Huiling Li

1.3k total citations
39 papers, 940 citations indexed

About

Huiling Li is a scholar working on Molecular Biology, Nephrology and Cancer Research. According to data from OpenAlex, Huiling Li has authored 39 papers receiving a total of 940 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 9 papers in Nephrology and 7 papers in Cancer Research. Recurrent topics in Huiling Li's work include Chronic Kidney Disease and Diabetes (6 papers), Cancer-related molecular mechanisms research (6 papers) and Circular RNAs in diseases (6 papers). Huiling Li is often cited by papers focused on Chronic Kidney Disease and Diabetes (6 papers), Cancer-related molecular mechanisms research (6 papers) and Circular RNAs in diseases (6 papers). Huiling Li collaborates with scholars based in China, United States and Russia. Huiling Li's co-authors include Dongshan Zhang, Xudong Xiang, Zheng Dong, Jian Pan, Xiaozhou Li, Junxiang Chen, Yijian Li, Jinwen Chen, Bohao Liu and Xuejin Zhu and has published in prestigious journals such as Nature Communications, SHILAP Revista de lepidopterología and Scientific Reports.

In The Last Decade

Huiling Li

35 papers receiving 936 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Huiling Li China 19 481 349 208 121 118 39 940
Aaron McClelland Australia 10 524 1.1× 375 1.1× 149 0.7× 73 0.6× 64 0.5× 13 806
Congwei Luo China 16 529 1.1× 211 0.6× 272 1.3× 126 1.0× 69 0.6× 22 1.0k
Yan Dai China 15 431 0.9× 128 0.4× 295 1.4× 126 1.0× 125 1.1× 27 884
Zhengzhe Li China 19 471 1.0× 110 0.3× 359 1.7× 155 1.3× 160 1.4× 33 1.1k
Yonghong Shi China 17 372 0.8× 226 0.6× 117 0.6× 69 0.6× 73 0.6× 32 733
Motoi Kobayashi Japan 15 525 1.1× 213 0.6× 188 0.9× 129 1.1× 115 1.0× 21 1.1k
Elisa Conde Spain 17 413 0.9× 289 0.8× 133 0.6× 78 0.6× 94 0.8× 26 841
Shan Huang China 18 499 1.0× 400 1.1× 41 0.2× 75 0.6× 67 0.6× 61 926
Ying Xiao China 18 394 0.8× 150 0.4× 178 0.9× 64 0.5× 102 0.9× 41 746
Gexin Zhao China 16 477 1.0× 170 0.5× 199 1.0× 171 1.4× 96 0.8× 34 1.1k

Countries citing papers authored by Huiling Li

Since Specialization
Citations

This map shows the geographic impact of Huiling Li's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Huiling Li with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Huiling Li more than expected).

Fields of papers citing papers by Huiling Li

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Huiling Li. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Huiling Li. The network helps show where Huiling Li may publish in the future.

