Huayang Pan

671 total citations
18 papers, 489 citations indexed

About

Huayang Pan is a scholar working on Molecular Biology, Surgery and Cancer Research. According to data from OpenAlex, Huayang Pan has authored 18 papers receiving a total of 489 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 6 papers in Surgery and 5 papers in Cancer Research. Recurrent topics in Huayang Pan's work include Cancer, Lipids, and Metabolism (4 papers), RNA modifications and cancer (4 papers) and Ferroptosis and cancer prognosis (3 papers). Huayang Pan is often cited by papers focused on Cancer, Lipids, and Metabolism (4 papers), RNA modifications and cancer (4 papers) and Ferroptosis and cancer prognosis (3 papers). Huayang Pan collaborates with scholars based in China and New Zealand. Huayang Pan's co-authors include Yong Ma, Hongchi Jiang, Dawei Wang, Shangha Pan, Lianxin Liu, Jizhou Wang, Dongsheng Xu, Baolei Zhao, Yaohua Wu and Xueying Sun and has published in prestigious journals such as Cancer Letters, Autophagy and Molecular Nutrition & Food Research.

In The Last Decade

Huayang Pan

18 papers receiving 480 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Huayang Pan China 11 203 92 74 65 59 18 489
Chinenye Iwuji United Kingdom 8 289 1.4× 115 1.3× 96 1.3× 40 0.6× 42 0.7× 17 654
Ke Pan China 18 316 1.6× 80 0.9× 91 1.2× 130 2.0× 99 1.7× 73 936
Jung Yoon Jang South Korea 15 263 1.3× 87 0.9× 64 0.9× 35 0.5× 25 0.4× 22 571
Zeyao Tang China 17 397 2.0× 45 0.5× 80 1.1× 66 1.0× 34 0.6× 20 773
Xiuming Song China 14 379 1.9× 83 0.9× 98 1.3× 39 0.6× 75 1.3× 15 876
Yaohua Wu China 11 239 1.2× 34 0.4× 123 1.7× 51 0.8× 46 0.8× 17 432
Xianyong Bai China 13 290 1.4× 95 1.0× 72 1.0× 78 1.2× 29 0.5× 20 641
Guiwu Qu China 14 242 1.2× 52 0.6× 47 0.6× 39 0.6× 53 0.9× 27 586
Rong Lin China 16 435 2.1× 106 1.2× 47 0.6× 37 0.6× 39 0.7× 31 783
Karvannan Kanchana China 9 314 1.5× 74 0.8× 43 0.6× 65 1.0× 22 0.4× 22 548

Countries citing papers authored by Huayang Pan

Since Specialization
Citations

This map shows the geographic impact of Huayang Pan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Huayang Pan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Huayang Pan more than expected).

Fields of papers citing papers by Huayang Pan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Huayang Pan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Huayang Pan. The network helps show where Huayang Pan may publish in the future.

