Huarui Lu

527 total citations
19 papers, 428 citations indexed

About

Huarui Lu is a scholar working on Molecular Biology, Oncology and Ophthalmology. According to data from OpenAlex, Huarui Lu has authored 19 papers receiving a total of 428 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 8 papers in Oncology and 3 papers in Ophthalmology. Recurrent topics in Huarui Lu's work include DNA Repair Mechanisms (5 papers), Epigenetics and DNA Methylation (4 papers) and Cancer-related Molecular Pathways (4 papers). Huarui Lu is often cited by papers focused on DNA Repair Mechanisms (5 papers), Epigenetics and DNA Methylation (4 papers) and Cancer-related Molecular Pathways (4 papers). Huarui Lu collaborates with scholars based in United States and China. Huarui Lu's co-authors include Haojie Huang, Timothy C. Hallstrom, Colleen L. Forster, Min Huang, Jian Ding, Hong Zhu, Ze‐Hong Miao, Yi Chen, Yujun Cai and Jin‐Sheng Zhang and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Development.

In The Last Decade

Huarui Lu

18 papers receiving 425 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Huarui Lu United States 13 340 115 76 48 46 19 428
Rhonda Croxton United States 8 409 1.2× 217 1.9× 66 0.9× 63 1.3× 32 0.7× 8 568
Andy D.J. Pearson United Kingdom 10 395 1.2× 68 0.6× 97 1.3× 59 1.2× 80 1.7× 12 532
Philip E.D. Chung Canada 11 350 1.0× 200 1.7× 201 2.6× 51 1.1× 59 1.3× 15 512
Leyuan Bao China 11 299 0.9× 99 0.9× 133 1.8× 81 1.7× 20 0.4× 18 455
Hyeong‐Reh Choi Kim United States 5 230 0.7× 129 1.1× 185 2.4× 22 0.5× 17 0.4× 7 433
Bing An United States 11 505 1.5× 281 2.4× 62 0.8× 51 1.1× 27 0.6× 13 646
Mingzeng Sun United States 10 312 0.9× 109 0.9× 98 1.3× 35 0.7× 73 1.6× 15 585
Nathan Bushue United States 4 264 0.8× 32 0.3× 44 0.6× 53 1.1× 71 1.5× 4 342
Dawn A. Bradbury United Kingdom 8 149 0.4× 81 0.7× 60 0.8× 78 1.6× 55 1.2× 9 396

Countries citing papers authored by Huarui Lu

Since Specialization
Citations

This map shows the geographic impact of Huarui Lu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Huarui Lu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Huarui Lu more than expected).

Fields of papers citing papers by Huarui Lu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Huarui Lu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Huarui Lu. The network helps show where Huarui Lu may publish in the future.

