Huaxun Wu

1.4k total citations
50 papers, 1.1k citations indexed

About

Huaxun Wu is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Huaxun Wu has authored 50 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Immunology, 17 papers in Molecular Biology and 11 papers in Oncology. Recurrent topics in Huaxun Wu's work include Salivary Gland Disorders and Functions (10 papers), Rheumatoid Arthritis Research and Therapies (9 papers) and Immunotherapy and Immune Responses (7 papers). Huaxun Wu is often cited by papers focused on Salivary Gland Disorders and Functions (10 papers), Rheumatoid Arthritis Research and Therapies (9 papers) and Immunotherapy and Immune Responses (7 papers). Huaxun Wu collaborates with scholars based in China, Saint Kitts and Nevis and United States. Huaxun Wu's co-authors include Wei Wei, Yan Chang, Jingyu Chen, Qingtong Wang, Lingling Zhang, Yujing Wu, Pingfan Yuan, Shasha Song, Xiaoyi Jia and Wei Wei and has published in prestigious journals such as The Journal of Immunology, Scientific Reports and Journal of Pharmacology and Experimental Therapeutics.

In The Last Decade

Huaxun Wu

49 papers receiving 1.1k citations

Peers

Huaxun Wu
Huaxun Wu
Citations per year, relative to Huaxun Wu Huaxun Wu (= 1×) peers Yühong Huang

Countries citing papers authored by Huaxun Wu

Since Specialization
Citations

This map shows the geographic impact of Huaxun Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Huaxun Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Huaxun Wu more than expected).

Fields of papers citing papers by Huaxun Wu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Huaxun Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Huaxun Wu. The network helps show where Huaxun Wu may publish in the future.

