Hitomi Tsuchida

461 total citations
19 papers, 333 citations indexed

About

Hitomi Tsuchida is a scholar working on Reproductive Medicine, Social Psychology and Molecular Biology. According to data from OpenAlex, Hitomi Tsuchida has authored 19 papers receiving a total of 333 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Reproductive Medicine, 7 papers in Social Psychology and 6 papers in Molecular Biology. Recurrent topics in Hitomi Tsuchida's work include Hypothalamic control of reproductive hormones (15 papers), Ovarian function and disorders (9 papers) and Neuroendocrine regulation and behavior (7 papers). Hitomi Tsuchida is often cited by papers focused on Hypothalamic control of reproductive hormones (15 papers), Ovarian function and disorders (9 papers) and Neuroendocrine regulation and behavior (7 papers). Hitomi Tsuchida collaborates with scholars based in Japan and South Korea. Hitomi Tsuchida's co-authors include Naoko Inoue, Yoshihisa Uenoyama, Hiroko Tsukamura, Kana Ikegami, Masumi Hirabayashi, Makoto Sanbo, Teppei Goto, Koki Yamada, Kenta Kobayashi and Sho Nakamura and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and Scientific Reports.

In The Last Decade

Hitomi Tsuchida

17 papers receiving 333 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Hitomi Tsuchida Japan 9 258 126 64 58 51 19 333
Tselmeg Mijiddorj Japan 13 282 1.1× 130 1.0× 39 0.6× 104 1.8× 51 1.0× 30 389
Kana Ikegami Japan 15 433 1.7× 218 1.7× 90 1.4× 65 1.1× 76 1.5× 22 527
Luhong Wang China 11 198 0.8× 156 1.2× 86 1.3× 47 0.8× 86 1.7× 17 405
S. León Spain 7 173 0.7× 112 0.9× 50 0.8× 27 0.5× 17 0.3× 8 315
Sangeeta Dhamija United States 9 335 1.3× 263 2.1× 76 1.2× 47 0.8× 98 1.9× 10 532
Nobuyuki Masumoto Japan 10 118 0.5× 105 0.8× 60 0.9× 22 0.4× 30 0.6× 15 365
Christina Yang United States 8 74 0.3× 56 0.4× 41 0.6× 19 0.3× 11 0.2× 11 253
Samuel D. Quaynor United States 6 275 1.1× 160 1.3× 36 0.6× 25 0.4× 31 0.6× 8 406
Jacques Young France 3 333 1.3× 183 1.5× 51 0.8× 25 0.4× 13 0.3× 5 446
C Taragnat France 8 125 0.5× 140 1.1× 41 0.6× 14 0.2× 13 0.3× 9 380

Countries citing papers authored by Hitomi Tsuchida

Since Specialization
Citations

This map shows the geographic impact of Hitomi Tsuchida's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hitomi Tsuchida with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hitomi Tsuchida more than expected).

Fields of papers citing papers by Hitomi Tsuchida

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hitomi Tsuchida. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hitomi Tsuchida. The network helps show where Hitomi Tsuchida may publish in the future.