Co-authorship network of co-authors of Huarui Lu

This figure shows the co-authorship network connecting the top 25 collaborators of Huarui Lu. A scholar is included among the top collaborators of Huarui Lu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Huarui Lu. Huarui Lu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Wang, Xiaohong, et al.. (2024). UHRF2 accumulates in early G 1 -phase after serum stimulation or mitotic exit to extend G 1 and total cell cycle length. Cell Cycle. 23(5). 613–627. 1 indexed citations
2.
Yu, Raymond C., et al.. (2022). Abstract 3432: A novel anti-CLDN6-CD137 bispecific antibody (NBL-028) for treating Claudin 6 positive solid tumors. Cancer Research. 82(12_Supplement). 3432–3432.
3.
Wang, Xiaohong, Aaron L. Sarver, Christopher L. Seiler, et al.. (2022). UHRF2 regulates cell cycle, epigenetics and gene expression to control the timing of retinal progenitor and ganglion cell differentiation. Development. 149(6). 10 indexed citations
4.
Sarver, Aaron L., Chencheng Xie, Megan Riddle, et al.. (2021). Retinoblastoma tumor cell proliferation is negatively associated with an immune gene expression signature and increased immune cells. Laboratory Investigation. 101(6). 701–718. 7 indexed citations
5.
Sum, Chi Shing, Alla Pritsker, Ray Lin, et al.. (2021). Abstract 1869: Novel TNFR2 antibodies to overcome T cell exhaustion and suppressive tumor microenvironment. Cancer Research. 81(13_Supplement). 1869–1869. 4 indexed citations
6.
Xie, Chencheng, Huarui Lu, Xiaohong Wang, et al.. (2017). Retinoblastoma cells activate the AKT pathway and are vulnerable to the PI3K/mTOR inhibitor NVP-BEZ235. Oncotarget. 8(24). 38084–38098. 21 indexed citations
7.
Lu, Huarui, Hongbo Wang, Xiaohong Wang, et al.. (2016). Loss of UHRF2 expression is associated with human neoplasia, promoter hypermethylation, decreased 5-hydroxymethylcytosine, and high proliferative activity. Oncotarget. 7(46). 76047–76061. 18 indexed citations
8.
Lu, Huarui, et al.. (2015). Rb1 and Pten Co-Deletion in Osteoblast Precursor Cells Causes Rapid Lipoma Formation in Mice. PLoS ONE. 10(8). e0136729–e0136729. 14 indexed citations
9.
Xie, Chencheng, Huarui Lu, Alice Nomura, et al.. (2015). Co-deleting Pten with Rb in retinal progenitor cells in mice results in fully penetrant bilateral retinoblastomas. Molecular Cancer. 14(1). 93–93. 32 indexed citations
10.
Lu, Huarui & Timothy C. Hallstrom. (2013). The Nuclear Protein UHRF2 Is a Direct Target of the Transcription Factor E2F1 in the Induction of Apoptosis. Journal of Biological Chemistry. 288(33). 23833–23843. 27 indexed citations
11.
Lu, Huarui & Timothy C. Hallstrom. (2012). Sensitivity to TOP2 Targeting Chemotherapeutics Is Regulated by Oct1 and FILIP1L. PLoS ONE. 7(8). e42921–e42921. 17 indexed citations
12.
Lu, Huarui, et al.. (2011). Jab1/CSN5 mediates E2F dependent expression of mitotic and apoptotic but not DNA replication targets. Cell Cycle. 10(19). 3317–3326. 19 indexed citations
13.
Lu, Huarui, Ping Liu, Yunqian Pan, & Haojie Huang. (2011). Inhibition of Cyclin-Dependent Kinase Phosphorylation of FOXO1 and Prostate Cancer Cell Growth by a Peptide Derived from FOXO1. Neoplasia. 13(9). 854–863. 24 indexed citations
14.
Lu, Huarui, et al.. (2011). Inhibition of ovarian cancer by RGD‐P125A‐endostatin‐Fc fusion proteins. International Journal of Cancer. 129(3). 751–761. 12 indexed citations
15.
Lu, Huarui & Haojie Huang. (2011). FOXO1: A Potential Target for Human Diseases. Current Drug Targets. 12(9). 1235–1244. 74 indexed citations
16.
Lu, Huarui, Xiang Wang, & Ya Wang. (2006). A Stronger DNA Damage-Induced G2 Checkpoint Due to Over-Activated CHK1 in the Absence of PARP-1. Cell Cycle. 5(20). 2364–2370. 16 indexed citations
17.
Lu, Huarui, Hong Zhu, Min Huang, et al.. (2005). Reactive Oxygen Species Elicit Apoptosis by Concurrently Disrupting Topoisomerase II and DNA-Dependent Protein Kinase. Molecular Pharmacology. 68(4). 983–994. 65 indexed citations
18.
Hu, Baocheng, Hongyan Wang, Xiang Wang, et al.. (2005). Fhit and CHK1 Have Opposing Effects on Homologous Recombination Repair. Cancer Research. 65(19). 8613–8616. 43 indexed citations
19.
Lu, Huarui, Linghua Meng, Min Huang, et al.. (2004). DNA damage, c-myc suppression and apoptosis induced by the novel topoisomerase II inhibitor, salvicine, in human breast cancer MCF-7 cells. Cancer Chemotherapy and Pharmacology. 55(3). 286–294. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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