Co-authorship network of co-authors of Huaxun Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Huaxun Wu. A scholar is included among the top collaborators of Huaxun Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Huaxun Wu. Huaxun Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhou, Tongtong, Hui Liu, Lijun Li, et al.. (2023). MANF inhibits Sjögren's syndrome salivary gland epithelial cell apoptosis and antigen expression of Ro52/SSA through endoplasmic reticulum stress/autophagy pathway. International Immunopharmacology. 122. 110582–110582. 6 indexed citations
2.
Zhang, Jing, Xiao Zhang, Xingjie Shi, et al.. (2023). CXCL9, 10, 11/CXCR3 Axis Contributes to the Progress of Primary Sjogren’s Syndrome by Activating GRK2 to Promote T Lymphocyte Migration. Inflammation. 46(3). 1047–1060. 13 indexed citations
3.
Zhang, Feng, Gang Cheng, Xiaoyang Yuan, et al.. (2022). Attenuation of experimental osteoarthritis with human adipose-derived mesenchymal stem cell therapy: inhibition of the pyroptosis in chondrocytes. Inflammation Research. 72(1). 89–105. 11 indexed citations
4.
Zhao, Zhiwei, et al.. (2020). CD4+ T cells and TGFβ1/MAPK signal pathway involved in the valvular hyperblastosis and fibrosis in patients with rheumatic heart disease. Experimental and Molecular Pathology. 114. 104402–104402. 15 indexed citations
5.
Zhou, Weijie, Dandan Wang, Yu Tai, et al.. (2020). Deficiency of β-arrestin2 exacerbates inflammatory arthritis by facilitating plasma cell formation. Acta Pharmacologica Sinica. 42(5). 755–766. 19 indexed citations
6.
Liu, Qi, Heinrich Körner, Huaxun Wu, & Wei Wei. (2019). Endoplasmic reticulum stress in autoimmune diseases. Immunobiology. 225(2). 151881–151881. 21 indexed citations
7.
Jia, Xiaoyi, Yan Chang, Xiaojing Sun, et al.. (2019). Regulatory effects of paeoniflorin-6′-O-benzene sulfonate (CP-25) on dendritic cells maturation and activation via PGE2-EP4 signaling in adjuvant-induced arthritic rats. Inflammopharmacology. 27(5). 997–1010. 16 indexed citations
8.
Yan, Shangxue, et al.. (2018). Angiotensin II and tumor necrosis factor-α stimulate the growth, migration and invasion of BEL-7402 cells via down-regulation of GRK2 expression. Digestive and Liver Disease. 51(2). 263–274. 5 indexed citations
9.
Wu, Huaxun, Jingyu Chen, Chun Wang, et al.. (2018). β2-adrenoceptor signaling reduction is involved in the inflammatory response of fibroblast-like synoviocytes from adjuvant-induced arthritic rats. Inflammopharmacology. 27(2). 271–279. 10 indexed citations
10.
Chen, Jingyu, Ying Wang, Huaxun Wu, et al.. (2018). A Modified Compound From Paeoniflorin, CP-25, Suppressed Immune Responses and Synovium Inflammation in Collagen-Induced Arthritis Mice. Frontiers in Pharmacology. 9. 563–563. 43 indexed citations
11.
Chen, Xiaoyun, Huaxun Wu, & Wei Wei. (2018). Advances in the diagnosis and treatment of Sjogren’s syndrome. Clinical Rheumatology. 37(7). 1743–1749. 36 indexed citations
12.
Yan, Shangxue, Huaxun Wu, Jingyu Chen, et al.. (2017). The influence of TNF-α and Ang II on the proliferation, migration and invasion of HepG2 cells by regulating the expression of GRK2. Cancer Chemotherapy and Pharmacology. 79(4). 747–758. 17 indexed citations
13.
Wu, Huaxun, Shasha Song, Pingfan Yuan, et al.. (2016). β2-adrenoceptor signaling reduction in dendritic cells is involved in the inflammatory response in adjuvant-induced arthritic rats. Scientific Reports. 6(1). 24548–24548. 45 indexed citations
14.
Chang, Yan, Xiaoyi Jia, Wei Fang, et al.. (2016). CP-25, a novel compound, protects against autoimmune arthritis by modulating immune mediators of inflammation and bone damage. Scientific Reports. 6(1). 26239–26239. 61 indexed citations
15.
Chen, Jingyu, Qingtong Wang, Huaxun Wu, et al.. (2016). The ginsenoside metabolite compound K exerts its anti-inflammatory activity by downregulating memory B cell in adjuvant-induced arthritis. Pharmaceutical Biology. 54(7). 1280–1288. 28 indexed citations
16.
Chen, Jingyu, Huaxun Wu, Qingtong Wang, et al.. (2015). Ginsenoside Metabolite Compound K Suppresses T-Cell Priming via Modulation of Dendritic Cell Trafficking and Costimulatory Signals, Resulting in Alleviation of Collagen-Induced Arthritis. Journal of Pharmacology and Experimental Therapeutics. 353(1). 71–79. 40 indexed citations
17.
Sheng, Kangliang, Jingyu Chen, Yujing Wu, et al.. (2015). Regulation of PGE2 signaling pathways and TNF-alpha signaling pathways on the function of bone marrow-derived dendritic cells and the effects of CP-25. European Journal of Pharmacology. 769. 8–21. 28 indexed citations
18.
Chen, Jingyu, Huaxun Wu, Qingtong Wang, et al.. (2014). Ginsenoside Metabolite Compound K Alleviates Adjuvant-Induced Arthritis by Suppressing T Cell Activation. Inflammation. 37(5). 1608–1615. 46 indexed citations
19.
Song, Shasha, Pingfan Yuan, Huaxun Wu, et al.. (2014). Dendritic cells with an increased PD-L1 by TGF-β induce T cell anergy for the cytotoxicity of hepatocellular carcinoma cells. International Immunopharmacology. 20(1). 117–123. 69 indexed citations
20.
Sun, Wu‐Yi, Wei Wei, Di Wang, et al.. (2012). Involvement of the prostaglandin E receptor EP2 in paeoniflorin-induced human hepatoma cell apoptosis. Anti-Cancer Drugs. 24(2). 140–149. 29 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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