Co-authorship network of co-authors of Huayang Pan

This figure shows the co-authorship network connecting the top 25 collaborators of Huayang Pan. A scholar is included among the top collaborators of Huayang Pan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Huayang Pan. Huayang Pan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Liu, Sheng, et al.. (2025). PFKFB3 confers cisplatin resistance in gastric cancer by inhibiting ferroptosis through SLC7A11/xCT dephosphorylation. International Immunopharmacology. 159. 114914–114914. 3 indexed citations
2.
Liu, Zhengyu, Huayang Pan, Yuechen Liu, et al.. (2025). Acetylation-induced degradation of ECHS1 enhances BCAA accumulation and proliferation in KRAS-mutant colorectal cancer. Journal of Experimental & Clinical Cancer Research. 44(1). 164–164. 1 indexed citations
3.
Peng, Yifeng, Kai Yang, Sheng Liu, et al.. (2024). PROTAC based STING degrader attenuates acute colitis by inhibiting macrophage M1 polarization and intestinal epithelial cells pyroptosis mediated by STING-NLRP3 axis. International Immunopharmacology. 141. 112990–112990. 7 indexed citations
4.
Gao, Ran, Jinge Wang, Jingjing Huang, et al.. (2024). FSP1-mediated ferroptosis in cancer: from mechanisms to therapeutic applications. APOPTOSIS. 29(7-8). 1019–1037. 21 indexed citations
5.
Wu, Yue, Fuqi Ma, Huayang Pan, et al.. (2023). Exploration of the mechanism of Qi-Xian decoction in asthmatic mice using metabolomics combined with network pharmacology. Frontiers in Molecular Biosciences. 10. 1263962–1263962. 2 indexed citations
6.
Wu, Jiaming, Shuang Zhu, Peng Wang, et al.. (2022). Regulators of epigenetic change in ferroptosis‑associated cancer (Review). Oncology Reports. 48(6). 12 indexed citations
7.
Han, Lin, Jinge Wang, Tong Wang, et al.. (2021). The LncRNA MIR503HG/miR-224-5p/TUSC3 Signaling Cascade Suppresses Gastric Cancer Development via Modulating ATF6 Branch of Unfolded Protein Response. Frontiers in Oncology. 11. 708501–708501. 18 indexed citations
8.
Huang, Jingjing, Huayang Pan, Jinge Wang, et al.. (2021). Unfolded protein response in colorectal cancer. Cell & Bioscience. 11(1). 26–26. 50 indexed citations
9.
Pan, Huayang, Fukai Liu, Jinge Wang, et al.. (2020). Dihydromethysticin, a natural molecule from Kava, suppresses the growth of colorectal cancer via the NLRC3/PI3K pathway. Molecular Carcinogenesis. 59(6). 575–589. 11 indexed citations
10.
Wang, Dawei, Zhuoxin Cheng, Ming Zhao, et al.. (2019). <p>PTPN9 induces cell apoptosis by mitigating the activation of Stat3 and acts as a tumor suppressor in colorectal cancer</p>. Cancer Management and Research. Volume 11. 1309–1319. 13 indexed citations
11.
Ma, Yong, Jizhou Wang, Huayang Pan, et al.. (2016). Analysis of 300 consecutive cases of pancreatic adenocarcinoma in a single-center in China. Hepatobiliary & pancreatic diseases international. 15(2). 189–197. 3 indexed citations
12.
Wu, Yaohua, Yong Ma, Dawei Wang, et al.. (2014). Sodium orthovanadate inhibits growth of human hepatocellular carcinoma cells in vitro and in an orthotopic model in vivo. Cancer Letters. 351(1). 108–116. 49 indexed citations
13.
Xu, Dongsheng, Yong Ma, Baolei Zhao, et al.. (2014). Thymoquinone induces G2/M arrest, inactivates PI3K/Akt and nuclear factor-κB pathways in human cholangiocarcinomas both in vitro and in vivo. Oncology Reports. 31(5). 2063–2070. 56 indexed citations
15.
Ma, Yong, Dawei Wang, Lianxin Liu, et al.. (2013). Establishment of mice model of partial hepatic ischemia reperfusion injury. Zhōnghuá xiāohuà wàikē zázhì/Zhonghua xiaohua waike zazhi. 12(9). 703–707. 1 indexed citations
16.
Pan, Huayang, Yong Ma, Dawei Wang, et al.. (2013). Effect of IFN-α on KC and LIX expression: Role of STAT1 and its effect on neutrophil recruitment to the spleen after lipopolysaccharide stimulation. Molecular Immunology. 56(1-2). 12–22. 6 indexed citations
17.
Pan, Shangha, Lianxin Liu, Huayang Pan, et al.. (2013). Protective effects of hydroxytyrosol on liver ischemia/reperfusion injury in mice. Molecular Nutrition & Food Research. 57(7). 1218–1227. 50 indexed citations
18.
Wang, Dawei, Yong Ma, Zhengtian Li, et al.. (2012). The role of AKT1 and autophagy in the protective effect of hydrogen sulphide against hepatic ischemia/reperfusion injury in mice. Autophagy. 8(6). 954–962. 91 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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