Co-authorship network of co-authors of Hitomi Tsuchida

This figure shows the co-authorship network connecting the top 25 collaborators of Hitomi Tsuchida. A scholar is included among the top collaborators of Hitomi Tsuchida based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hitomi Tsuchida. Hitomi Tsuchida is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
2.
Uenoyama, Yoshihisa, et al.. (2024). Central δ/κ opioid receptor signaling pathways mediate chronic and/or acute suckling-induced LH suppression in rats during late lactation. Journal of Reproduction and Development. 70(5). 327–337. 1 indexed citations
3.
Tsuchida, Hitomi, et al.. (2024). AVPV <i>Kiss1</i> neuron-specific knockdown of purinergic P2X2 receptor suppresses LH surge and ovulation in <i>Kiss1-Cre</i> rats. Journal of Reproduction and Development. 70(6). 379–388.
4.
Tsuchida, Hitomi, et al.. (2023). Enkephalin-δ Opioid Receptor Signaling Mediates Glucoprivic Suppression of LH Pulse and Gluconeogenesis in Female Rats. Endocrinology. 164(3). 10 indexed citations
5.
Yamada, Koki, Yuki Enomoto, Hitomi Tsuchida, et al.. (2023). Conditional Oprk1-dependent Kiss1 deletion in kisspeptin neurons caused estrogen-dependent LH pulse disruption and LH surge attenuation in female rats. Scientific Reports. 13(1). 20495–20495. 6 indexed citations
6.
Yamada, Koki, Hitomi Tsuchida, Teppei Goto, et al.. (2023). Sex difference in developmental changes in visualized <i>Kiss1</i> neurons in newly generated <i>Kiss1-Cre</i> rats. Journal of Reproduction and Development. 69(5). 227–238. 6 indexed citations
7.
Tsuchida, Hitomi, et al.. (2023). Enkephalin-δ opioid receptor signaling partly mediates suppression of LH release during early lactation in rats. Journal of Reproduction and Development. 69(4). 192–197. 4 indexed citations
8.
Inoue, Naoko, et al.. (2023). Hindbrain Adenosine 5-Triphosphate (ATP)-Purinergic Signaling Triggers LH Surge and Ovulation via Activation of AVPV Kisspeptin Neurons in Rats. Journal of Neuroscience. 43(12). 2140–2152. 8 indexed citations
9.
Shima, Yuichi, Kanako Miyabayashi, Koji Ono, et al.. (2022). Intronic Enhancer Is Essential for Nr5a1 Expression in the Pituitary Gonadotrope and for Postnatal Development of Male Reproductive Organs in a Mouse Model. International Journal of Molecular Sciences. 24(1). 192–192. 2 indexed citations
10.
11.
Uenoyama, Yoshihisa, et al.. (2022). Opioidergic pathways and kisspeptin in the regulation of female reproduction in mammals. Frontiers in Neuroscience. 16. 958377–958377. 21 indexed citations
12.
13.
Tsuchida, Hitomi, et al.. (2021). Central µ-Opioid Receptor Antagonism Blocks Glucoprivic LH Pulse Suppression and Gluconeogenesis/Feeding in Female Rats. Endocrinology. 162(10). 17 indexed citations
14.
Minabe, Shiori, Kinuyo Iwata, Hitomi Tsuchida, Hiroko Tsukamura, & Hitoshi Ozawa. (2021). Effect of diet-induced obesity on kisspeptin-neurokinin B-dynorphin A neurons in the arcuate nucleus and luteinizing hormone secretion in sex hormone-primed male and female rats. Peptides. 142. 170546–170546. 9 indexed citations
15.
Uenoyama, Yoshihisa, et al.. (2021). Role of KNDy Neurons Expressing Kisspeptin, Neurokinin B, and Dynorphin A as a GnRH Pulse Generator Controlling Mammalian Reproduction. Frontiers in Endocrinology. 12. 724632–724632. 63 indexed citations
16.
Uenoyama, Yoshihisa, Hitomi Tsuchida, Kana Ikegami, et al.. (2021). Direct evidence that KNDy neurons maintain gonadotropin pulses and folliculogenesis as the GnRH pulse generator. Proceedings of the National Academy of Sciences. 118(5). 108 indexed citations
17.
Watanabe, Takashi, Tatsunori Nishimura, Takanori Hayashi, et al.. (2020). Upregulation of S100A10 in metastasized breast cancer stem cells. Cancer Science. 111(12). 4359–4370. 22 indexed citations
18.
Tsuchida, Hitomi, Koki Yamada, Yuki Enomoto, et al.. (2020). Paraventricular Dynorphin A Neurons Mediate LH Pulse Suppression Induced by Hindbrain Glucoprivation in Female Rats. Endocrinology. 161(11). 27 indexed citations
19.
Tsuchida, Hitomi, Nahoko Ieda, Kana Ikegami, et al.. (2018). Somatostatin-Somatostatin Receptor 2 Signaling Mediates LH Pulse Suppression in Lactating Rats. Endocrinology. 160(2). 473–483. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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