the geographic impact of Hung‐Ming Lam's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hung‐Ming Lam with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hung‐Ming Lam more than expected).

Fields of papers citing papers by Hung‐Ming Lam

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hung‐Ming Lam. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hung‐Ming Lam. The network helps show where Hung‐Ming Lam may publish in the future.

Co-authorship network of co-authors of Hung‐Ming Lam

This figure shows the co-authorship network connecting the top 25 collaborators of Hung‐Ming Lam. A scholar is included among the top collaborators of Hung‐Ming Lam based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hung‐Ming Lam. Hung‐Ming Lam is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Li, Shan, Alicia Y. W. Wong, Vipul Bhatia, et al.. (2024). A combinatorial genetic strategy for exploring complex genotype–phenotype associations in cancer. Nature Genetics. 56(3). 371–376. 6 indexed citations

Pornour, Majid, Heeyoung Jeon, Hyunju Ryu, et al.. (2024). USP11 promotes prostate cancer progression by up-regulating AR and c-Myc activity. Proceedings of the National Academy of Sciences. 121(31). e2403331121–e2403331121. 8 indexed citations

Han, Wanting, Dong Han, Zifeng Wang, et al.. (2022). Exploiting the tumor-suppressive activity of the androgen receptor by CDK4/6 inhibition in castration-resistant prostate cancer. Molecular Therapy. 30(4). 1628–1644. 25 indexed citations

Kim, Yeon, Andrew C. Hsieh, & Hung‐Ming Lam. (2022). Bladder Cancer Patient-derived Organoids and Avatars for Personalized Cancer Discovery. European Urology Focus. 8(3). 657–659. 7 indexed citations

Labrecque, Mark P., Lisha G. Brown, Ilsa M. Coleman, et al.. (2021). RNA Splicing Factors SRRM3 and SRRM4 Distinguish Molecular Phenotypes of Castration-Resistant Neuroendocrine Prostate Cancer. Cancer Research. 81(18). 4736–4750. 23 indexed citations

Chatterjee, Payel, Michael T. Schweizer, Jared M. Lucas, et al.. (2019). Supraphysiological androgens suppress prostate cancer growth through androgen receptor–mediated DNA damage. Journal of Clinical Investigation. 129(10). 4245–4260. 67 indexed citations

Lam, Hung‐Ming, Holly M. Nguyen, Mark P. Labrecque, et al.. (2019). Durable Response of Enzalutamide-resistant Prostate Cancer to Supraphysiological Testosterone Is Associated with a Multifaceted Growth Suppression and Impaired DNA Damage Response Transcriptomic Program in Patient-derived Xenografts. European Urology. 77(2). 144–155. 34 indexed citations

Zhu, Sen, Dongyu Zhao, Yan Lin, et al.. (2018). BMI1 regulates androgen receptor in prostate cancer independently of the polycomb repressive complex 1. Nature Communications. 9(1). 500–500. 63 indexed citations

Lam, Hung‐Ming, Ryan P. McMullin, Holly M. Nguyen, et al.. (2016). Characterization of an Abiraterone Ultraresponsive Phenotype in Castration-Resistant Prostate Cancer Patient-Derived Xenografts. Clinical Cancer Research. 23(9). 2301–2312. 17 indexed citations

10.

Zhang, Xiaotun, Ilsa M. Coleman, Lisha G. Brown, et al.. (2015). SRRM4 Expression and the Loss of REST Activity May Promote the Emergence of the Neuroendocrine Phenotype in Castration-Resistant Prostate Cancer. Clinical Cancer Research. 21(20). 4698–4708. 129 indexed citations

11.

Erdmann, E, Simon Harding, Hung‐Ming Lam, & Alfonso Pérez. (2015). Ten‐year observational follow‐up of PROactive : a randomized cardiovascular outcomes trial evaluating pioglitazone in type 2 diabetes. Diabetes Obesity and Metabolism. 18(3). 266–273. 67 indexed citations

12.

Zhang, Xiaotun, Ilsa M. Coleman, Nolan G. Ericson, et al.. (2015). Epithelial mesenchymal-like transition occurs in a subset of cells in castration resistant prostate cancer bone metastases. Clinical & Experimental Metastasis. 33(3). 239–248. 27 indexed citations

13.

Sosa, María Soledad, Falguni Parikh, Alexandre Gaspar‐Maia, et al.. (2015). NR2F1 controls tumour cell dormancy via SOX9- and RARβ-driven quiescence programmes. Nature Communications. 6(1). 6170–6170. 253 indexed citations

14.

Lam, Hung‐Ming, Bin Ouyang, Jing Chen, et al.. (2014). Targeting GPR30 with G-1: a new therapeutic target for castration-resistant prostate cancer. Endocrine Related Cancer. 21(6). 903–914. 44 indexed citations

15.

Ouyang, Bin, C. Stuart Baxter, Hung‐Ming Lam, et al.. (2012). Hypomethylation of Dual Specificity Phosphatase 22 Promoter Correlates With Duration of Service in Firefighters and Is Inducible by Low-Dose Benzo[a]Pyrene. Journal of Occupational and Environmental Medicine. 54(7). 774–780. 34 indexed citations

16.

Lam, Hung‐Ming, C. V. Suresh Babu, Jiang Wang, et al.. (2012). Phosphorylation of human estrogen receptor-beta at serine 105 inhibits breast cancer cell migration and invasion. Molecular and Cellular Endocrinology. 358(1). 27–35. 29 indexed citations

17.

Leung, Yuet‐Kin, et al.. (2012). Estrogen receptor-beta and breast cancer: Translating biology into clinical practice. Steroids. 77(7). 727–737. 51 indexed citations

18.

Leung, Yuet‐Kin, Hung‐Ming Lam, Shulin Wu, et al.. (2010). Estrogen receptor β2 and β5 are associated with poor prognosis in prostate cancer, and promote cancer cell migration and invasion. Endocrine Related Cancer. 17(3). 675–689. 120 indexed citations

19.

Schwartzberg, Lee S., Lorrin Yee, B. Mirtsching, et al.. (2007). A phase II, randomized, open-label study to assess the efficacy of extended-dose schedule administration of darbepoetin alfa in cancer patients with chemotherapy-induced anemia. 5. 22–23. 1 indexed citations

20.

Jiang, Ren‐Wang, Kit-Man Lau, Hung‐Ming Lam, et al.. (2004). A comparative study on aqueous root extracts of Pueraria thomsonii and Pueraria lobata by antioxidant assay and HPLC fingerprint analysis. Journal of Ethnopharmacology. 96(1-2). 133–138. 107 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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