Co-authorship network of co-authors of Huiling Li

This figure shows the co-authorship network connecting the top 25 collaborators of Huiling Li. A scholar is included among the top collaborators of Huiling Li based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Huiling Li. Huiling Li is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Li, Xiaozhou, Yang Xia, Yong Guo, et al.. (2025). PRDM16 acts as a homeostasis regulation factor to suppress the transition of AKI to CKD via upregulation of eukaryotic initiation factor 6. Cellular and Molecular Life Sciences. 82(1). 252–252.
2.
Wu, Dengke, et al.. (2024). Proximal tubular MBD2 promotes autophagy to drive the progression of AKI caused by vancomycin via regulation of miR ‐597‐5p/ S1PR1 axis. The FASEB Journal. 38(7). e23562–e23562. 2 indexed citations
3.
Li, Xiaozhou, Fang Xu, Pan Zhang, et al.. (2024). Overexpression of PRDM16 attenuates acute kidney injury progression: genetic and pharmacological approaches. SHILAP Revista de lepidopterología. 5(10). e737–e737. 3 indexed citations
4.
Feng, Yuqing, Bohao Liu, Jinwen Chen, Huiling Li, & Dongshan Zhang. (2023). The Circ_35953 induced by the NF‐κB mediated the septic AKI via targeting miR‐7219‐5p/HOOK3 and IGFBP7 axis. Journal of Cellular and Molecular Medicine. 27(9). 1261–1276. 9 indexed citations
5.
Xu, Fang, Hongwei� Jiang, Xiaozhou Li, et al.. (2023). Discovery of PRDM16‐Mediated TRPA1 Induction as the Mechanism for Low Tubulo‐Interstitial Fibrosis in Diabetic Kidney Disease. Advanced Science. 11(7). e2306704–e2306704. 17 indexed citations
6.
7.
Li, Xiaozhou, Jian Pan, Huiling Li, et al.. (2022). DsbA-L interacts with VDAC1 in mitochondrion-mediated tubular cell apoptosis and contributes to the progression of acute kidney disease. EBioMedicine. 76. 103859–103859. 26 indexed citations
8.
Ai, Kai, Xiaozhou Li, Pan Zhang, et al.. (2022). Genetic or siRNA inhibition of MBD2 attenuates the UUO- and I/R-induced renal fibrosis via downregulation of EGR1. Molecular Therapy — Nucleic Acids. 28. 77–86. 27 indexed citations
9.
Pan, Jian, Yuxin Xie, Huiling Li, et al.. (2022). mmu-lncRNA 121686/hsa-lncRNA 520657 induced by METTL3 drive the progression of AKI by targeting miR-328-5p/HtrA3 signaling axis. Molecular Therapy. 30(12). 3694–3713. 31 indexed citations
10.
Ai, Kai, Jian Pan, Pan Zhang, et al.. (2022). Methyl-CpG-binding domain protein 2 contributes to renal fibrosis through promoting polarized M1 macrophages. Cell Death and Disease. 13(2). 125–125. 25 indexed citations
11.
Zhao, Tantai, et al.. (2021). Altered oxylipin levels in human vitreous indicate imbalance in pro-/anti-inflammatory homeostasis in proliferative diabetic retinopathy. Experimental Eye Research. 214. 108799–108799. 16 indexed citations
12.
Li, Xiaozhou, Jian Pan, Huiling Li, et al.. (2020). DsbA-L mediated renal tubulointerstitial fibrosis in UUO mice. Nature Communications. 11(1). 4467–4467. 84 indexed citations
13.
Xie, Yuxin, Bohao Liu, Jian Pan, et al.. (2020). MBD2 Mediates Septic AKI through Activation of PKCη/p38MAPK and the ERK1/2 Axis. Molecular Therapy — Nucleic Acids. 23. 76–88. 32 indexed citations
14.
Zhang, Pan, Lei Yi, Xiaozhou Li, et al.. (2020). The Biomarker TCONS_00016233 Drives Septic AKI by Targeting the miR-22-3p/AIFM1 Signaling Axis. Molecular Therapy — Nucleic Acids. 19. 1027–1042. 61 indexed citations
15.
Zhang, Ran, et al.. (2020). Mbd2 Mediates Retinal Cell Apoptosis by Targeting the lncRNA Mbd2-AL1/miR-188-3p/Traf3 Axis in Ischemia/Reperfusion Injury. Molecular Therapy — Nucleic Acids. 19. 1250–1265. 26 indexed citations
16.
Wang, Juan, Dengke Wu, Yu Zhou, et al.. (2019). lncRNA NR_038323 Suppresses Renal Fibrosis in Diabetic Nephropathy by Targeting the miR-324-3p/DUSP1 Axis. Molecular Therapy — Nucleic Acids. 17. 741–753. 88 indexed citations
17.
Wang, Juan, et al.. (2017). MBD2 upregulates miR-301a-5p to induce kidney cell apoptosis during vancomycin-induced AKI. Cell Death and Disease. 8(10). e3120–e3120. 60 indexed citations
18.
Yang, Ruhao, Xuan Xu, Huiling Li, et al.. (2017). p53 induces miR199a-3p to suppress SOCS7 for STAT3 activation and renal fibrosis in UUO. Scientific Reports. 7(1). 43409–43409. 82 indexed citations
19.
Wang, Xiaofei, et al.. (2016). Genetic and immunohistochemical analysis of HSPA5 in mouse and human retinas.. PubMed Central. 22. 1318–1331. 2 indexed citations
20.
Li, Huiling, et al.. (2014). Down-regulation of GRP78 enhances apoptosis via CHOP pathway in retinal ischemia-reperfusion injury. Neuroscience Letters. 575. 68–73. 39 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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