Hiroyuki Ido

923 total citations
10 papers, 754 citations indexed

About

Hiroyuki Ido is a scholar working on Molecular Biology, Immunology and Allergy and Radiology, Nuclear Medicine and Imaging. According to data from OpenAlex, Hiroyuki Ido has authored 10 papers receiving a total of 754 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Immunology and Allergy and 4 papers in Radiology, Nuclear Medicine and Imaging. Recurrent topics in Hiroyuki Ido's work include Cell Adhesion Molecules Research (6 papers), Monoclonal and Polyclonal Antibodies Research (4 papers) and CAR-T cell therapy research (3 papers). Hiroyuki Ido is often cited by papers focused on Cell Adhesion Molecules Research (6 papers), Monoclonal and Polyclonal Antibodies Research (4 papers) and CAR-T cell therapy research (3 papers). Hiroyuki Ido collaborates with scholars based in Japan and United States. Hiroyuki Ido's co-authors include Kiyotoshi Sekiguchi, Ritsuo Nishiuchi, Maria Hayashi, Noriko Sanzen, Yoshiko Yagi, Tsutomu Tsuji, Junichi Takagi, Masashi Yamada, Sugiko Futaki and Shaoliang Li and has published in prestigious journals such as Journal of Biological Chemistry, Genes & Development and Biochemical and Biophysical Research Communications.

In The Last Decade

Hiroyuki Ido

10 papers receiving 749 citations

Peers

Hiroyuki Ido

ONCOL

Carl‐Fredrik Tiger Sweden

Petri Nykvist Finland

Petra Sonneveld Netherlands

Christian Frie Germany

Olena Jacenko United States

Sophie Astrof United States

Takahiro Suzuki Japan

Emiko Fukumoto Japan

Ariel Boutaud United States

Yoshihiko Yamada United States

Carl‐Fredrik Tiger Sweden

Hiroyuki Ido

484 ×1.2

507 ×1.3

309 ×1.2

48 ×0.6

80 ×1.0

ONCOL

Citations per year, relative to Hiroyuki Ido Hiroyuki Ido (= 1×) peers Carl‐Fredrik Tiger

Countries citing papers authored by Hiroyuki Ido

Since Specialization

Citations

This map shows the geographic impact of Hiroyuki Ido's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hiroyuki Ido with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hiroyuki Ido more than expected).

Fields of papers citing papers by Hiroyuki Ido

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hiroyuki Ido. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hiroyuki Ido. The network helps show where Hiroyuki Ido may publish in the future.

Co-authorship network of co-authors of Hiroyuki Ido

This figure shows the co-authorship network connecting the top 25 collaborators of Hiroyuki Ido. A scholar is included among the top collaborators of Hiroyuki Ido based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hiroyuki Ido. Hiroyuki Ido is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

Uchibori, Ryosuke, Takeshi Teruya, Hiroyuki Ido, et al.. (2018). Functional Analysis of an Inducible Promoter Driven by Activation Signals from a Chimeric Antigen Receptor. Molecular Therapy — Oncolytics. 12. 16–25. 16 indexed citations

Uchibori, Ryosuke, Takeshi Teruya, Soranobu Ninomiya, et al.. (2015). 225. Activation Signals from CD19-CAR Permit NFAT-Controlled Inducible Expression of Transgenes in PBMCs. Molecular Therapy. 23. S89–S89. 1 indexed citations

Tsukahara, Tomonori, Ken Ohmine, Chihiro Yamamoto, et al.. (2013). CD19 target-engineered T-cells accumulate at tumor lesions in human B-cell lymphoma xenograft mouse models. Biochemical and Biophysical Research Communications. 438(1). 84–89. 17 indexed citations

Taniguchi, Yukimasa, Hiroyuki Ido, Noriko Sanzen, et al.. (2009). The C-terminal Region of Laminin β Chains Modulates the Integrin Binding Affinities of Laminins. Journal of Biological Chemistry. 284(12). 7820–7831. 65 indexed citations

Gao, Jing, Mindy C. DeRouen, Chih‐Hsin Chen, et al.. (2008). Laminin-511 is an epithelial message promoting dermal papilla development and function during early hair morphogenesis. Genes & Development. 22(15). 2111–2124. 95 indexed citations

Ido, Hiroyuki, Yukimasa Taniguchi, Maria Hayashi, et al.. (2008). Laminin Isoforms Containing the γ3 Chain Are Unable to Bind to Integrins due to the Absence of the Glutamic Acid Residue Conserved in the C-terminal Regions of the γ1 and γ2 Chains. Journal of Biological Chemistry. 283(42). 28149–28157. 41 indexed citations

Ido, Hiroyuki, Aya Nakamura, Reiko Kobayashi, et al.. (2007). The Requirement of the Glutamic Acid Residue at the Third Position from the Carboxyl Termini of the Laminin γ Chains in Integrin Binding by Laminins. Journal of Biological Chemistry. 282(15). 11144–11154. 84 indexed citations

Nishiuchi, Ritsuo, Junichi Takagi, Maria Hayashi, et al.. (2006). Ligand-binding specificities of laminin-binding integrins: A comprehensive survey of laminin–integrin interactions using recombinant α3β1, α6β1, α7β1 and α6β4 integrins. Matrix Biology. 25(3). 189–197. 311 indexed citations

Ido, Hiroyuki, Kenji Harada, Yoshiko Yagi, & Kiyotoshi Sekiguchi. (2005). Probing the integrin-binding site within the globular domain of laminin-511 with the function-blocking monoclonal antibody 4C7. Matrix Biology. 25(2). 112–117. 29 indexed citations

10.

Ido, Hiroyuki, Kenji Harada, Sugiko Futaki, et al.. (2004). Molecular Dissection of the α-Dystroglycan- and Integrin-binding Sites within the Globular Domain of Human Laminin-10. Journal of Biological Chemistry. 279(12). 10946–10954. 95 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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